Herpes virus infection pathology. NZ DermNet

Facts about skin from the New Zealand DermNet Trust. Topic index: A B C D E F G H I J K L M N O P Q R S T U V W X Y Z Herpes simplex, varicella zoster and herpes infections show identical histology. Clinical correlation or immunohistochemistry and / or viral culture or PCR (of an Appropriate swab or tissue specimen) is required to differentiate viral infections in Original. Histology of herpes infections The histology of herpes infections is very distinctive. The low power pattern of a typical lesion is of an intraepidermal blister (Figure 1). The key feature is acantholysis With solitary Within the blister cavity keratinocytes (Figures 1, 2, 3). Keratinocytes will show the viral infection of nuclear Changes. These included margination of the nuclear chromatin, multinucleation and nuclear inclusions (Figures 3, 4, 5). The viral inclusions are small pink deposits with a clear halo seen Within the nucleus. When present in herpes virus infection and present With The Changes of other nuclear esta infection Cowdry They are called Type A inclusions. Cowdry Type B inclusions are Associated With other infections: such as polio virus and do not Have the other nuclear Changes of herpes infection.

Early Changes of vacuolation in the cytoplasm May be seen along the basal keratinocytes. As the cells swell and separate, the eosinophilic cytoplasm Becomes remarkable Particularly in the multinucleated cells. The inflammatory infiltrate is mixed, lymphocytes and neutrophils Predominantly With faq frequently seen scattered eosinophils (Figure 6). Histological variants of herpes infections Herpes folliculitis: Changes In This variant can be subtle and HENCE Particularly in early Easily overlooked lesions or When the follicle is only partially Represented. The key finding is of a necrotic hair follicle With An Associated dense surface and deep lymphocytic infiltrate With neutrophils. Typical viral Changes can be seen in acantholytic epithelial cells of the infundibular region Predominantly Involved in or adjacent epidermis. Verrucous varicella zoster and herpes: In this variant there is hyperkeratosis and epidermal acanthosis prominent in Addition to the viral cytopathic changes. Special tests in herpes infections Immunoperoxidase staining by monoclonal antibody is available to herpes simplex types 1 and 2 and to varicella zoster. It is possible to perform PCR Present and / or an Appropriate culture specimen to distinguish Between Both herpes simplex viruses and the herpes zoster. Differential diagnosis of herpes infections Coxsackie virus infection / hand foot and mouth disease: Blisters show intraepidermal vesiculation and acantholysis nuclear inclusions or without multinucleation.

Pemphigus vulgaris: Clinically These conditions would rarely be confused. With NOTED acantholysis suprabasal vesicle formation is in advanced lesions, and small numbers of cells are seen acantholytic Within esta space. Viral Changes are lacking. However, it Should be disseminated herpes simplex That NOTED infection (Kaposi’s varicellifom eruption) pemphigus vulgaris can complicate virus. In troublesome cases immunofluorescence studies are decisive but IF NECESSARY Usually the additional special studies NOTED above May Also be used. It is worth Noting a number of conditions That Have Been Reported to Arise in the site of prior herpes zoster infection. These include chronic lymphocytic leukemia, granuloma annulare, lichen planus, keloid scarring, vasculitis, sarcoidosis, morphea, lymphoma and skin cancers. The acute viral infection of Changes are evident Usually not. Related information References: Skin Pathology (2nd edition, 2002). Weedon D Pathology of the Skin (3rd edition, 2005).

PH McKee, J. Calonje JE, Granter SR On DermNet NZ: Books about skin diseases: See the DermNet NZ bookstore Author: Dr Ben Tallon, Dermatologist / dermatopathologist, Tauranga, New Zealand.

Herpes Virus “Dr. Oscar Santis L.

WHAT IS HERPES? Genital herpes is a contagious disease caused by type 2 (HSV-2) and herpes simplex virus type 1 (HSV-1). Most genital herpes are caused by HSV-2. Most people with HSV-1 or HSV-2 no signs. If signs occur, they appear as small blisters on the genitals. The blisters break, leaving tender ulcers (sores) that may take two to four weeks to heal. It tends to recur periodically. You can stay indefinitely in the body, outbreaks tend to decrease as the years pass. It is a very common condition that has now increased significantly by the sexual freedom of youth and non correct use of protective elements. In these times is seen to appear from 12 years, one out of five adolescents and adults, have had genital HSV infection. Infection genital HSV-2 is more common in women (approximately one out of four women) than among men (almost one out of five). This may be because it is more likely that transmission occurs male to female transmission from women to men. TYPES OF VIRUS

HSV-1 and HSV-2 can be found in ulcers caused by viruses and released by them, but between outbreaks the virus can also be released by the skin that does not seem affected or do not have ulcers. Usually, a person can only be infected with HSV-2 during sexual contact with someone who has an infection with HSV-2 genital. Transmission can occur from an infected partner who does not have a visible sore and may not know they are infected. HSV-1 can cause genital herpes, but it more commonly causes infections of the mouth and lips, also called “fever blisters. ” It can be caused by oral-genital or genital-genital contact with a person who has HSV-1 infection. Outbreaks of HSV-1 genital recur less regularly than outbreaks of genital HSV-2. Most people infected with HSV-2 are unaware they have the infection. However, if signs and symptoms occur during the first outbreak, they can be quite pronounced. The first outbreak occurs within the following transmission of the virus two weeks and sores typically heal within two to four weeks. You can give flu, including fever and swollen glands symptoms. Most people with HSV-2 infection may have very mild signs that they do not even realize or confused with insect bites or another skin condition. Most people who have been diagnosed with a first episode of genital herpes will have several (typically four or five) outbreaks (symptomatic recurrences) within a period of one year. Usually these recurrences decrease in frequency as time passes.

It can cause the repeated presence of genital ulcers, can be severe in people with immunocompromised systems. Frequently causes psychological distress in people who know they are infected. It can contribute to the spread of HIV, the virus that causes AIDS. It makes people more susceptible to HIV infection and causes people living with HIV more infectious. Genital HSV can cause potentially fatal infections in newborns. It is important that women avoid contracting herpes during pregnancy because the occurrence of a first episode during pregnancy causes a greater risk of the virus being transmitted to the baby. If a woman has active genital herpes at delivery it is usually performed a cesarean delivery. The signs and symptoms associated with HSV-2 can vary widely. Doctors can diagnose genital herpes by visual inspection if the outbreak is typical, and by obtaining a sample from an ulcer sent for laboratory analysis. It can be difficult to diagnose infections if there is no clear outbreak. Blood tests, which detect HSV-1 or HSV-2 may be useful, although the results may not definitional. There is no treatment that can cure herpes, but antiviral medications can shorten and prevent outbreaks during the time the person takes the medication. Daily treatment for symptomatic herpes can reduce the possibility of transmission to sexual partners of infected people.

The safest way to avoid transmission of sexually transmitted diseases, including genital herpes, is to abstain from sexual contact or have a lasting relationship with a stable and only couple with evidence (-) and who is not infected. Correct and consistent use of latex condoms reduces the risk of contracting herpes only if the infected area and the area of ​​potential exposure is protected. It is possible that the condom may not cover all infected areas, even correct and consistent use of latex condoms can guarantee protection against genital herpes. People with herpes should abstain from sexual activity with uninfected sexual partners when you have injuries or other symptoms of herpes. It is important to know that even if the person has no symptoms can still infect their sexual partners. This should be advised to sexual partners of infected people. Sexual partners should get tested to determine if they are infected with HSV. A screening test for HSV-2 whose results are positive indicates that most likely have genital herpes infection. This applied to herpes is also considered in condyloma oncogenic virus (human papilloma virus or HPV). (EXTRACT http://www. cdc. gov/std/Spanish/STDFact-Herpes-s. htm#Whatis)

Herpes labialis

Concept It is an infection that usually appears as small painful blisters in the mouth, gums, lips, corners and around, caused by herpes simplex. It is extremely common, approximately 80% of Americans have been exposed to herpes simplex at the age of 20 years, which causes cold sores. Causes Cold sores are caused by the infection of herpes simplex type 1, initially may be no symptoms or appear small ulcers in the mouth. Subsequently, the virus remains dormant (inactive) in the nervous tissue. Sometimes it reactivates herpes and reappears in the same place as before. The virus that causes genital herpes, the type 2 virus, can also be reached in some cases (oral sex) to cause cold sores. Herpes viruses are easily spread. One can get this virus if: It has intimate or personal contact with someone who is infected. Toca infected with the virus as razors, towels, plates and other contaminated items that are shared elements. symptoms

The first appearance of cold sores can be mild or severe and usually occurs between the first year and 5. The first symptoms usually appear after 1 or 2 weeks after exposure to the virus and include: Fever that can extend up to 5 days and can occur before the appearance of blisters Sore throat Swollen lymph nodes in the neck Labial herpes recurrence is usually milder and can occur during: The symptoms of an outbreak usually include: Injuries, wounds or rashes around the mouth, lips and gums Small blisters filled with clear yellow liquid Appearance of small blisters that formed after break open and drain Scabs, yellowish shells that flow from the affected area and expose the skin is healing Agglomeration of several small blisters that sprout forming a large Diagnosis

The diagnosis is established taking into account the appearance of the wound, examined whether glands in the neck or groin are swollen or through a culture of it. Treatment Symptoms may disappear on their own without treatment within one or two weeks. Your doctor may prescribe medication to fight the virus, called antivirals. These can help reduce pain and symptoms disappear faster. Medications used to treat mouth ulcers include: acyclovir famciclovir valacyclovir Antiviral topical creams can be used to treat cold sores but must be applied every two hours and are quite costly Wash blisters with soap and water (preferably one antibacterial) to minimize the chances of the virus spreading to other skin areas Place ice can relieve some pain Share:

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Files veterinary medicine – Rhodococcus equi pneumonia and colitis in a foal: First report in

First case described in Chile of pneumonia and colitis Rhodococcus equi in a potrillo Rhodococcus equi pneumonia and colitis in a foal: First report in Chile E. Paredes1, M. V. , Dr. med. vet . ; R. GALLEGO2, M. V .

; A. M. Channel3, M. V. , M. Sc . ; O. ARAYA4, M. V. , Ph. D . ; E.

CHAHUÁN5, T. M . ; P. THOMAS5, T. M . ; J. ZAMORA5, M. V. 1Institute of Animal Pathology, Fac. Veterinary Science, Universidad Austral de Chile. Military 2Criadero Riñihue, Chilean Army. Animal 3Patología. Fac.

Of Agriculture and Veterinary. Universidad Nacional del Litoral, P. Kreder 2805, 3080 Esperanza, Santa Fe, Argentina. 4Instituto of Veterinary Clinical Sciences, Fac. Veterinary Science. 5Instituto of Microbiology, Faculty of Sciences, Universidad Austral de Chile. ; Casilla 567, Valdivia, Chile. SUMMARY Rhodococcus equi was isolated for the first time in Chile from lung sample, bronchial lymph node and mesenteric lymph node Obtained from a 3 month chronic enteritis and foal With pneumonia. Gross lesions Were Characterised by pneumonia pyogranulomatous multiple abscesses varying size. Multiple well-defined necrotic foci, 1 to 3 cm in diameter, in the mucosal surface Were of the colon, and mesenteric lymph nodes Were enlarged. Due to the infectious disease condition, it is Necessary to carry out further studies to determine STI epidemiological relevance in Chile.

Keywords: pneumonia, colitis, Rhodococcus equi foal. Key Words: pneumonia, enterocolitis, Rhodococcus equi foal. INTRODUCTION Rhodococcus equi (R. equi), formerly classified in the genus Corynebacterium as C. equi, is an important pathogen of foals being pyogranulomatous etiologic agent of bronchiolitis and ulcerative enteritis, preferably less than 6 months old young horses (Burks, 1996; Nay, 1996; Fernandez, 1997; Castagna Vargas, 1998) with abscess formation of thoracic and mesenteric lymph nodes (Knottenbelt, 1993; Lopez 1995; Van Kruiningen, 1995) also may occur joint injuries, skin, bone , eye and various viscera (Fernandez et al. , 1995; Van Kruiningen, 1995; Burks, 1996; Nay 1996). R. equi has also been isolated from healthy and diseased animals of other species, such as pigs, cattle, sheep and also in free-living animals (Bern and Lämmler, 1994; Soedarmanto et al, 1997, 1998. ). In man is gaining importance as an emerging disease, particularly in immunosuppressed individuals (Prescott, 1991; Bern and Lämmler, 1994; Fernandez et al. , 1995; Takai et al. , 1995; and Lämmler Fuhrmann, 1997).

This article aims to communicate the first isolation in the country of R. equi, describe the typical injuries and cultural characteristics and antibiotic sensitivity of germ found in a dead foal pneumonia in the Tenth Region, Chile. MATERIAL AND METHOD In a stud farm 10 cases of diarrhea as the only symptom in foals of different breeds of 20-45 days old were detected in mid-November 1995, which did not respond to therapy potentiated sulfa (sulfa + trimethoprim) for 5 days and supportive therapy (oral rehydration salts, ringer lactate serum EV, bismuth subnitrate Flumixin oral Meglumine, EV); due to inclement weather, the offspring had remained confined stabling night with their mothers in a shed for three days, without spare bed. At the end of November totaled 40 cases detected (80% of total offspring), when Salmonella sp is isolated. E. coli and from faecal samples, starting specific treatment according to antibiogram sensitivity (Advocin® and Baytril) for 4 days, with no clinical improvement in most of them. In January 1996, 14 offspring remain with diarrhea (35% of affected), 9 of which had also respiratory compromise, which were treated with gentamicin and sodium penicillin E. V. (3 times a day for 10 days), with no remission of the symptoms. It was decided to leave without antibiotic treatment to a colt race Selle Français, 90 days old, being sacrificed later. necropsy according to the technique described by Walls and Cubillos (1995) was performed, taking samples in neutral formalin 10% of affected tissues (lungs and large colon) and samples of lung, lymph node bronchial and mesenteric lymph node for microbiological examinations. To this effect it was plated on sheep blood agar and chocolate agar, later to make morphological, physiological and biochemical isolates according to described by Holt et al exam.

(1994) and Fuhrmann and Lämmler (1997). In addition, isolates were determined sensitivity to different antibiotics by conventional methods. RESULTS AND DISCUSSION a) Clinical manifestations and laboratory tests. The clinical symptomatology was characterized by watery diarrhea began at 36 days of age, with no initial respiratory compromise or alteration of physiological constants situation is described by Yager (1987) and Burks (1996), who argue that infection R . equi, diarrhea this may be the only sign. At 75 days of age, respiratory symptoms (mild cough, crackles and hissing noises cranioventral level of both lungs) and fever, similar to that described by Prescott (1991), Vivrette (1992) and Burks (1996) are detected. During the course of the disease he continued suckling foal. After initiation of respiratory symptoms blood count was performed, finding neutrofilia (10. 0 mil / microliter) with deviation to the left (N. baciliform 1. 5 mil / ul) and increased plasma fibrinogen concentration 10 g / l). The insidious presentation of the disease in this colt, characterized first by diarrhea and later by bronchopneumonia tending to chronicity and the history of other respiratory symptoms sporadically presented earlier in the stud farm, became suspicious of R.

equi infection , since the clinical characteristics coincided fully with those described in the literature (Vivrette, 1992; Knottenbelt. , 1993; Fernandez et al, 1995; Lopez, 1995; Van Kruiningen, 1995, Burks, 1996; Nay, 1996; Fernandez et al . , 1997; Vargas Castagna, 1998). b) Findings anatomical and histopathological. Necropsy performed the animal accentuated suspicion, since the postmortem examination revealed that serious injuries were confined to lungs and large colon. Both lungs were observed without collapsing, irregularly firm, with the presence of multiple nodules of varying sizes scattered all lobes and protuían into the pleural surface. When cutting the lung tissue, these nodules were observed as multiple focal and confluent areas of yellowish white color, completely replacing the lung tissue (Figure 1) and corresponding to areas of chronic inflammation, central necrosis and discrete purulent exudate. Bronchial lymph nodes were enlarged, hyperemic and exudative. Figure 1. Lung (cutting surface): confluent foci of necrosis and purulent inflammatory reaction. Lung (cut surface): large and confluent granulomas caseated. In addition, the lining of the colon showed greater number of circular ulcers, up to 3 cm in diameter, with a thick border of cream and a depressed with traces of necrotic tissue (Figure 2) center. Mesenteric lymph nodes were increased volume and had a cutting surface with great whitish exudates.

Figure 2. greater Colon: Circular mucosal ulcerations. Mucosal surface of the colon: well defined necrotic foci. Furthermore, histopathology found in the lungs the presence of large areas of necrosis with severe inflammatory reaction neutrohistiocitaria type, finding some giant cells. In turn, the more severe inflammatory bowel exhibited neutrohistiocitaria reaction type with large areas of necrosis, covering mucosa and submucosa. c) Microbiology. All lesions observed, and that characterize a disorder caused by this bacterium box, were confirmed with isolation lung, bronchial lymph nodes and mesenteric pure culture of a Gram positive bacteria and weakly acid resistant Ziehl – Neelsen modified, pleomorphic aspect cocoide (0,71,4 microns), distributed in cuneiform palisades or encapsulated. Colonies were small, 2 to 3 mm in diameter, smooth, shiny, pale mucous salmon acquired after March 4 days of culture and development in gelatin presented a strong orange. Other features are included in Table 1. TABLE 1. biochemical and physiological characteristics of R. equi strains isolated. Biochemical and physiological properties of the isolated strains.

biochemical and physiological characteristics Motility catalase gelatinase lipase urease Nitrate reduction CAMP Glucose acid Acid Lactose Acid Mannitol – +

– + + + + – – – According to the stated characteristics, it was identified as Rhodococcus equi strain, being sensitive to different antibiotics that are included in Table 2. TABLE 2. Antibiograms of the R. equi isolates. Antibiogram of the isolated R.

equi strains. Antibiotic Sensitivity Penicilinaaa Cloxacilinaa Erythromycin Lincospectin Bipencilaaa Baytrilaaaa Advocinaaa Ceftiofuraaa Resistant Resistant

Sensitive Sensitive Sensitive Sensitive Sensitive Sensitive Although considered to R. equi has a cosmopolitan distribution, they had not described in Chile infections in foals by this agent. The table causes this microorganism is generally sporadic in enzootic areas, but also outbreaks have been reported responsible for great economic losses in the stud farm, although the most common is that the infection goes unnoticed (Knottenbelt, 1993, Burks, 1996; Soedarmanto , 1998). R. equi, causing a suppurative bronchopneumonia Subacute to chronic, associated with purulent lymphadenitis, pneumonic infections that 50% are complicated by intestinal infection (Yager, 1987), especially in foals under 6 months of age and particularly those of January 3 months of life, in which it has not yet fully matured their immune system being immunocompromised adults also particularly susceptible to infection (Burks, 1996). You can not ignore that, in addition, on some occasions it is believed that viral infections may be predisposing factors, such as with equine herpesvirus type 2 (EHV-2) (Nordengrahn et al. , 1996).

Treatment of the disease is difficult, not by the resistance R. equi may have antibacterial, but rather the facultative intracellular bacteria property, a factor that probably concurred in failure of therapy instituted in previous cases, since the isolated strain was sensitive to several antibiotics (table 2), including employees at the farm, although it must be remembered that in vitro conditions are different from those in vivo, which does not allow the conclusion that the antimicrobial susceptibility test result can be the same in the animal. Also, it should be recalled that the organism quickly becomes resistant when the antibiotic is used alone, hence it is necessary to use simultaneously more than one drug in prolonged, being recommended the primary application of erythromycin and rifampin, reinforced therapy support such as dimethylsulfoxide, aminophylline and even ranitidine (Fernandez et al. , 1995; Burks, 1996). However, Gustafsson et al. (1997) report that erythromycin may induce severe colitis in horses associated with large changes in intestinal microbiota of these animals. The discovery of R. equi in the country to presume that the infection is quite widespread, causing serious problems, given the survival and development of the agent in the external environment (especially where there is overcrowding, agglomeration of fecal material and dusty environments), the different routes of transmission, the difficulty of achieving an early diagnosis by the characteristic insidious disease development and the progressive increase in the incidence denouncing the literature, are a set of attributes that make R. equi one of the most problematic pathogens, refractory to treatment and causes a high rate of mortality in foals (Higuchi et al. , 1998). It is therefore high convenience continue studies on the subject to determine the epidemiological importance in the country, not only for its role in the health of the horses, but also because it is considered an important agent of emerging infections the human species, especially in immunosuppressed people. SUMMARY Described for the first time in the country, isolation of Rhodococcus equi from an infection in a colt of 3 months old, who suffered from chronic enteritis and pneumonia.

Also, they disclosed the lesions observed in the postmortem and histopathological examination consisting of multiple pulmonary nodules with areas of chronic inflammation, central necrosis with purulent exudate and bronchial lymph nodes increased in size: hyperaemic and exudative. In addition, the large colon showed ulcerations with necrotic tissue and mesenteric lymph nodes were enlarged with great exudation, lesions which show typical features that characterize this pyogranulomatous infection of foals. As this is an emerging infectious disease, it is necessary to continue studies, in order to determine its epidemiological relevance in Chile. _________________________________ Accepted: 05/02/2000. BIBLIOGRAPHY BERN, D. , CH. Lämmler. 1994. Biochemical and serological Characteristics of Rhodococcus equi isolates from animals and humans. J. Vet.

B Med. 41: 161165. [Links] Burks, B. 1996. Managing Rhodococcus equi infections in foals. Vet. Med. 91: 656662. [Links] CASTAGNA VARGAS, A. 1998. Infecçâo Rhodococcus equi.

Quoted in RIET-CORREA, F. , A. L. Schild, M. C. MENDEZ (Eds. ). Doenças of ruminants and equines. Edit. University, Pelotas, Brazil. [Links] FERNÁNDEZ, A. S.

, S. M. Estein, P. SOTO. 1995. Rhodococcus equi: a model of immunopathology. Arch. Med. Vet. 27: 512. [Links] FERNÁNDEZ, A. S.

, J. F. PRESCOTT, V. M. NICHOLSON. Protective effect 1997. Against Rhodococcus equi infection in mice of purified IgG from horses Vaccinated With virulance associated protein (VAP) enriched antigen. Vet. Microbiol. 56: 187-192. [Links] Fuhrmann, C. , C.

Lämmler. 1997. Characterisierung von Rhodococcus equi Mensch und Pferd von isolaten. Berliner Münch. Tierarztliche Wochenschr. 110: 5459. [Links] Gustafsson, A. , V. BAVERUD, A. GUNNARSSON, M. H. Rantzien A.

Lindholm, A. , FRANKLIN, M. HORN-Rantzien. 1997. The association of erythromycin ethylsuccinatte With acute colitis in horses in Sweden. Equine Vet. J. 29: 314318. [Links] Higuchi, T. , S. TAHARAGUCHI, A. HASHIKURA, S.

Hagiwara, C. GOJO, S. Satoh M. Yoshida, S. TAKAI. 1998. Physical and serological examinations of foals at 30 and 45 days of age for early diagnosis of Rhodococcus equi infection on endemically infected farms. JAVMA 212: 776 781. [Links] HOLT, J. G. , N. R.

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Knottenbelt, D. C. 1993. Rhodococcus equi infection in foals: a report of an outbreak on a thoroughbred stud in Zimbabwe. Vet. 132 Rec. 7985. [Links] LOPEZ, A. 1995. Respiratory System. Quoted in: CARLTON, W. W.

and M. D. Mc GAVIN. Thomson’s Special Veterinary Pathology. 2nd Edit. Mosby, St. Louis. [Links] NAY, T. S. 1996. Extra-pulmonary Rhodococcus equi in a foal throroughbred. Dog.

Vet. J. 37: 623624. [Links] NORDENGRAHN, A. , M. RUSVAI, M. MERZA, J. EKSTROM, B. Morein, S. Belak. Equine herpesvirus 1996. Type 2 (EHV-2) as a predisposing factor for Rhodococcus equi pneumonia in foals: prevention of the disease bifactorial With EHV-2 immunostimulating complexes.

Vet. Microbiol. 51: 5568. [Links] WALLS, E. , V. CUBILLOS. 1995. Manual of Necropsy in Domestic Animals. Faculty of Veterinary Science, Universidad Austral de Chile, Valdivia. [Links] PRESCOTT, J. F.

Rhodococcus equi 1991: an Animal and human pathogen. Clinical Microbiology Reviews 4: 20-34. [Links] SOEDARMANTO, I. , R. OLIVEIRA, CH. Lämmler, H. DURRLING. 1997. Identification and epidemiological relationship of Rhodococcus equi isolated from cases of lymphadenitis in cattle. Zbl. Bak. 286: 457467.

[Links] SOEDARMANTO, I. , W. Zhicai, A. SETYAMAHANANI, CH. Lämmler. 1998. Pheno- and genotyping of Rhodococcus equi isolated from faeces of healthy horses and cattle. Res. Vet. Sci. 64: 181185. [Links]

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Did you know that beauty concoctions, including mascara and rouge, are personal? Dermatologists explain why amiguis beauty products should not be paid. Consider the main problems as being good people: You can stick herpes: “The person may suffer from herpes and, although it has not broken out, could spread to the other girl who lends lipstick; even the person without being herpes because the virus is still there. The bug is hidden, caught the person weak and the virus appears. The mouth is an area of ​​high risk for the subject of the mucosa, “explains dermatologist John Honeyman, of the School of Medicine at UC. He insists that the virus that causes rashes remains with the person throughout life and that contagion occurs, even if no visible injuries. “If I have a cold and occupied a lip, the boto. Otherwise, if you still use run the risk of following autoinoculando the bug, “the dermatologist Cristina Montero Avansalud. A lipstick can last two years, but well maintained: “There are people who when they have a cold cast lipstick, arguing that that are passed. Always hold lipstick, leaving viral particles in this object. They are autoinoculando the bug. ”

Conjunctivitis sight: under no circumstances should share or provide mascara and eyeliner because it can increase the presence of eye infections. “A hobby that people have is to open the tube and shove the brush several times. With that oxygenates and promotes the growth of bacteria. That puts and takes is lousy, “explains Dr. Montero. He adds that “six months have to fire him despite last longer. We must think of bacterial growth. ” Dr. Honeyman said also the mascara can be transmitted bacteria and germs. “There is more risk given a cream because the skin has more defenses than the mucosa of the conjunctiva,” he warns. “The mascara has an expiration date: no more than six months,” agrees Roger Reyna, director of Bobbi Brown, who has painted to Sarah Jessica Parker and Antonio Banderas. He says: “The 60 percent of women use expired products unknowingly and even bacteria accumulates on the brush mascara.

If the friend takes the product can be transferred to your eye. ” It can infect the skin: Dr. Montero explains that creams can cause skin infections. “Each person has their own bacterial flora. Then a skin can be colonized by a bug that I have and sharing makeup, I catch that same bug in my skin, it can be pathological. ” He adds that this is the chance. “I can not pass anything, but it is likely that the girl occupying my makeup just have a bug. For example, a Demodex infestation (mite). If you have rosacea, acne or seborrheic dermatitis (skin diseases that are superinfected) . . . there are bugs stuck inside.

” ¿Make-up ? : “Today we are not just cosmetic makeovers; now they come with other properties: have vitamin E, an antioxidant, sunscreen, hyaluronic acid, “says Dr. Montero. But all these additional improvements expire when the product reaches the day and time of maturity. Alvaro Marquez, the Chamber of Cosmetics Industry, suggests that you have to buy name brand products and have well labeled the origin of import, who address matters and to your phone. He adds that skin creams and face, now also used by men, should not last more than a year. “People buy to use and not to keep it,” he says. To learn more about cosmetic and health, visit www. ispch. cl/cosmeticos “As a general rule, if I have a cold and occupied a lip, the boto”

Cristina Montero, dermatologist Avansalud

University Institute of the Hospital Italiano de Buenos Aires

Viral infections such as bio-anthropological markers of human migration events The research aims to deepen the knowledge of the ethnic diversity of contemporary Argentina population, through the study of evolution and genotypic characterization of Latin American strains of HTLV 1/2, HHV-7 and HHV-8 associated with migratory events ancestral humans. The project by Dr. Trinks and Lic. María Laura Hulaniuk (doctoral fellow at the Institute of Basic Sciences and Experimental Medicine, University Institute). This project, in collaboration with the Genetic Fingerprinting (SHDG) of the Faculty of Pharmacy and Biochemistry, University of Buenos Aires, led by Dr. Daniel Corach, part of the idea that viruses are microorganisms that have accompanied the man throughout human development, and this migration have also evolved. By studying the evolution of these viruses and their distribution throughout the planet, you can trace the pattern of human migrations. “They developed techniques to map or trace the ancestry of modern Argentina evolution, studying markers you are in our DNA. If we add what they do to us what we do here at the Institute from the point of view of the virus- we will be able to increase the knowledge of the origin of the current population Argentina “explains Dr. Trinks and broad” work with virus they have certain characteristics, among which, we are working at this moment-and for which we already have some results presented in congresos- is the Herpes virus type 8 “. The Herpes Virus Human type 8 (HHV-8) is the cause of Kaposi’s sarcoma, a malignant tumor that is, a form of cancer, which may appear on the skin or the viscera and, today, it is closely associated immunosuppression, mainly related to Acquired Immune Deficiency Syndrome (AIDS). It has also been shown to have a global geographic distribution and is most prevalent in African and Latin American populations.

Unfortunately, in our country there is no data on the prevalence of this virus in the population. “One of the first steps of this project was to study groups of blood donors from the Italian Hospital and across the country, to determine the prevalence of this type of herpes virus. We found truly surprising results: in the northwest region in Jujuy specifically, we found a high prevalence of 12. 3% in units of blood were transfused. ” This virus, which is not normally It tests in blood banks is being analyzed samples from infected patients provided by the Department of Pathology of infected patients in the Italian Hospital, to study its variability and compare it with the ancestral population, through molecular methods. The contributions of the research project The project has two input lines: on the one hand, the ancestral population since the Argentina population is extremely diverse and unique composition worldwide for the high degree of mixing it presents. For anthropologists, forensic or even those engaged in forensic genetics, is a great addition to using different resources, and in this case the virus bring its action to further deepen understanding of the highly complex having the composition of the population. On the other hand is relevant from the point of view of the virus, “this is a virus that our country has not been extensively studied. Then we are moving this way. For virology or infectious diseases to determine the prevalence of different virus the different regions of our country and is also important from the point of view of public health because it allows doctors to know what we are are facing in today’s society “emphasizes Dr. Trinks. the subsidy

The grant helped mainly for the purchase of reagents, which allowed us to make rapid progress on the project. It is able to manage the flow of financial resources allowed them to speed up administrative processes and get faster reagents. “It allowed us to move much faster in the project have the reagents. We achieved several publications in congresses and we are already writing the first manuscript to send to publish the project, so it really was an important help “develop Dr. Trinks and explains how the investigation continues” we intend to achieve two publications with our work with the Herpesvirus kind. At the moment, we have finished the job of first publication, but we continue to work with patient samples provided by the Department of Pathology “. In the future, it is thought also expand to other viruses, such as herpes type 7 human virus (HHV 7) or human T lymphotropic virus type 1 and 2 cells (HTLV 1/2) which also serve as markers of human migrations. To learn more about the research, download here the summary presented at the XI Argentine Congress of Virology. open call Currently and until July 31 can be applied to a new edition of research grants University Institute Hospital Italiano. – “Grant Dr. Enrique Beveraggi” to research related to subjects of the Clinical Cycle – “Grant Dr.

Ruben Gutman” Research related to matters of Biomedical Cycle – “Grant Dr. Enrique Caruso” to research related to Education for Health Professionals – “Grant Dr. Alejandro Ayzaguer” the oriented Clinical Research Imaging More info: http://goo. gl/uKGJMn

Genital herpes

Genital herpes is caused by herpes simplex virus. It is an acute inflammatory disease of the male or female genital tract that can result from primary or recurrent infection. sexually transmitted infection is considered. It has a high prevalence in sexually active people. Herpes classes It affects the mouth and lips. is acquired by direct contact with secretions from oral cavity come from an excretory virus. Ulcers are of variable size and number Erythematous and painful, fever, headache, malaise, rectal and vaginal secretions. skin lesions characterized by small grouped vesicles that progress to ulcerative lesions. Can be: Epidemiological: history of contact with a person who presents symptoms consistent with infection.

Clinical: presence of symptoms and signs described above. Laboratory: confirm the diagnosis of infection. What is herpes? Diagnosis: Genital herpes It affects men and women symptoms: male: -Ampoyas In the rectum or around; -Llagas That are slow to heal; Flu-like-symptoms (fever and swollen glands)

female: -blisters In the vagina or rectum or near these; -Llagas That take weeks to heal; Flu-like-symptoms. Possible long-term effects: psychological -Aflicción -can Produce a life-threatening infection in infants of mothers with the disease. Two types of herpes: Herpes simplex virus type 1: Marinelly Lopez Health 12-R Herpes simplex virus type-two

It is acquired later in life, after initiated sex and is transmitted by infected genital fluids. Clinical manifestations TRY: Tzanck test: involves scraping cells from the ulcer in order to identify infected cells, it must be done in the early days into the lesion. Oncological Cytology: The same procedure is performed, the difference is that it is done to women and cells are taken from the vaginal wall and cervix. Viral culture Virus isolation in cell culture can be performed on urine, blood, skin lesions, or mucous membranes. Clinical Picture: 3 to 7 days incubation PRIMARY INFECTION multiple vesicular lesions like papules, extremely painful.

inguinal lymphadenopathy. THEY may ulcerate Ulcer flat, painful, It disappears in 7 to 10 days Leaves no scar Effect on pregnant women If the herpes is active at the time of birth, the mother can infect her baby. The risk of transmission is higher if you caught herpes for the first time in the last months of pregnancy. The best option is to have a birth by caesarean. It is important to know that the mother can continue to breastfeed even if I have herpes. Treatment Genital herpes requires Acyclovir 400mg 3-5 times a day. It should be given psychological support patient education and companion.

Avoid having sex while genital lesions. HERPES no cure, ONLY TREATMENT Side effects Abstinence mandatory or compulsory use of condoms spontaneous abortions. premature births. The touching the affected area, also it affects the rest of the body.

HSV 1 positive igg

HSV 1 positive igg Advertising GEMINI LABORATORIES C. A. ELISA portfolio. ELISA is a procedure whose name immunosorbent assay results from the association of the initials of his name . . . http: //www. labgeminis. com/plantilla. php id_sub_seccion = 207 1-2 seroprevalence of herpes simplex antibodies in Brazil

RESUMO. To estimate anticorpos to serum prevalence of herpes simplex virus (HSV-1 and HSV-2) and analisar fatores associados not Brazil. METHODS: Estudo . . . http: //www. scielo. br/scielo. php script = sci_arttext \x26amp; pid = S0034-89102010000400017 INTERNATIONAL DIAGNÓSTICA, S. A. DE C. V.

– Company . . . IMMUNOLOGY AMPLIFICADA chemiluminescence. B12 VITROS 1-2. 100 wells. Ferritin. 100 wells. Folate. 100 wells. Folate-red cell. 50 wells. CK-MBhttp: //www.

analisisclinicosplm. com/diagnostica-internacional-sa-de-cv-16-1 Toxoplasmosis: literature review (1997 to 2000) Brazilian Society UVEÍTES. Toxoplasmose – bibliographic Levantamento from 1997 to 2000. Toxoplasmosis – Literature review (1997 to 2000) http://www. scielo. br/scielo. php? script=sci_arttext\x26amp;pid=S0004-27492001000300020 Research: Meningoencefalite | MedicinaNET 17/10/2012. Revisões Internacionais.

Enteric Infecções swerve – Nino Khetsuriani, Umesh D. Parashar . . . no anterior horn gives poliovirus spinal cord ou no . . . http: //www. medicinanet. com. br/pesquisas/meningoencefalite. htm HERPES VIRUS 1, 2, 6, 7 and 8 – coli.

usal. es INTRODUCTION. The VH are members of the family of human herpes virus among those who also found the virus varicella-zoster, cytomegalovirus and . . . http: //coli. usal. es/web/abydl/biblioteca/bibelectro . alu / documents / protocols3 / herpes / herpes. htm PATHUS – Pathology Laboratory Marker: Aplicação Diagnóstica: ACTH tumors hipófise Group: Actin muscle alpha (sm) Diferenciação smooth muscle (leiomiossarcomas, myofibroblasts .

. . http: //www. pathus. com. br/pathus2012/imunoistoquimica. html HERPES | Symptoms and tratamento »MD. Saúde Causes of genital herpes and cold sores. Symptoms do cold sores. Symptoms do genital herpes. Transmissão do herpes.

Tratamento do cold sores and herpes do genital. http: //www. mdsaude. com/2009/01/dst-herpes-labial-e-genital. html Medical Journal of Chile – High seroprevalence . . . Background: Cytomegalovirus (CMV), herpes simplex type 1 (HSV-1) and Epstein Barr virus (EBV) infections are latent persistent. Their reactivation May cause illnesses . . . http: //www.

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CT and MRI findings of human herpesvirus-associated encephalopathy 6: comparison of herpes simplex findings With

OBJECTIVE: It is Important to differentiate human herpesvirus 6 (HHV-6) -associated from herpes simplex encephalitis encephalopathy (HSE). These conditions are like, Although With regard to Involvement of the mesial temporal lobe, HSE is sensitive to acyclovir but HHV-6 encephalopathy is not. We Compared the imaging findings of the two conditions. MATERIALS AND METHODS: We Encountered eight cases of HHV-6 encephalopathy and nine cases of HSE. We divided into an observation time early, middle, and late periods defined as 0-2, 3-30 and more than 30 days from the onset of neurologic symptoms. Differences Between HHV-6 encephalopathy and HSE on CT scans in the early period and in distribution and temporal Changes in the regions AFFECTED on MR images in the three periods Were Analyzed. RESULTS: At MRI in the early and middle periods, all eight Patients With HHV-6 encephalopathy HAD exclusive Involvement of the mesial temporal lobes, and all nine Patients With HSE HAD Involvement of Both the mesial temporal lobes and the extratemporal regions (p <0. 01). Among WHO Patients underwent MRI head, six of six With HHV-6 but none of six encephalopathy With HSE HAD resolution of high signal intensity on T2-weighted and FLAIR images (p <0. 01). Among Patients WHO underwent head CT in the early period, none of the four With HHV-6 encephalopathy and six of the seven With HSE HAD abnormal findings, treats including parenchymal swelling, Decreased attenuation of affected regions, and abnormal gyral enhancement (p <0. 05) . CONCLUSION: Serial MRI Showed abnormal transient signal intensity in the mesial temporal lobes in Patients With HHV-6 encephalopathy but persistent abnormal signal intensity in the mesial temporal lobes Both the extratemporal and regions in Patients With HSE. CT in the early period Showed no abnormality in Patients With HHV-6 encephalopathy but definite abnormal findings in Patients With HSE. These Differences May be useful in the differential diagnosis of the two conditions.