genital ulcers | Header Medicine

What are genital sores? They are lesions on the skin or mucosal tissue where needed, can hurt, bleed, leaving some liquid or pus, with or hardening of the circulating area. I could have malaise, fever and swollen lymph nodes near, painful, swollen, hot. What are the causes of occurrence of genital ulcers? The causes are often infectious usually transmitted through sexual contact (STDs) or self injury from scratching or rubbing with a material that hurts or gives allergy. Sometimes genital ulcers are manifestations of disease elsewhere in the body as psoriasis, Crohn’s disease, autoimmune diseases. The extra significance of these lesions is favoring the entry of other germs sometimes more serious as the acquired immunodeficiency virus to be injured and exposed the area. The most common causes are listed below: Genital herpes virus. Symptoms include a wound that heals quickly, which makes it hurts, sometimes itchy, usually girl and accompanied by swollen glands in the area. For example, if the ulcer is of a female labia majora the right side, the right groin ganglion inflamed. Syphilis causes ulcers can disappear up to 3 months without antibiotics, although it is the patient with the disease. Usually only not give symptoms, although some pain when touched, itching, have a hard bottom and accompanied by nodes in the area.

Chancroid or chancroid is caused by a bacterium called Haemophilus ducreyi. The ulcer is very painful, releases much fluid or pus, inflamed around and are accompanied by nodes that ulcerate and often go outside of the skin and also form abscesses. Sometimes you have to drain the infected node. The inguinal granuloma is a very rare cause ulcers that take years to heal without antibiotics disease. It is transmitted by a bacterium called Donovania granulomatis. They tend to destroy tissue inhabited by their persistence. LGV is transmitted by a bacterium Chlamydia trachomatis that causes small, painful lesions disappear soon, and are accompanied by large nodes that can coalesce. There are other causes of ulcers unrelated to sexual contact as those caused by parasites such as amebiasis and cutaneous leishmaniasis. Others are ulcers inflammation of the blood vessels of Behcet’s disease, ulcers and Crohn self-inflicted injuries or ulcers. How are genital ulcers diagnosed? What concerns the patient, clinical examination, risk factors, and in some cases specific laboratory studies will help to approach a proper diagnosis. In all cases of suspected sexually transmitted diseases corresponds look for other diseases that have been infected on the same occasion. How are genital sores treated?

Treatment will differ depending on the disease that causes it. You should always take extra hygiene measures so that injuries do not hurt themselves more and not infected with other germs. In addition, you should seek medical attention to determine the cause, give specific treatment and avoid infecting them. In some cases the sores go away without treatment, but this does not mean that the disease does not follow housed in the body and affecting other organs. How preventable infectious ulcers? Care in sex for STDs. Condom use reduces the chance of infecting them, but there are skin lesions that are not covered by a condom. Contact with these infected. Anal sex is greater risk of infection because the anal area has greater increased risk of tearing, hurt and be exposed to all kinds of infections.

How to Cure Genital Herpes – Technique to cure Fast

how to cure genital herpes In this article I want to teach a technique on how to cure genital herpes quickly and without suffering, that is a very efficient and very easy to perform technique, and certainly not cause any kind of adverse effect. All you have to do is read this article and pay attention to the details of this technique to teach you. The cause of genital herpes is herpes simplex virus type 2. This is a virus that causes much discomfort, embarrassment and pain. It causes blisters appear on the genital area and reddens the whole genital area, which almost completely prevents the sex life of a person while suffering from this disease. There are people who anyway have sex, but they must be very careful in doing so, because if you do when you are having an outbreak of genital herpes most likely from becoming infected your partner or lover of this terrible disease, and that would not be good for her. So you have to be aware of what you do and not just think about your own pleasure at the cost of great suffering. Time to time, when you heal and have the chance to do so as often want without spreading. According to statistics around 500,000 people in the United States are diagnosed with genital herpes virus each year, and more than 49 million cases of this disease which is currently estimated that there are in the active and latent stage. The mode of transmission of genital herpes virus can occur in the following ways, oral, vaginal and anal intercourse, including kissing and direct contact with skin. Oral herpes can also be transmitted to the genital area and vice versa. A person who is infected with this virus can be transmitted or she himself / this virus to other parts of your body, this occurs if by touching one of the blisters and then touching or scratching other parts of your body is not he has washing hands properly.

Being a highly contagious disease must be very careful to maintain proper hand hygiene in the bathroom, and personal hygiene items such as soap, towels, razors, etc. Now I want to show a good technique on how to cure genital herpes easily, so that you begin to give battle to this virus and end this problem as soon as possible. Technical About How to Cure Genital Herpes Getting a piece of aloe vera and cotton Aloe leaf cut in half, using a knife or cutting element. Apply cotton aloe pulp and soak well. Then apply on the affected cotton and keep it there with tape area. Doing this for about 3 weeks and begin to see very favorable results. I recommend this technique on how to cure genital herpes because it is very effective and generates a relief in the affected areas, promoting healing due to the properties of aloe vera much. One important thing is to have patience as results are not immediate, everything takes time, but with good results. He thinks that by doing this, shortly you’ll be enjoying your sex life again and regain your self-esteem. Of course there is always a better and more effective way to cure genital herpes, because this is a very difficult virus to eradicate and requires a specialized method to achieve this and we will see below. The Best Way To Cure Genital Herpes Forever

There is now a completely natural and specifically developed to cure herpes and genital herpes any kind of method, and this is what Dr. Fernando Castro development, and explains in his guide Live Without Herpes. Live Without Herpes Fernando Castro It was thanks to this guide with which I myself could cure genital herpes which was suffering a long time and could not find any way to cure him. Today I am proud for having completely cured of the disease. If you truly want to learn how to cure genital herpes the best and most effective way to achieve this it is thanks to Live Without Herpes. I invite you to visit the page and acquire a copy of this guide, the results will be amazing. Visit the official website Live Without Herpes CLICKING HERE I hope this article on how to cure genital herpes you have been helpful and invite you to see the following two articles, which I’m sure will interest you much. 1- My Personal History: In this article I’ll tell you how it was that I contracted the virus and how I could cure him permanently 2- My Personal Review: In this section I give my sincere and impartial review of the book Lives Without Herpes Fernando Castro and if verada worth to learn how to cure genital herpes.

¿Ophthalmic ocular herpes or shingles …: The eternal question!


I’m sure we all know that the disease caused by ocular herpes zoster, when it affects the first branch of the trigeminal nerve (VZV -heir of childhood chickenpox) and herpes simplex (HSV – type 1 and / or 2) and its dendritic keratitis, is not the same identity, but I think not be the only one that when you have before you mess up and have to re-think and see “the big book Petete” (in my case Fisterrae) the difference between both and management. Author: Cristina Ibeas, medical PAC OSI Bidasoa. Thank you Cris! After attending one of my past guards to a young man who came from foreign body sensation, burning, tearing and pain in the left eye from 2-3 days ago regarding a recurrent herpetic corneal ulcer that had already had on other occasions I saw him again review and now I tell you. As I said, my patient had already presented the same symptoms sometimes, the diagnosis was simple and also after fluorescein staining a dendritic ulcer was drawn – Serpiginous central corneal location without injury vesicular-Crusted erythematous base and grouped typical VHZ in the eyelids, forehead, face or ipsilateral nasal wing. Operation is simple: topical treatment with acyclovir ointment application 1/4 hours sleep respecting cycloplegic eye drops + 1 drop / 6-8h and review by ophthalmologist 24-48 hours for control and monitoring, who confirmed the diagnosis, returned to revalue in twice more to finally to discharge the patient. I will only speak of keratitis in general and caused by herpes simplex. If you want more information on the VZV with ocular involvement and management, I will refer to the clinical guidelines of Fisterra which concerns him and for my taste addresses each and every one of the cases the same as for diagnostic and therapeutic indications (Guide clinic Herpes zoster and post-herpetic neuralgia – May 2011) Keratitis are defined as inflammatory processes of the cornea. Transparency and altering its curvature can affect vision. They are classified as: actinic keratoses: the most common, is the welders keratitis sicca

Keratitis foreign body: eyelashes, contact lenses . . . Keratitis systemic diseases connective tissue diseases infectious keratitis: ulcerative bacterial (Pseudomonas aeruginosa involved in contact lens wearers) and nonulcerative, viral (adenovirus, herpes simplex and zoster, EBV), fungal (Candida, Aspergillus) and parasitic (Acanthamoeba). Pain, burning sensation are the main symptoms and exploration highlights a pupil of normal appearance with little bleary tearing and discharge. Diagnosis is based on clinical history and eye examination; fluorescein staining may show erosions, stippled or dendrites (typical herpes). The primary infection usually causes herpes simplex unspecific conjunctivitis and self-limiting with discrete degree of blepharitis, and relapses usually occurs epithelial keratitis with dendritic corneal ulcer so arborescent and thin branches. Treatment of keratitis is generally based on the administration of eyedrops broad spectrum antibiotics and eye drops cycloplegic short / 6-8h to relieve pain resulting ciliary muscle spasm and prevent adhesions; and in particular in the case of herpes it is further recommended topical acyclovir ointment 5 times in / day / 2-3 weeks and control ophthalmology. BIBLIOGRAPHY Clinical Infectious keratitis Guide: 22/12/2011 Fisterra Clinical Guide Herpes zoster and post-herpetic neuralgia: Fisterra 03/05/2011) no clinical guide genital Herpes simplex: 07/26/2013 Fisterra

case report and review of the literature

Case Report Toxoplasmosis of the spinal cord in an immunocompromised patient: case report and review of the literature Medullary toxoplasmosis in an immunosuppressed patient: Case report and literature review Carolina Rodríguez * 1 Martínez2 Ernesto Guillermo Bolívar3, Sandra Edwin Sánchez4 Carrascal4 1 University del Valle. Hospital Universitario del Valle. Department of Internal Medicine. Cali, Colombia. 2 Universidad del Valle and Free University. Valle University Hospital, Department of Internal Medicine. Cali, Colombia. 3 Free University, Department of Internal Medicine. Cali, Colombia.

4 Universidad del Valle. Valle University Hospital, Department of Pathology. Cali, Colombia * Corresponding Author: E-mail address: arpicaro@hotmail. com (Rodriguez C), emarbui@gmail. com (Martinez E), lunasa12@hotmail. com (Sandra Sanchez). © 2013 Universidad del Valle. This is an open-access article distributed under the terms of the Creative Commons Attribution License, Which Permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are the Credited. Article history: Received: 22 February 2012 Received in revised form 15 March 2012, Accepted: 16 August 2013 Available online 30 December 2013 Abstract We, HEREIN, describes an HIV-positive patient With toxoplasmosis of the spinal cord.

We Also Carried out a comprehensive literature review of this topic, with emphasis on the diagnostic tools and therapeutic approach. Keywords: Spinal cord toxoplasmosis, central nervous system, myelopathy in immunocompromised patient Summary the case of an HIV seropositive patient diagnosed with spinal toxoplasmosis in conjunction with the review of the literature of the few cases to date, with emphasis on key diagnostic and therapeutic approach is presented. Keywords: Toxoplasmosis spinal cord, central nervous system, myelopathy in immunocompromised patient Introduction Toxoplasmosis is the first cause of intracranial lesions Associated to neurological deficit in HIV / AIDS¹. ACCORDING to geographic location, the frequency varies between 3 and 50% of These Patients. The risk factor strongly related to this MOST infection is CD4-positive lymphocyte count below 200 / mm3, and Especially below 50 / mm3 3-6. Described locations are extracerebral With less frequency, in less than 11% of the cases2,6, with myelitis due to Toxoplasma being an uncommon condition, with only 19 cases in medical literature, only seven of Which Have Been confirmed in living patients1,3. Herein, we present the case of an HIV-infected patient diagnosed With myelitis due to Toxoplasma confirmed by biopsy, and review the published literature reviews on this condition. Literature search in PubMed was Carried out, Medline, LILACS, and SciELO databases by using the terms: Toxoplasma, toxoplasmosis, medullary, medullary, spinal, myelitis, myelopathy. Descriptions in Spanish and Inglés Were Considered of toxoplasmosis infection due to spinal cord in adult Among Patients.

Two cases Described in French Were included. Case description: The case was presented in the Internal Medicine Service at Hospital Universitario Emergency del Valle in Cali, Colombia. Clinical data was Collected from the medical chart and signed informed consent was Obtained from the patient for publication ITS. This was a 40 year-old, Latin American, heterosexual, male patient, with history of HIV infection diagnosed seven years ago. The patient was under treatment antiretroviral. His past medical history revealed an episode of cerebral toxoplasmosis five years ago, diagnosed through positive IgG for Toxoplasma and suggestive clinical presentation and imaging scan. This former episode was Treated With standard dose of pyrimethamine and sulfadiazine and images with good clinical response, followed by prophylaxis with Trimethoprim sulfamethoxazole intermittent. The patient presented to emergency room at our hospital with a two-year history of evolution of lumbar pain of moderate to severe intensity, Associated to diminished strength in the lower limbs, more pronounced on the lower right limb, with compromise of the urinary sphincter During last months. The CD4 count was 60 cells / mm3 and the viral load was 55. 110 copies / mL. Physical exam revealed a patient in good nutritional condition, bedridden, with neurological deficit Characterized by plegia in lower right limb, with greater compromise in distal roots of L3, L4, L5 and paresis and in the lower left limb. Further exam Showed lack of bilateral Achilles and patellar reflex. Sensitivity was unaltered.

The CSF extension not suitable for exam Resulted cell count due to sample coagulation, with glucose of 6 mg / dL, proteins of 4,100 mg / dL, and LDH of 274 U / L. Magnetic resonance imaging (MRI) of thoracolumbar spine With gadolinium (Figs. 1A, 1B) Showed an expansive lesion, with affectation of the distal medullary cone, isointense on T1 to spinal cord, heterogeneous intensity, and areas of hyperintensity on T2. The lesion extended from T10 to T12 and presented peripheral enhancement in relation to contrast With a probably infectious inflammatory process, suggesting toxoplasmosis as first Possibility. Surgical exploration was Conducted of the medullary cone, finding a thickened and hardened epiconus, with arachnoid and healthy skin, a tough avascular fibrous intra-axial lesion, from Which Were samples taken. The pathological study acute vasculitis Identified With granulomatous component, extensive necrosis, and tachyzoites Compatible with toxoplasmosis (Fig. 2). Special stains and cultures for acid fast bacilli and fungi Were negative. The immunohistochemical study was positive for Toxoplasma (specific monoclonal antibody Against Toxoplasma gondii – Dako) (Fig. 3). The PCR studies in CSF for herpes simplex virus types 1 and 2, Epstein-Barr virus, Mycobacterium tuberculosis, and cytomegalovirus Were negative. Electromyography of the four limbs provided abnormal results, with electrophysiological evidence of engine and distal axonal polyneuropathy sensitivity in lower limbs. The plasma folate and vitamin B12 levels Were usual.

The patient received second-line treatment7 with Trimethoprim sulfamethoxazole at a dose of 10 mg / kg / d IV and clindamycin 1,200 mg IV every 6 hours, According to the availability in the institution, with adequate tolerance. Clinical improvement was Observed With partial recovery of neurological deficit accomplishing His Déambulation with a walker aid at eight weeks of treatment, leaving him with a sequel of a right foot drop. Discussion: vacuolar myelopathy is a common condition With medullar compromise in HIV-positive Patients, found in over 30% of autopsies prior the start of the era of antiretroviral therapy1. Other causes and possible Described broadly differential diagnoses to bear in mind include HTLV I or II, herpes simplex 1 or 2, varicella zoster, cytomegalovirus, syphilis, and tuberculosis, Among the infections; and lymphoma or nutritional deficiencies, Among the non-infectious causes6 To date, only 18 cases Have Been Described of myelitis due to Toxoplasma diagnosed histologically, via biopsy or autopsy, or through successful therapeutic trials Within the context of a consistent clinical condition (Table 1 ). From the epidemiological point of view, These Were almost all of Patients male gender (90%), Between the third and fourth decade of life. All the cases Described Have Been Associated to immunodeficiency, Which only in three of These was not related to HIV. The symptoms Described in MOST of the cases are lumbar pain, loss of function With engine Especially in lower limbs compromise (70%), bladder dysfunction (55%), specific and sensitive alteration With medullar level (75%). One patient presented Brown-Sequard syndrome. In all cases in Which to cerebrospinal fluid study was Conducted, Alterations Were found, with Increased protein levels being The most common finding, with values ​​up to 2. 2 g / dL. The Toxoplasma IgG antibody was positive in all but one of the Patients EVALUATED. Magnetic resonance imaging (MRI) With gadolinium was the preferred diagnostic imaging method, with enhanced intra-medullar solitary lesions as The most frequent findings. The Most Frequently compromised segment was the thoracic (55%) and cerebral simultaneous and medullar Involvement was established in half the cases described1,3.

All the cases Were Patients immune suppressed, with the vast majority being HIV-positive With One case Among These Suspected toxoplasmosis as a result medullary Immune Response of Inflammatory Syndrome (IRIS) 2. With regards of treatment, similar courses of antibiotics to standard Considered for Those Were Given cerebral toxoplasmosis. , According to current guidelines, Variable Obtained results are as sulfadiazine pyrimethamine With the first option, with early diagnosis factor being the best prognosis for complete recovery of These patients4. No special mention of using steroids to the Associated antibiotic regime was found, two, Although Their use in cases Have Been Described With success. In our case, steroid treatment was administered During the first eight weeks of treatment, with good relative response in Spite of the late diagnosis. More studies are needed to recommend this strategy in the future and determine the adequate manner for follow up and assessment of These Patients.  table 1 Conclusion In Spite of the few cases Described in the literature, myelitis due to Toxoplasma gondii Could be a condition more common than thought. By being a treatable disease Whose prognosis Improves With early diagnosis, toxoplasmosis must be Considered in the differential approach of all HIV-positive Patients With suggestive clinical history, presence of medullar Involvement During magnetic resonance study (especially if it is a solitary lesion), in With positive IgG antibody combination Toxoplasma. Timely treatment can result in the patient’s significant improvement. Conflict of interest: The authors declare there is not That conflict of interest That Could be perceived as prejudicing the impartiality of the information reported almost.

References 1. R Vyas, Ebright JR. Toxoplasmosis of the spinal cord in a patient With AIDS: case report and review. Clin Infect Dis. nineteen ninety six; 23 (5): 1061-5. [Links] 2. Kung DH, Hubenthal EA, JY Kwan, Shelburne SA, Goodman JC, JS Kass. Toxoplasmosis myelopathy and myopathy in an AIDS patient: a case of immune reconstitution inflammatory syndrome? Neurologist. 2011; 17 (1): 49-51. [Links]

3. Garcia-Gubern C, Fuentes R. C, Colon-Rolon L, Masdival D. Spinal cord toxoplasmosis as an unusual presentation of AIDS: case report and review of the literature. J Emerg Med 2010 inte. ; 3: 439-42. [Links] 4. Arshad S, D Skiest, Granowitz EV. Subacute onset of paralysis in a person with AIDS. AIDS Read. 2009; 19 (1): 32-5. [Links]

5. CK, Wodak J, Benson E. Spinal cord toxoplasmosis in a patient With human immunodeficiency virus infection. Int J STD AIDS 1992; 3: 366-8. [Links] 6. Singer E, Valdes-Sueiras M, Commins D, Levine A. Neurologic Presentations of AIDS. Neurol Clin. 2010; 28: 253-75. [Links] 7. Guidelines for prevention and treatment of opportunistic infections in HIV-infected adults and adolescents: Recommendations from CDC, the National Institutes of Health, and the HIV Medicine Association of the Infectious Diseases Society of America.

MMWR Morb Mortal Wkly Rep. 2009; 58: 1-198. [Links]

serological evidence of infection with equine herpesvirus types 1 and 4 in two regions of


Keywords: Department of Antioquia, Meta Department, seropositivity, ELISA, Department of Antioquia, Meta Department, frequency of soropositividade, teste Elisa, Antioquia State, ELISA test, frequency of seropositivity, Meta State Abstract: Equine herpes virus 1 and 4 (EHV-1 and EHV-4) are world wide distribution and cause serious economic losses. Primary infection with both viruses occurs in the respiratory tract, progresses through the mucosa and reach other organ systems causing abortions in the last trimester of gestation, perinatal death and unspecific neurological syndromes. EHV-1 is associated mainly with abortions, while EHV-4 is associated with respiratory disease. Affected animals recover without treatment, but remain infected throughout life. In 2001 it was reported in Colombia isolation of EHV, but to date no studies that confirm and determine the type of isolated virus is not known. The aim of this study was to conduct a serological survey to determine the presence of EHV-1 and EHV-4 in clinically healthy horses not vaccinated against EHV. To achieve our goal 139 serum samples from horses of two regions of Colombia (Antioquia and Meta) were taken, from which an indirect ELISA test was performed to detect the presence of antibodies directed against the glycoprotein G of HVE- 1 and EHV-4. positive for EHV-4 98. 7 and 96. 6% respectively in Antioquia and Meta met; for EHV-1, seropositivity was 18. 8% in the department of Antioquia and 33.

3% in Meta department. This is the first study reporting seropositivity in clinically healthy horses not vaccinated in Colombia, suggesting the presence of the virus and its establishment in the equine population of the regions evaluated, and possibly other areas of Colombia. Or equine herpesvirus type-1 and -4 (EHV-1 and EHV-4) s or you distribui agents or global and serious noxious effects causam economic monkeys. A infec or primária by both you vírus ocorre not respiratório treatment, I progredindo using da mucosa and atingir podem outros organic systems causing abortions last third or gives no deed or perinatal morte e n o-specific neurological syndromes. Or EHV-1 and associado mainly to abortions, enquanto or EHV-4 is associado to as respiratory doen. Os animais affected recover tratamento sem, more infected permanecem life. Em 2001 foi na recounted mbia Col isolamento EHV, but até agora No teve conhecimento of estudo qualquer to confirm and identify Isolados or type of herpesvirus. Or goal deste estudo foi um or perform serologic levantamento to determine presen to EHV-1 and EHV-4 cavalos clinically saudável em, No vacinadas against or EHV. To alcan ar nosso or Full-Text

Herpes Test Kit

Tags Herpes Cure Herpes, Herpes Test, Herpes Test Kit THE HERPES TEST [Embedded content] UNTIL NOW THERE WAS NO TEST THAT CAN YOU TELL IF YOUR PARTNER HAS HERPES VIRUS PRESENT A CONTAGIOUS RIGHT NOW. FOR AN INDIVIDUAL IN ORDER TO BE INFECTED WITH THE HERPES VIRUS THEY MUST COME IN CONTACT WITH A VIRUS THAT HAS ITS ABILITY TO Retained REPLICATE (A CONTAGIOUS VIRUS). THE HERPES VIRUS NOT SURVIVE LONG DOES THE BODY OUTSIDE, THUS THE HERPES VIRUS INFECTION WITH ALMOST IS ASSOCIATED WITH DIRECT invariably HUMAN PHYSICAL CONTACT.

THE VIRUS MAKES ITS HOME IN THE NERVE CELLS OF AN INFECTED INDIVIDUAL AND WHILE THERE IN THE “DORMANT” IT IS NOT CONTAGIOUS STAGE. IT CAN REMAIN IN THIS DORMANT STAGE FOR MONTHS OR YEARS AND NOT EVEN BE SPREAD TO YOUR PARTNER. AT TIMES, THE VIRUS TRAVELS ALONG THE NERVES UP TO THE SURFACE OF THE SKIN. THIS PROCESS IS Called “VIRAL SHEDDING” OR JUST “Shedding”. SOMETIMES THIS IS SHEDDING Accompanied by SYMPTOMS, LESIONS BLISTERS OR IN THE INFECTED AREA. MOST INDIVIDUALS HAVE BEEN ADVISED NOT TO ENGAGE IN SEXUAL ACTIVITY WITH THE PRESENCE OF SYMPTOMS.

DURING THE SHEDDING Replicating VIRUS IS AND IS VERY CONTAGIOUS. BUT SOMETIMES THIS Noticeable OCCURS WITHOUT ANY SYMPTOMS OR “Asymptomatic shedding”. WITH NO OBVIOUS SIGNS OR SYMPTOMS OF AN INFECTION ACTIVE IT WAS VIRTUALLY IMPOSSIBLE TO TELL WHEN YOUR PARTNER IS CONTAGIOUS UNTIL NOW . . MEDICAL SCIENCE HAS DEVELOPED SUCH AS THE PCR TESTS (Polymerase Chain Reaction) WHICH CAN DETECT MINUTE OF DNA VIRAL PARTICLES Indicating THAT MAY BE PRESENT THE VIRUS BUT THESE ARE EXPENSIVE AND CAN TEST TAKE DAYS FOR RESULTS. (KINDA KILLS THE MOMENT OF THE SPUR) I THINK WE MAY HAVE THE ANSWER THAT YOU ARE LOOKING FOR.

WHAT IS THE HERPES TEST QUESTIONS AND ANSWERS • THE HERPES TEST is the first of Its kind in home, immediate results, test for the presence of the live herpes virus. HOW IS IT DIFFERENT THAN THE TEST GIVEN BY MY DOCTOR? • YOUR DOCTOR Usually you will give a blood test to check for the presence of antibodies. This test can tell you if you are infected but can not tell you if you are contagious. your Doctor Would Have to take a culture and tell you if it Several days later Showed positive for the growth of live viruses.

There is no commercial test available to medical science to determine if Immediately you are “shedding” or “Contagious” at the moment With immediate results. THE HERPES TEST tells you right now if the live virus is present or if you are shedding the virus is present and even though you are not in a current outbreak. Most infected Individuals Know That When They are Having an outbreak They are contagious and know to avoid sexual contact. It is very unusual to know if you are in a state of asymptomatic shedding and contagious Because May there be no signs of an outbreak and you May not feel any different than When the virus is in the “inactive or dormant” stage.

During periods of “Asymptomatic shedding” you are contagious. Asymptomatic Shedding Often’ve Been called a “sub-clinical” outbreak. In other words there are no obvious sores or With an Associated irritation normally outbreak. WHAT IS SHEDDING? • When the herpes virus is not active on the surface of the skin it reside in a sleeping state inside the nerve cells and other tissues. At times (Which can not always be Predicted), the virus will Become active and travel up the nerve pathways to the surface of the skin. It is at These

When the virus times you ‘surfaced’ to the viral shedding skin That OCCURS. Viral shedding the herpes virus is When is active and “shedding” at the site of infection. The herpesvirus Considered is contagious During this “active” time and can be spread THEREFORE through direct Contact with the infected area. It is possible for the virus to Actively be shedding itself at the site of infection without showing any visible signs or symptoms. This is Referred to as “Asymptomatic Viral Shedding”. Viral Shedding Facts: •

There is lots of research still needs to be done That on viral shedding. • The chance of spreading herpes due to viral shedding is very low if you avoid direct skin to skin contact With the area When there are signs or symptoms. • If the herpes virus is shedding Actively May Not Necessarily there be any noticeable symptoms. Some people never show any signs or symptoms of herpes but can still transmit the virus to Their partner. • The herpes virus can cause severe blistering in one partner but be

totally unnoticeable in the other, even if Both partners are infected With the exact same strand of the Herpes simplex virus. • HSV Type 1 is less likely to shed than HSV Type 2 and women are Often prone to a higher rate of shedding. • It is possible for a person to carry the herpes virus without Knowing That They have it, since up to 70% of people infected With Who are HSV-2 show no signs of the infection. So it is very easy for a person to unwittingly transmit Their infection to the partner. WHERE AND WHEN WILL VIRAL SHEDDING OCCUR?


• Due to the fact That can Occur viral shedding without any signs or symptoms, Until now it has-been impossible to determine exactly When viral shedding is occurring. for This reason, there will always be a small risk of transmission. You can however determine your high risk periods and completely avoid Contact with the infected area During These Times. For People Who Outbreaks active experience, shedding is most likely to to Occur a few days before any symptoms show and as soon as there are any That warning sensations an outbreak is starting. Whenever there are noticeable symptoms, and a few days after the outbreak have healed, are high risk Present times for shedding.

Condoms can be used in Between the high risk times and medications can be taken to further reduce the risk. Researchers Viral Shedding estimate the following Statistics: • Viral shedding (that OCCURS With or Without symptoms) is Estimated to Occur in 20 to 40% of the initial six days During month period after exposure to the herpes virus. • This statistic is almost halved after a person has the virus for some ADH time, with shedding Estimated to Occur on 5 to 20% of days after the first 6 months of infection.

• In symptomatic cases, half of all herpes virus shedding is Estimated to Occur in the 7 days before and after the outbreak. • Shedding is Estimated to Occur at one time or another in all People Who With the virus are infected, but the rates are likely to shedding of vary in People with and without active Outbreaks. The rate of viral shedding Tends to vary Greatly Between the type of HSV and the location of the infection. The following is an example Estimated rates of shedding based on current research: In genital infections: •

HSV Type 1 = 3 to 5% (of days EVALUATED) • HSV Type 2 = 15 to 20% (of days EVALUATED) In oral (mouth) infections: • HSV Type 1 = 18% (of days EVALUATED) • HSV Type 2 = 1% (of days EVALUATED) Note That the rates of shedding Tend to be higher When the Herpes simplex virus type OCCURS in its “typical” or preferred location of infection, HSV-1: such as cold sores or genital herpes HSV-2. What can I do to help reduce the risk of Asymptomatic Viral Shedding?

There are things that you ‘can do to reduce (but not Eliminate) the risk of herpes viral shedding. • Avoid contact with the infected area a few days before, During and After any symptoms are noticed. • Use latex condoms in between to help protect Against Outbreaks shedding. • Antiviral Medications and some herbs, Olive Leaf: such as, Have Been proven to help reduce herpes viral shedding, helping to lower (but not Eliminate) the risk of transmission. It is definitely possible To have a long term relationship and not spread herpes.

In fact, one study of couples WHO avoided sexual contact During Recurrences found That, over 12 months, only 1 out of 10 passed the virus on to Their partner. In That study, Were not used condoms. Using condoms May reduces the risk of infection even further. HOW DOES THE HERPES TEST HELP ME? • Most infected Individuals know to abstain from sexual contact During Outbreaks obvious but can not determine if They are at the present time shedding. When you are shedding you are contagious. (The virus is present) THE HERPES TEST Detects the presence of the live virus shedding or an Individual infected THUS

would know to abstain from sex if They Knew They Were shedding. What are the side effects of HERPES THE TEST? • THE HERPES TEST has-been thoroughly tested and found to be completely safe. May, Although the solution tested stain the area for a few days, it is harmless and easy to remove with soap and water . . Early clinical trials Reported That fewer than 3 per 10,000 applications Individuals experienced an allergic reaction to the activated solution at the test site, Which Were Easily resolved with a mild topical steroid cream. A skin sensitivity test can be done prior to

Patients use on skin Especially sensitive to allergies, Quickly How will I see results? • Results are immediate. Photo’s below show positive tests. The shedding site will fluoresce Within a minute or two after Applying solution and using special UV light provided your test kit With. SEE ATTACHED PICTURES fluorescence Observable HOW DOES THE HERPES TEST WORK? • The live herpes virus is stained With the solution Easily provided in your kit and always will fluoresce in the presence of the special UV light

provided. Dead viruses will not stain and THUS will not fluoresce in the presence of the same UV light. No Means the live virus fluorescence is not THUS present and you are not contagious. Some fungus and other materials is May Also fluoresce THUS to false positive test is possible but is esta occurrence rare. In other words it is possible to live viruses are present Indicate When They are not but the opposite is true Also no fluorescence That means no live viruses. What exactly is the herpes virus? •

For more information acerca herpes viruses, Please visit the Understanding Herpes section on the pull down tab of the Home page. I’m not sure I have herpes. • It is Important to be professionally diagnosed by a health care professional. If you don ‘t have the herpes virus active in your body, esta test you will simply confirm testing physician. It is always Suggested that you ‘ contact your physician prior to purchasing HERPES THE TEST. I have just Conducted my HERPES TEST and my injuries did not fluoresce. What Should I do?

• Have fun tonight! Do You Currently Have Herpes (Polls)

Garlic lip | VICE | Spain

The first thing Amaia of Kokoshca told me that he preferred to talk is nonsense because the talks music and data overwhelm and off. All right. The first thing Inaki de Kokoshca told me he likes the magazine VICE because it always hides a lucid message. Well also. Saturday they are traveling with his group to Madrid for the official presentation of Singles Club with Microcosmos and Courage. That day I want to watch the concert so we have already discussed everything in advance. Vice: Is there anything that you worry lately? Iñaki: I think that for cold sores is best to use natural remedies, such as garlic rub the affected area. Amaia: The herpes never heal, once you have slept always there . . . waiting for you to slacken. And that to me but spare me even garlic in the food.

I: Well, garlic is fundamental. A: Garlic is the king of food I: Exactly. A: Everything is better with garlic. Kisses dragon. I: In fact part of the Spanish Court Corella moved to a village in the south of Navarre, to cure tuberculosis queen with its garlic. The queen died. But garlic is a natural antibiotic. I you are aturullando. I: I wanted to talk about Moon Duo. A: Moon Duo have gone to Corella to eat garlic and cure your herpes. I: Moon Duo can hide behind his big beard herpes. It must be horrible to have herpes.

I already have enough to bite my lip. I: It’s a very erotic biting the lips gesture. For example Paz de la Huerta biting his lips. A: I like the wounds on the lips. The dry, chapped lips have always liked. Sometimes I crack itself. A: Cracks of those that do not let you smile? After they swell and are more beautiful. I: I knelt things in the gum. Inaki you scare me. Q: But I do not say a fakir plan. A: The pain that gives you the opening of the crack rocks. I: Slightly.


Although I see more harmful bite his lip. A: The pain is sometimes joyful. The only pleasant pain is small bruises. Tighten there. A: Remove scabs! I: I Am scratching the scabs. But if you rascáis the scabs will be scars. I: Yes. A: The scars are not ugly. My chicken pox I have many memories. I: My pediatrician and ripped me a scab neighbor in the elevator and I still have the mark. I spent that same eye doctor. I was injured with an old machine and I still have the scar.

A: Scarring all. I always have bruises. I’m clumsy and I stick with it. I: I’m nearsighted. A: I have astigmatism. I: Look, Amaia has a nice history of glasses. She pretended to look bad. Tell Amaia. A: I love the glasses since childhood. My mother took me to the eye doctor and pretended. I said wrong aposta letters to put me glasses. They put me and I got to see evil. Now I need but barely use.

I spent years with them. If I take them off at first I left me strange eyes. A: Snoopy Eyes. Chiquiticos. You clothe yourselves quite normal. He did not get to Navarra Basque technical clothing? A: In the Basque Country it is fashionable to wear hair like predator of the movie. I: angular Hairs. A: Cut with axes. They dress as if they were to Everest to go for the bread. I: Ciclos Iturgaiz know. A: That clothing is very expensive indeed. And very ugly.

With how well dressed the Basques in the 70s and 80s. I: Like your father Amaia. How they are worn by the Basques in the seventies? A: As a modern baleen Barcelona jersey and wool bunting. My father wore great. He loves the Basque Country. Now you dress like Steve Jobs. A friend of Steve Jobs dressed in carnival but instead put one black jersey beige. Le mistook the protagonist of Mar Adentro. I: Hahaha. A: Hahaha. Another very important thing what are you going to eat today? I: Lentils and tomato salad.

A: I like my parents. My mother made red beans. I do not know how to cook. I like to cook me. I feel sorry for you: A: Why? Because you’re not going to eat my mashed zucchini. It will not be easy, but try to be happy without it. A: It will try.

Dermatology 007: Virus Diseases (Group 3)

Virus Diseases The viruses are easily spread diseases caused by viruses. These pathogens are very small, so tiny living things that can only be seen with electron microscopes, which can infect cells and cause infections. They multiply with extraordinary rapidity. The virus according to the tissue that attack, are classified as: Dermatrópicos, if they affect the skin. Pneumotrópicos, if they affect the lungs. Neurotropic, if they affect the nervous system. Viscerotropic, if they affect the viscera and internal organs. Viruses have different ways of being transmitted, some do it through the air, such as influenza, mumps, smallpox, chickenpox. The rabies virus is transmitted by saliva of an infected animal. Others are transmitted by mosquito bites, such as those that produce yellow fever and dengue hemorrhagic fever. Others do it through water or contaminated food.

Viral infections affecting the skin are produced by several of these microorganisms, the rash diseases are viral loads, which are manifested in the skin secondarily, so we will deal with injuries that initially manifested in it. These viruses are produced by:  Herpes virus (herpes simplex I and II and varicella – zoster)  Pox virus (molluscum).  Papovavirus (warts and genital warts). SIMPLE AND Zoster The lesions caused by herpes simplex type I, mainly affect mucous membranes and skin of the face, trunk and upper extremities; primary infection takes place in the early years of life can be in the fingers (herpetic whitlow) or the oral mucosa, with a severe acute illness, characterized by edema and erythema of gums, severe pain and blistering, accompanied of lymphadenopathy. The pictures are repetitive, since the virus is housed in a ganglion of cranial nerves or the spinal dorsal roots and topography which is to be presented is the closest to that node site; there needs to be a situation in which the immune balance of the individual is altered, as happens concomitantly fevers of various origins, prolonged sun exposure, intake of immunosuppressive drugs or other debilitating disease. Therefore the frequency of occurrence of the injury will depend on the patient and the conditions that are conducive, so be subintrantes, monthly or once or twice per year. The clinical picture is very characteristic: Erythema, edema and transparent vesicles clustered in an area of ​​2 or 3 cm, that rupture and release their exudate leads to the formation of serohemáticas crusts, accompanied by sensation of paresthesia, pain and burning, a5 lasts 3 days, is self-limiting and relapsing. The ideal treatment is acyclovir at doses of 200 mg c / 5hrs / 5days, in the early hours of evolution; treatment is repeated or prolonged, depending on the frequency of dermatoses. It can also be used as a symptomatic treatment. drying fomentation and antiviral cream.

MOLLUSCUM CONTAGIOSUM. It is a common skin disease, caused by a poxvirus (described are the types I and II) mainly affects children, in any topography, but predominates in extremities. It consists of umbilication whitish papules, a few millimeters, which tend to spread quickly if the patient manipulates and are asymptomatic. The treatment is carried out with the removal of the lesion by a lancet; if they are numerous, retinoic acid may be applied once a day, as it is expected will produce secondary eczematisation and injuries that persist are removed. When present in genital region, usually in adults, is considered a sexually transmitted disease. In immunocompromised patients, these neoplasms are larger and can have tumor-like.

FDA Approves Groundbreaking New Treatment That Gives Cancer Tumors Killer Case of Herpes

Image via (cc) Flickr user Oriol Salvador In what’s being called a major advance toward “a complete change in the game,” the FDA has for the first time Given the green light to a new form of cancer treatment-One That Researchers hope Might someday make chemotherapy, and Its adverse side effects , a thing of the past. The key, as it turns out, is-more viruses Specifically, herpes. Last week the FDA approval of ITS Announced to “oncolytic herpes genetically modified live virus therapy” in Which viruses are engineered to hunt and kill Specifically the malignant cells Associated With the skin cancer melanoma. The treatment-talimogene laherparepvec ( “T-VEC”) – will be sold under the brand name Imlygic by BioVex Inc. and Its parent company, Amgen. Reportedly this is the first instance of a viral cancer treatment’s approval by the agency. Explains an FDA release: Image via (cc) Flickr user mr_d_logan Imlygic [. . . ] is used to treat melanoma lesions can not be removed That completely by surgery.

Imlygic is injected into the lesions Directly melanoma, Where It replicates inside cancer cells and causes the cells to rupture and die. A treatment course Imlygic With Consist of a series of injections into the melanoma lesions. After the initial injection, a second dose is administered three weeks later, followed by additional doses every two weeks for six months at Least, UNLESS other treatment is required or there are no remaining Until injectable to treat lesions. What makes herpes virus esta particularly special is unique ITS genetic engineering, Which causes it to infect malignant cells while ostensibly healthy Avoiding ones. In other words, it targets cancer, with minimal risk of giving herpes to patient, as we know it Commonly (, Although, the FDA warns, some risk still remains for Those With suppressed immune systems, or are pregnant women WHO). And while the agency does list Several other potential side effects,: such as fever, chills, and “flu-like symptoms,” these seem mild Compared to the draining effect of current cancer treatments. An article published in the esta past May Journal of Clinical Oncology describe the results of a large round of clinical testing of the procedure. It Concludes That using the modified herpes virus did, in fact, shrink tumors and Patients Afford to marginally longer lifespan. The results Were not groundbreaking-this is a treatment, not a cure-and but as the study’s authors write, “T-VEC dealer to a potential novel therapy for metastatic melanoma Patients With. ” Beyond melanoma, however, see the FDA’s Researchers approval of Imlygic as an Important milestone when it comes to using smart viruses to treat other forms of cancer. Ultimately, the goal for many physicians is to end the need for physically-and oftentimes psychologically-trying therapies: such as chemo or radiation. As oncolytic virotherapy Mayo Clinic researcher Dr.

Stephen Russell Explained to The Guardian, the FDA’s approval of Imlygic opens the door to further research into the use of viruses to target diseased cells. I says: “We can not prematurely That We’ve Achieved claim our ultimate goal, Because We Have not; this really is a single step along That path. But it’s a Very Important and very significant step. ” Herpes, as it turns out, Could help save your life someday. [Via The Guardian, ars technica]

herpes simplex

herpes simplex Concept: infectious disease, viral cause, by Herpes Simplex virus types I and II. Relapse. Etiology Herpes simplex virus types I and II. triggers: sun, stress, trauma, menstruation, and caquectizantes immunosuppressive diseases, HIV / AIDS, etc. Clinical picture: • Injury eritematovesiculosas grouped in a cluster, evolutionarily crusts are formed by drying the vesicles. approximately 7 days. There may be regional lymphadenopathy. • Itching, burning, sometimes prodromal symptoms also. • Frequent Locations: fingers, around the mouth, back, buttocks, genitals. Differential diagnoses: Acute contact dermatitis, Herpes Zoster, Varicella

Date: 07/29/10 Owner: Virtual University of Health 9 elements herpes simplex Herpes simplex in woman 46 years old Date: 01/01/80 Owner: Virtual University of Health Views: 10255 herpes simplex Herpes simplex in woman 46 years old Date: 01/01/80 Owner: Virtual University of Health Views: 3759

herpes simplex Herpes simplex in woman 46 years old Date: 01/01/80 Owner: Virtual University of Health Views: 3257 Herpes Simplex Type I Herpes Simplex Type I, woman 24 years Date: 01/31/07 Owner: Virtual University of Health Views: 3259 Herpes Simplex Type I, woman 24 years Herpes simplex type I, woman 24 years Date: 01/31/07

Owner: Virtual University of Health Views: 3781 Herpes Simplex Type II, women 52 years Herpes Simplex Type II, 52-year Date: 06/08/10 Owner: Virtual University of Health Views: 5179 Herpes Simplex Type II, women 52 years Herpes simplex type II, 52 year old woman Date: 06/08/10 Owner: Virtual University of Health Views: 5816 Herpes Simplex, teenager 16 years

Herpes simplex, 16 years old Date: 07/20/10 Owner: Virtual University of Health Views: 3063 Herpes Simplex, adoslescente 16 years Herpes simplex, 16 years old Date: 07/20/10 Owner: Virtual University of Health Views: 3179