AIDS

HSV Eraser Protocol
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AIDS stands for acquired immunodeficiency syndrome. It was first recognized in the USA in 1981, being isolated the HIV virus in 1983 and 1984 demonstrating that this was the causative agent of AIDS. The virus is a retrovirus from a family of lentiviruses. In 1985 an analysis of the enzyme-linked immunosorbent assay (ELISA) that allows detection was developed. From then until now AIDS has become a pandemic. According to the CDC, in the United States estimate that from 800,000 to 900,000 people are living with HIV and AIDS. Until June 2000, a total of 753. 907 AIDS cases have been reported to CDC. Based on the calculations of the AIDS program of the United Nations (UN AIDS), approximately 47 million people have been infected with HIV since the start of the global epidemic. Until December 2000, it is estimated to have killed about 21. 8 million children and adults, and some 36. 1 million people are living with HIV infection or AIDS. UN-AIDS estimated that in 2000 there were 5.

3 million new HIV infections. This represents almost 16,000 new cases a day. Some 3. 0 million adults and children died of HIV / AIDS in 2000. When we consider that an HIV-infected person has AIDS? In two situations: a) When contracting any disease marker (Table 1) b) All patients with <200 CD4 / ul have AIDS regardless of whether or not marker disease (Table 2). Table No. 1: Clinical categories HIV infection Category A: one or more of the following tables in adolescent or adult with HIV infection Category. B: appearance in HIV + pictures not C with one of the following criteria: HIV attributed to boxes or clinical course complicated by treatments or HIV Category C: marker Diseases. AIDS surveillance case asymptomatic HIV infection bacillary angiomatosis Candidiasis of trachea, bronchi or lungs Persistent generalized lymphadenopathy oropharyngeal candidiasis esophageal candidiasis Acute primary HIV infection Persistent vulvovaginal candidiasis invasive cervical cancer cervical dysplasia / carcinoma in situ Coccidioidomycosis disseminated or extrapulmonary Consumptive syndromes (fever or diarrhea +1 month) extrapulmonary cryptococcosis peripheral neuropathy PML Oral hairy leukoplakia Cryptosporidiosis chronic intestinal (> 1 month duration) Shingles> 2 episodes in two different dermatomes Cytomegalovirus disease (no liver, spleen or lymph nodes) Idiopathic thrombocytopenic purpura Recurrent pneumonia listeriosis Wasting syndrome related to HIV Pelvic inflammatory disease

Herpes simplex (ulcers> 1 month or bronchitis, pneumonia or esophagitis) Pneumocystis carinii pneumonia related to HIV encephalopathy Recurrent Salmonella septicemia cerebral toxoplasmosis histoplasmosis chronic intestinal Isosporiasis Kaposi’s sarcoma Burkitt lymphoma primary brain lymphoma M. avium complex Tuberculosis

Table No. 2: Classification system according to revised CD4 levels in 1993 CD4 A Asymptomatic acute B Symptomatic not A or C AIDS Disease marker C 500 / ul A1 B1 * C1 200- 499 / ul A2 B2

* C2 <200 / ul * A3 * B3 * C3 How long does it take for HIV to cause AIDS ? : Since 1992, scientists have calculated that about half of people with HIV develop AIDS within 10 years of infection. This time varies greatly from person to person and can depend on many factors including the health of the individual and their behavior. etiologic agent It is a retrovirus which belongs to a subfamily lentivirus. The four known human retroviruses are: a) T lymphotropic virus human HTL and HTL-V1-V2; b) HIV HIV-1 and HIV-2 which are cytopathic viruses. The most frequently identified is HIV-1. Electron microscopy icosahedral virion form provided with numerous projections formed by the two major proteins gp120 and gp41 transmembrane outer shell observed; genomic RNA, the reverse transcriptase enzyme, p18 of the inner membrane and the core protein p24. Transmission HIV is transmitted: 1) through sexual contact with an infected person 2) sharing needles or syringes with an infected person 3) through contaminated blood transfusions or clotting factors contaminated 4) the children of mothers carrying the HIV become infected before or during birth or being breastfed. 5) health workers infected accidents puncture syringes containing blood infected with HIV, or after the infected blood comes into contact with an open wound or mucous membrane (eg eyes or inside the nose ). According to the CDC research they have been conducted with more than 22,000 patients of 63 physicians, dentists and infected with HIV and have not detected other cases of this type of transmission in the United States surgeons. Pathophysiology and Pathogenesis The main feature of HIV infection is immunodeficiency caused by the progressive, quantitative and qualitative deficit of T lymphocytes, which have on their surface the CD4 molecule that functions as the main receptor for HIV after which the infection occurs and destruction direct these cells. To that HIV can penetrate the target cell must also exist CD4, co-receptors belonging to the family of seven transmembrane receptors coupled to protein G. When the level of CD4 cells drops below a certain level the patient is at risk of a number of opportunistic illnesses that define AIDS. Diagnosis
Diagnosis is based on the demonstration of anti HIV Ac, of any component or both. Ac anti HIV often appear in the circulation of 4 to 8 weeks after infection. Conventional test is the ELISA has a sensitivity of 99. 5% but its specificity is low, the results are expressed as: 1) Positive (intense reation) Negative (no reaction) or Indeterminate (partial response). Due to the low specificity when we have a + or inconclusive ELISA should another test: Western blot. A “quick test” to detect antibodies against HIV is a screening test that produces very quick results, generally in 5-30 minutes. In comparison, test results of screening antibodies against HIV most commonly used, the ELISA (enzyme linked immunosorbent assay), is not available for 1-2 weeks. The only rapid HIV test currently approved by the FDA for use in the United States is the “Single Use Diagnostic System for HIV-1 (SUDS)” prepared by Murex. The availability of this test may be different from place to place. The results of the rapid HIV test are considered as accurate as the ELISA results. Both the rapid test and the ELISA seek the presence of HIV antibodies in the blood. Like all screening tests (including ELISA), a result of HIV reagent must be confirmed before it can be given a diagnosis of infection. ELISA addition, other tests now available include:

· The radioimmunoprecipitation assay (RIPA): A confirmatory analysis of blood that can be used when antibody levels are very low or difficult to detect or when the results of the Western blot test are uncertain. An expensive test, the RIPA requires time and expertise to perform. · The rapid latex agglutination test: A blood analysis simplified, low cost can be useful for medically disadvantaged areas where there is a high prevalence of HIV infection. · The test immunobinding dot-blot: A quick blood test screening that is effective in terms of costs and that can become an alternative to conventional ELISA and Western blotting. · The test apprehensions p24 antigen: Also known as the apprehension of HIV-1 antigens. This blood was added as an interim measure by the Food and Drug Administration (FDA for its acronym in English) in 1996 to protect the blood supply further until other tests are available to detect HIV infection early before antibodies are fully developed. Since an activity of p24 antigen is unpredictable, this test is not useful in helping people to determine their HIV status. · The polymerase chain reaction (PCR): A specialized blood analysis seeking genetic information of HIV. Although expensive and laborious, the test can detect the virus even in someone recently infected. To further protect the blood supply, the FDA has indicated the development and execution of tests for HIV genetic material as CPR. Of high sensitivity, there are two forms: DNA PCR and RNA PCR. DNA PCR can be used to make the diagnosis of HIV infection poviral amplifying the DNA. RNA PCR has been used to monitor changes in the levels of plasma HIV genome but can be used for early diagnosis.

, It is positive in 98% of patients and detected up to 40 copies / ml HIV RNA. How long to wait for HIV test after possibly being exposed to the virus? The tests commonly used to detect HIV looks for antibodies produced by the body to fight HIV in the blood. Most people develop these antibodies detectable before a period of 3 months after infection, the average being 25 days. In rare cases, it can take up to 6 months. For this reason, the CDC currently recommends getting tested 6 months after the last possible exposure (oral vaginal, anal, or unprotected, or sharing needles). It is rare that the body takes more than 6 months to develop detectable antibodies. It is important that during the 6 months between exposure and testing, one to protect yourself and others from other possible exposures to HIV. Clinical manifestations The clinical manifestations of HIV infection extend from an acute syndrome that occurs associated with primary infection through prolonged until the phase advanced disease asymptomatic state. HIV disease can be divided empirically according to the degree of immune deficiency: early stage: CD4> 500 / ul intermediate stage: 200- 500 CD4 / ul

Advanced stage <200 / ul Most opportunistic infections and malignancies AIDS-defining appear in advanced stages of the disease, while neurological disease and Kaposi are not strictly related to the level of immunodeficiency. The CDC classifies HIV disease in: Group I: S. acute associated with HIV Group II: asymptomatic infection Group III: persistent generalized lymphadenopathy Group IV: Other diseases A: Constitutional disease B: Neurological disease C: Secondary Infectious Diseases D: Secondary Neoplasms E: Other processes Acute HIV syndrome Up to 70% of HIV patients with acute clinical syndrome 3 to 6 weeks after primary infection. Clinical manifestations of it are: general neurologic dermatological gastrointestinal respiratory Fever 97% Meningitis maculopapular erythematous rash thrush Cough Pharyngitis 73% Encephalitis mucocutaneous ulcers Sickness Lymphadenopathy 77% peripheral neuropathy vomiting Headache / retro-orbital pain myelopathy Diarrhea Arthralgia / myalgia 58% Lethargy / discomfort Anorexia / weight loss The appearance of these maifestaciones coincides with an outbreak of plasma viremia and p24 antigenemia. Symptoms usually last from one to several weeks and gradually refer to as the immune response to HIV develops and plasma levels fall viremia. Laboratory At first decreases the total number of lymphocytes and all T cell populations (CD4 and CD8), then a reversal of the CD4 / CD8 ratio occurs due to the elevation of the latter. The number of CD8 may remain elevated or standardized but CD4 levels usually remain somewhat diminished. lymphadenopathy appear in 70% of individuals with primary infection. Most people recover spontaneously from this syndrome and have a slight decline in CD4 remain stable for a period until they begin to decline gradually. In people with acute HIV syndrome anti HIV1 Ac by ELISA detected within the first few weeks of onset of symptoms. P24 antigen can be detected 24 hours after onset of symptoms. In a series p24 it was detected in 13 patients from whom samples were obtained 18 days after the onset of symptoms. differential diagnosis The major differential diagnosis of acute HIV syndrome are: Mononucleosis Epstein Barr Mononucleosis Cytomegalovirus toxoplasmosis Rubella viral hepatitis secondary syphilis disseminated gonococcal infection Primary herpes simplex infection Drug reaction The factors that differentiate one Epstein Barr mononucleosis are a SAHIV Acute HIV syndrome Mononucleosis Epstein Barr acute onset insidious onset Tonsillar hypertrophy little or no Tonsillar hypertrophy important Enanthem on the hard palate Enanthem in hard and soft palate exudative pharyngitis is rare exudative pharyngitis is common Mucocutaneous ulcers are common It does not mucocutaneous ulcers Jaundice is rare Jaundice (8%) Diarrhea may no diarrhea Rash is common Rash is rare The poor prognostic factors at the time or after acute HIV syndrome are: Clinical A- Virologic B- Immunological C- Asymptomatic Stadium. Clinical latency The time between initial infection and the first clinical manifestations of the disease varies, the average is 10 years. HIV disease with active virus replication often progresses during this asymptomatic period. The rate of progression of the disease is directly related to the levels of HIV RNA. Those with high levels of viral RNA progress faster. CD4 progressively decrease during this period at a rate of 50 cells per year when the number of CD4 is <200 / ul immunodeficiency state is such that the patient is at risk of developing opportunistic infections and secondary malignancies.

AIDS

HSV Eraser Protocol
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AIDS is Acquired Immune Deficiency Syndrome, is a viral disease that affects the human immune system, damaging the body’s defense capacity. Without resistance, a person with AIDS is susceptible to various diseases and infections. The disease was first diagnosed and 1981 in the United States, and Argentina in 1982. Syndrome: Set of manifestations (symptoms) that They characterize a disease. Immuno: Related to the defense system of our body. Deficiency: Indicates that the defense system does not work or malfunctioning. Acquired: That is acquired. It is not congenital or hereditary. The cause of AIDS is an infectious agent, the HIV virus (HIV), which is introduced into the human body in different ways and housed in cells of the immune system, weakening it. This virus is an extremely small particle (0.

0001 mm. ), Which live need to be introduced inside a cell. HIV has the distinction of attack lymphocytes are the directors of our defense system. Initially HIV remains dormant, ie, “asleep” within lymphocytes. In some cases, after a time, often years, for reasons not yet well determined, the virus is activated, ie “wakes up” and begins to destroy the cells. Thus, progressively weakens the immune system to destroy it, the system is blocked and the body remains helpless and at risk of contracting any disease from a simple cold to more serious cancers, and all possible infections which It leads to death in a more or less long term. HIV Virus How is HIV transmitted? It can be transmitted by 3 way: Blood: Through the sharing of needles and syringes and / or any sharp object. During pregnancy and childbirth by uncontrolled blood transfusions.

Sexual transmission: Sexual intercourse can transmit HIV, whether homosexual or heterosexual. The passage of the virus performed through microscopic lesions or wounds. Perinatal transmission: An HIV carrier women can transmit the infection to her baby during pregnancy, childbirth or breastfeeding. This risk can be reduced substantially if the woman is treated early during pregnancy and avoid giving nursing her baby. Since a person becomes infected with HIV until AIDS develops usually elapse between 6 and 10 years. The study of the evolution of the disease can be performed through various laboratory markers or based on the sequence of appearance of different clinical manifestations. Among the biochemical markers is usually considered the decline in CD4 T lymphocyte count has, until relatively recently, been the main reference for cataloging the stage of evolution of the disease.

Since 1996, the determination of the amount of virus circulating in the blood of an infected person, who is called viral load has become the most important marker of disease progression. About three weeks after HIV infection, most patients experience flu symptoms pseudo as fever, headache, rash, lymphadenopathy and feeling of discomfort. These manifestations disappear after one or two weeks. During this phase, called acute phase of infection, HIV multiplies at high speed, suffering various genetic mutations. At first, a drop in the number of CD4 T lymphocytes occurs but, soon after, normal numbers are retrieved in response to an activation of the immune system. During this stage individuals are highly contagious. PREVENTION: In sexual intercourse condom use is the most effective way to prevent the spread of the virus and other sexually transmitted diseases (STDs): syphilis, gonorrhea, herpes, etc. PREVENTING BLOOD: People who for reasons of habit or medication must be injected frequently must use disposable syringes and needles, because this route is the most common form of transmission. Infected people should not, under any circumstances, donate blood or organs. Blood transfusions should be made in establishments that meet the standards of control and prevention force. SIGNS AND SYMPTOMS Early signs and symptoms may, in some cases, start being evident between six months and one year after infection by HIV, depending mainly on physiological varieties because each individual has different susceptibility to developing the disease, in some cases, start to appear even 5 or 10 years after infection The main signs and symptoms are listed below:

Fever. There may be a type of intermittent fever and persistent character. It lasts for at least two weeks and is accompanied by chills. Lymphadenopathy. People with AIDS have lymph nodes under the arms, neck and English. They can be painful or not. Generalized malaise. Profound fatigue and lethargy, which can last about two weeks, without apparently a cause for it. This restricts make above normal as climbing stairs, dressing or talking activities. Weightloss. An unexpected and severe weight loss that can be up to 10% of the total weight of a person, without due to diet or exercise. This factor is accompanied by a severe loss of appetite. AIDS in Central Africa, also occurs syndrome thinning or “slim disease”, causing people a radical weight loss.

night sweats. very copious and persistent night sweats, contributing to weight loss and dehydration. Diarrhea. Essentially chronic, severe, persistent and often bloody. This causes anemia, weight loss, electrolyte imbalance which is the continuous loss of body fluid. blood disorders. Tendency to have skin bruising, bleeding spontaneously and in abundance for no reason. This causes the pallor of the patient. Respiratory problems. There are chronic, dry, tight, persistent cough that can last more than two weeks. Skin problems. The AIDS patient can show on your skin a range of lesions, which are usually painless but are equipped with a purple or pink that increase in size and spread over many parts of the body. Lesions in the mouth.

They are formed on the tongue and the mucosa in the form of thick white stain. It can also appear in the throat. It is produced by a fungus known as Candida albicans and is very often found in young children born with AIDS. There may also be herpes lesions for more than four weeks, causing difficulties in swallowing food. Delay healing. The body loses its ability to partially tissue recovery. Any injury takes much longer to heal. infectious processes. Any infection, however simple it may be, will be much harder to control in people with AIDS. Problems in the external genitalia. Virus herpes lesions that last longer than a month. psychological and neurological problems. There may be multiple signs and symptoms of this type as a result of major neurological impairment that occurs in patients with AIDS.


Usually they are: -Apoplejía Or loss of consciousness and paralysis in various parts of the body. Loss of feeling in some parts of the body. Loss of physical and mental ability. -Temblores And shudder. Balance and coordination problems. Trouble behavioral and personality. Anxiety, fear, panic, depression, sadness, alignment and character and personality changes. -Alucinaciones. False sense of perception. Loss of memory, disorientation, headaches and blurred vision. The most serious disease in AIDS patients can be classified into two groups: Opportunistic infections a-

b-Some cancers to. Opportunistic infections are serious because the body can not stop its development spontaneously without treatment form, and sometimes despite treatment, can lead to death. When the immunity of an organism is deficient, these microbes called opportunists seize the opportunity to invade and cause serious infections. Its manifestations vary by microbe affected organ. Although the overall condition is weight loss, fatigue and weakness, these are the basic features. b. Some cancers are very common in the course of this disease. -The Kaposi’s sarcoma is the most common, affecting 35% of patients with AIDS. They are plaques or skin nodules blue or brown. These lesions are not only found in the skin but in lymph nodes, lungs and almost all the viscera. -The Lymphatic lymphomas are malignant tumors that usually occur more sick people with AIDS than in people without the disease. DEMONSTRATIONS

prolonged cough and fever Pneumocystis carinii Digestive tube chronic diarrhea Infection in the esophagus Invasion of fungi in the digestive system Paralysis Vision problems rare psychic manifestations Meningitis cryptococcal Toxoplasma

cutaneous herpes Herpesvirus 8. DIAGNOSIS HIV infection is diagnosed by detecting antibodies to the virus in the blood. It is clear that HIV tests are not entirely safe after infection because the body takes several weeks to develop these antibodies (6 to 12 weeks). The person will be asked for a blood test called ELISA (Enzyme-linked inmunsorbent assay). This test must be repeated if positive. If the test is positive again, another test called a Western blot to confirm the reservation. The Western blot test confirms the presence of HIV proteins in the blood. The Western blot test is important to avoid false positives. The person will receive an HIV diagnosis only if the 3 tests are positive. If the person is diagnosed with AIDS, the doctor will also ask for a viral load test blood.

This measures the amount of virus in the blood. Generally people with high viral loads generally have a poorer than those with a lower viral load outcome. Viral loads are also used to decide when to start or change treatment. Regional HIV and AIDS statistics, end of 2008 Adults and children They are living with HIV / AIDS Adults and children newly infected Adults often * deaths adults and children Sub-Saharan Africa 22400000

1900000 1. 4 million North Africa and Middle East South and Southeast Asia 3800000 Eastern Asia Latin America 20 million Eastern Europe and Central Asia 15 million Western and Central Europe North America

1. 4 million Total world 33400000 2. 7 million 20 million 9. TREATMENT At present there are about 15 drugs that are being used in the treatment of HIV infection. Treatment includes a combination of several antiretroviral drugs that prevent the immune deterioration and suppress viral replication. Antiretroviral therapy (ART) is complex because it involves the administration of at least three drugs (triple therapy) with a high number of shots and pills per day, which produce adverse effects, interact with other drugs and that must be taken in the presence or absence of food. With these drugs reduced the progression of the disease and the onset of opportunistic infections, which has achieved a remarkable reduction in mortality and hospital admissions of HIV-positive patients is achieved.

In developed countries, one can say that AIDS has become a chronic disease, and although currently incurable no longer mortal. conclusions Today practically everyone knows what is AIDS, what their consequences and how contracts. However this knowledge is not enough to stop the advance of the disease. Education and prevention are essential tools to combat evil and official campaigns are incomplete, limited and ineffective, education activities should go beyond a simple TV advertising: they must be aimed at each audience, with languages ​​and particular codes. In addition, campaigns in schools and custom work is needed. It is not desparramar fear, but to teach protected by respect for life. QUOTES FOR REFLECTION The only vaccine that protects us is prevention. Our behavior will depend on our future without AIDS or AIDS. We do not take care of people, let us beware of AIDS. Medical consultation for any reason is always confidential The HIV-infected individual shows no sign that sets you apart from not infected.

Anyone, even you. , Can be infected by AIDS. From you. It depends. AIDS does not discriminate, not discriminate us who suffer. AIDS has infected most accomplished among those who thought: “Me not going to happen. ” None of us can or should remain oblivious to the problem. The drug, prostitution and alcohol are fertile grounds for AIDS. Nobody gets AIDS because of the other. Bibliography:

AIDS

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Kaposi’s sarcoma In addition to the non-HIV-associated Kaposi’s sarcoma (see sarcomas), there is the HIV-induced sarcoma, which occurs in about a third of AIDS patients. symptoms: In the field of skin lesions are pinkish-bluish spots, purple-reddish indurated plaques and dark brownish or bluish tumor nodules. The older of these tumor nodules often have a yellow-green border as a hematoma. Intraoral the hard palate is infested by symmetrical blue and red knots most frequently. Regional lymph nodes may also be affected. In advanced stages occur gastrointestinal metastases and pulmonary infiltrates. Diagnosis: Histological examination of an excised node. Therapy: By cytostatic chemotherapy a temporary improvement is possible. The immune deficiency is amplified thereby.

Non-Hodgkin’s lymphoma The HIV-associated lymphomas differ from the other non-Hodgkin lymphomas (see sarcomas) in that predominantly young patients are affected. In addition to the involvement of the lymph is also extranoduläre locations in the bone marrow, central nervous system and gastrointestinal tract as well as in the oral cavity, lungs and liver. Diagnosis: Histological examination of an excised node. Therapy: By cytostatic chemotherapy and radiation is hardly an improvement to achieve. Consequences for the dental practice: In HIV suspicious oral manifestations and in persons at risk (homosexuals, prostitutes, drug addicts, hemophiliacs) recommendation of an evaluation of the situation by HIV testing. In case a patient from it, to undergo an HIV test, so it may still not be refused the necessary dental treatment. The usual disinfection and sterilization measures are sufficient for precise application to prevent transmission of HIV to healthy patients. Wearing protective gloves during treatment to avoid contact with blood or saliva. Avoid injury by instruments that are contaminated with blood or saliva.

Wearing protective glasses and a mouthguard with the use of spray-cooled grinding and drilling instruments and ultrasonic dental cleanings as protection against spray mist. Patients who are known as HIV carriers should be treated at the end of the consultation. After treatment, a thorough surface and room disinfection is required.

AIDS

HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews

Kaposi’s sarcoma In addition to the non-HIV-associated Kaposi’s sarcoma (see sarcomas), there is the HIV-induced sarcoma, which occurs in about a third of AIDS patients. symptoms: In the field of skin lesions are pinkish-bluish spots, purple-reddish indurated plaques and dark brownish or bluish tumor nodules. The older of these tumor nodules often have a yellow-green border as a hematoma. Intraoral the hard palate is infested by symmetrical blue and red knots most frequently. Regional lymph nodes may also be affected. In advanced stages occur gastrointestinal metastases and pulmonary infiltrates. Diagnosis: Histological examination of an excised node. Therapy: By cytostatic chemotherapy a temporary improvement is possible. The immune deficiency is amplified thereby.

Non-Hodgkin’s lymphoma The HIV-associated lymphomas differ from the other non-Hodgkin lymphomas (see sarcomas) in that predominantly young patients are affected. In addition to the involvement of the lymph is also extranoduläre locations in the bone marrow, central nervous system and gastrointestinal tract as well as in the oral cavity, lungs and liver. Diagnosis: Histological examination of an excised node. Therapy: By cytostatic chemotherapy and radiation is hardly an improvement to achieve. Consequences for the dental practice: In HIV suspicious oral manifestations and in persons at risk (homosexuals, prostitutes, drug addicts, hemophiliacs) recommendation of an evaluation of the situation by HIV testing. In case a patient from it, to undergo an HIV test, so it may still not be refused the necessary dental treatment. The usual disinfection and sterilization measures are sufficient for precise application to prevent transmission of HIV to healthy patients. Wearing protective gloves during treatment to avoid contact with blood or saliva. Avoid injury by instruments that are contaminated with blood or saliva.

Wearing protective glasses and a mouthguard with the use of spray-cooled grinding and drilling instruments and ultrasonic dental cleanings as protection against spray mist. Patients who are known as HIV carriers should be treated at the end of the consultation. After treatment, a thorough surface and room disinfection is required.

AIDS

HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews

From Wikipedia, the free encyclopedia Acquired Immune Deficiency Syndrome, also Acquired immunodeficiency syndrome (English for “acquired immune deficiency syndrome”), usually abbreviated as AIDS or AIDS, denotes a specific combination of symptoms in humans as a result of by infection with the human immunodeficiency virus (HIV virus, HIV ) induced destruction of the immune system occur. [1] In the patients it comes to life-threatening opportunistic infections and tumors. AIDS was recognized on December 1, 1981 as a distinct disease. According to UNAIDS lived in 2014 world about 36. 9 million HIV-positive people. 2014 there were about 2 million new HIV infections (d. H. Approximately 5,600 new infections per day), and about 1. 2 million people died as a result of HIV / AIDS. [2] Since the epidemic began, 36 million people died from AIDS. The proportion of HIV-infected people is the worldwide average at about 0. 8% of 15- to 49-year-olds, however, achieved in individual African States values ​​by 25%.

[3] The effective treatment of HIV-infected people with new drugs is AIDS has become rare in Central Europe. [4] Turned out solved from an immune cell HIV The red ribbon as a symbol of solidarity with HIV positive and AIDS-sick people Definition and Classification Classification in the ICD-10 Classification according to ICD-10 B20 Infectious and parasitic diseases due to HIV disease B20. 0 Mycobacterial infections related to HIV disease (Z.

B. Tuberculosis HIV disease resulting) B20. 1 Other bacterial infections due to HIV disease B20. 2 Cytomegalovirus HIV disease resulting B20. 3 Other viral infections due to HIV disease B20. 4

Candidiasis HIV disease resulting B20. 5 Other mycoses HIV disease resulting B20. 6 Pneumocystis pneumonia HIV disease resulting B20. 7 Several infections related to HIV disease B20. 8 Other infectious and parasitic diseases due to HIV disease

B20. 9 Unspecified infectious or parasitic disease due to HIV disease B21 Malignant neoplasms HIV disease resulting B21. 0 Kaposi’s sarcoma due to HIV disease B21. 1 Burkitt’s lymphoma HIV disease resulting B21. 2

Other types of non-Hodgkin’s lymphoma HIV disease resulting B21. 3 Other malignant neoplasms of lymphoid, haematopoietic and related tissue due to HIV disease B21. 7 Multiple malignant neoplasms HIV disease resulting B21. 8 Other malignant neoplasms HIV disease resulting B21. 9 Unspecified malignant neoplasms HIV disease resulting


B22 Other specified diseases due to HIV disease B22. 0 Encephalopathy HIV disease resulting B22. 1 Lymphoid interstitial pneumonia due to HIV disease B22. 2 Cachexia syndrome due to HIV disease B22. 7

Several diseases classified elsewhere HIV disease resulting B23 Other disease states due to HIV disease B23. 0 Acute HIV infection syndrome B23. 1 (Persistent) generalized lymphadenopathy HIV disease resulting B23. 2 Blood and immune abnormalities HIV disease resulting, not elsewhere classified B23.

8 Other specified disease states due to HIV disease B24 Unspecified HIV disease ICD-10 online (WHO version 2016) In the most recognized medical diagnosis classification system ICD-10 of the WHO, both the HIV-infection as well as numerous diseases can be encoded as a result of HIV infection. The exact nature of the sequela is encrypted in the updated last in 2013 by the World Health Organization version of ICD-10 in a fourth location, for example: B20. 6 Pneumocystis pneumonia HIV disease resulting. Classification of HIV infection and AIDS definition CDC definition According to case definitions of the US Centers for Disease Control and Prevention (CDC), a confirmed positive HIV test is considered proof of HIV infection. [5] The assessment of HIV infection on the degree of severity including AIDS was in 2008 changed significantly , Until then, clinical findings and laboratory values ​​were classified separately and presented as a letter-number code. Here [6] were asymptomatic and involved with HIV related but not AIDS-defining illnesses.

Since 2008, only the AIDS-defining illnesses are still considered. The division on the number of CD4 + T lymphocytes (helper T cells) and the list of AIDS-defining illnesses, however, remained unchanged. The in practice still widespread letter-number coding is thus not active. It should also be noted that the case definition is primarily of epidemiological surveillance of HIV and AIDS and not as a guide for clinical diagnostics. So can not be lowered again a case classification; she says therefore possibly nothing about the current status of a patient. HIV classification in adults and adolescents * [5] stage CD4 + cell counts per microliter in percent all lymphocytes 1 ≥ 500

> 29% and no AIDS-defining illness 2 200-499 14-28% and no AIDS-defining illness 3 (AIDS) <200  two within a year) Progressive multifocal leukoencephalopathy Salmonella septicemia, recurrent tuberculosis Toxoplasmosis, cerebral Wasting Syndrome (HIV-related) Cervical cancer, invasive European definition According to case the European Commission definition, a confirmed positive HIV test is considered proof of HIV infection. [8] [9] A review of HIV infection on the degree of severity is not made. As AIDS is considered proof of HIV infection and one of the European case definition for AIDS established diseases. These are identical to those of the CDC's formation identical (see above). An AIDS diagnosis solely on the basis of laboratory results without any clinical findings (CDC stage 3) was discarded. [10] The European case definition used for the epidemiological surveillance of HIV and AIDS. WHO definition According to the WHO case definition a confirmed positive HIV test is considered proof of HIV infection. [11] Immunological and clinical HIV-classification in adults and adolescents * [11] stage symptoms CD4 + cell count per microliter % Of all lymphocytes 1 Asymptomatic ≥ 500 Asymptomatic disease (s) 2 Mild symptoms 350-499 defined for this stage disease, z. B. Herpes Zoster 3 advanced symptoms <350 defined for this stage disease, eg. , oral Haarzellleukoplakie 4 (AIDS) severe symptoms <200

AIDS

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Behavior and actions after contamination – 1. After punctures, cuts first provocation of strong bleeding, then disinfection. In splash injury eye: rinse thoroughly with water for. For more traumatic injury immediate prospect of a surgical emergency. – 2. blood for HIV testing and for the blood test decrease. – 3. When contamination with HIV-positive material (or a correspondingly high probability) is today recommended that prophylactic administration of azidothymidine (AZT): within 30 minutes after the injury first capsule AZT 250 mg, then daily for 14 days 5 250 capsules mg. (4stündliche administration, night keep free). Contraindications: pregnancy Important are the accurate documentation and the enlightening discussion with the physician (occupational physician). Further checks of HIV status for at least 1 year is necessary.

Antiretroviral therapy There is no consensus on when to start antiretroviral therapy: – Therapy immediately after an infection regardless of the stage of disease or – Therapy only in symptomatic patients or patients start with a high viral load. There is general agreement on the objective of preventing the progression of asymptomatic HIV infection as long as possible and also to begin therapy before irreversible damage of the immune system have occurred. The chosen therapy to suppress virus replication maximum. This is also important in order to prevent development of resistance. A reduction in the morbidity and mortality can already by lowering the viral load by about 1 – 2 log10 achieve. The selection of resistant virus mutants can be long-lasting but only be avoided if HIV replication is possible completely inhibited. This requires a high antiviral activity of the drug combination used. Combination therapy is recommended: When selecting the initial drug combinations other factors such as particular way of life, comorbidity, and other necessary therapies to consider excluding viral load and disease stage. For an effective initial treatment, a number of options available.

These options are as follows: combination of a – Protease Inhibitors (PI) with two nucleoside reverse transcriptase inhibitors (NRTI) – Combination of a non-nucleoside reverse transcriptase inhibitor (NNRTI) with two NRTIs – Combination of three NRTIs (See also anti-infective Overview: Antivirals) Checkups during this therapy: – CD4 cell count for each month – Virus concentration determination 2 times before starting treatment, then monthly until reaching the target size (preferably below the detection limit), then every 2-3 months – The focus is on the control and prophylaxis of opportunistic infections and neoplasms Treatment of opportunistic infections Candidiasis (oral cavity and esophagus) – Fluconazole 50-100 mg / day

– Ketoconazole 2x 200 mg / day – Then pause or conversion to non-absorbable preparations, eg amphotericin solution or lozenge or miconazole. Cryptococcosis (meningitis) – Amphotericin B 0. 1 mg / kg / day, increasing to 0. 5 (-0. 6) mg / kg / day in combination with flucytosine 150 mg / kg / day, total of 6 weeks maintenance treatment: – fluconazole 100-400 mg lifetime to prevent relapse – According to recent releases good effect of monotherapy with fluconazole. Good diffusion in the cerebrospinal fluid and elimination in unchanged form in the urine Cytomegalovirus (- retinitis) – Ganciclovir 3x 5 mg / kg body weight and day for 2-3 weeks (The intestinal CMV infection is treated the same way). Because of the myelotoxic effect of ganciclovir possibly concurrent AZT administration settle. Blood samples

– Alternatively, if necessary intolerance of ganciclovir foscarnet (DNA polymerase inhibitor): 3x daily infusion of 60 mg / kg for 3 weeks. (Caution: nephrotoxicity – dose reduction) Because of nephrotoxicity appropriate laboratory tests Permanent ophthalmologic monitoring of patients is absolutely necessary Zoster and herpes simplex Aciclovir 5x 200 mg / day for 5 days or famciclovir. In ophthalmic zoster possibly double dose (i. v. or oral). Ophthalmological controls Atypical Mycobacterial Infection) (M. avium, M. kansasii)

Pathogens are largely resistant to conventional antituberculosis. In vitro rifampicin derivative ansamycin appears to be effective. Result therapeutic studies with combination treatments (ansamycin + ethambutol + Prothionamid (PTH) + gyrase inhibitor) remains to be seen Pneumocystis carinii pneumonia (PCP) – Method of choice is co-trimoxazole. Dosage: 20 mg trimethoprim + 100mg sulfamethoxazole / kg / day in 3-4 doses in 500 ml of physiological NaCl solution as an infusion. Duration 2-3 weeks At the same time daily 15-45 mg folinic acid depending on the blood count. In severe hypoxia (O2 <60 mm Hg) is the simultaneous administration of prednisolone (200-1000 mg / day) helpful - Alternative treatment options are (less effective) Clindamycin 2400 mg / day of primaquine + - 15 mg / day for 3 weeks, or clarithromycin Preventive and postpneumonische PCP prophylaxis Every 4 weeks inhalation with 300 mg pentamidine. In addition, per week 1 tablet combination preparation pyrimethamine-sulfadoxine Cerebral toxoplasmosis - Pyrimethamine 4x 25 mg / day for several days, then 25-50 mg + clindamycin 2. 4 g daily for 3 weeks - Pyrimethamine + sulfadiazine 4x 1 g daily for 2 weeks. Given folinic acid 15-45 mg In Suilfonamid allergy possibly 50 mg pyrimethamine daily (+ folinic acid) prophylaxis 2 x 1 tablet sulfonamide combination preparation Tuberculosis (M. tuberculosis) Both pulmonary and b

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AIDS and its treatment by Chinese Herbal Medicine © 1996 by Gunter R. Neeb In the beginning of the eighties, on patients with severe immune deficiencies a new virus which found by showing did most of prosthesis patients had antibodies against this virus. Nowadays this virus is known in two types, called Human Immunedeficiency Virus Type 1 and 2 (HIV-1 and HIV-2) of Which the first type is more common in the western world. It belongs to a subgroup of the retrovirus-Class, the Lentovirus-Group, Which lead only slowly to disease after infect Their host, seeking Causing a persisiting infection Which remains inactive for a long time. The symptomeless stage may take 10 Years or more, Which to aproximate maximum of 15 years afterwhich most patients show symptoms of mortal immune-deficiency. The virus’ genome besteht of RNA All which is copied to a complementary RNA-string by the enzyme reverse transcriptase. After the first flu-like acute infection in Which a high viral titer can be seen, the virus has killed and infected many CD4 T-Cells and macrophages. The reaction immunic besteht of antibodies, CD-4 and Helpercells -inflammative cells, as well as of cytotoxic CD 8-Cells. After this acute phase the blood is Effectively frred from the virus with a few circulating CD 4-Cells carrying one copy of the inactive genome of the virus. Although the peripheral blood contains no viruses during the inactive phase, the virus itself Reproduces Actively in the lymphatic tissue, brain, and someother regions.

this chronic infection Gradually destroys the CD4 T-Cells I n in a not yet completely Call explained ‘way. After almost all CD-4 T-Cells are destroyed, i. e. if Their count is below 200 per ul, the imunedeficiency stage is reached, and the syndrome is called AIDS. Gradually lymphatic folliculary dendritic cells are damaged leading to a loss of lymphatic follicles and thereafter a complete destruction of the lymphatic tissue. A similar process destroys the thymusstroma. If a new acute infection with HIV-1 or HIV-2 is Suspected or early enough detected, Several antivirus herbs from Chinese Medicine can be prescribed to secure a low infection rate. Since the toxicity of some is very low, They Can be used even as a preventive measure in order to keep the virus from spreading. One promising candidate among them is “zi hua di ding” or viola yedoensis (Pls. Refer to the Following table). Chinese medical herbs Affecting various viruses: List 1 name

Chinese name Effects on Virus / toxicity or LD50 Viola yedoyensis Zi hua di ding HIV / low toxicity arcticum lappa Niu ban zi HIV / low tox. Andrographis paniculata * Chuan xi lian HIV, ECH011 / 13. 4 g / kg Lithospermum rythrorhizon

Zi cao HIV, Jinke, flu, polio, hepatitis / 681. 13mg / kg Altemanthera philoxeroides Lung xin lian zi cao HIV, flu, encephalitis B. retrovirus rabies virus / 455. 5g / kg Lonicera japonica Jin yin hua HIV, flu (PR8), herpes / orphan (mice) 53g / kg Coptis chinensis Huang-lian

HIV, hepatitis B RuS, Newcastle disease / 24. 3mg / kg Epimedium grandiflorum Yin yang huo HIV, polio, ECHO 6. 9, Coxsackie A9, B4, B5 / 36g / kg Woodwardia unigemmata Guo ji zhong guan jue HIV, flu (PR8, Jinke 68-1,57-4, NewA1, Lee, C1232, D), England. II, polio II, Coxsackie, herpes simplex, encephalitis / 1. 7g / kg (mice) Prunella vulgaris Xia ku cao

HIV / low toxicity Senecio scandens Qian li guang HIV / 302. 6g / kg *** Hypericum japonicum Di he cao HIV, hepatitis B / no obvious toxicity Scutellaria baicalensis Huang qin HIV, flu PR8, (Asian A-flu) Xiantai, rhinovirus 17, adenovirus 7 / 3. 081g / kg * Baphicacanthis folium

Da qing ye flu, mumps, encephalitis B / no obvious toxicity Baphicacanthis rhizoma, radix Ban lan gen flu, hepatitis A, B, mumps, herpes encephalitis B. / no obvious toxicity Bupleuri radix Chai hu flu, smallpox / 4. 7g / kg gland (mice) Ledebouriellae radix Fang feng flu, Columbia SK

Polygonum cuspidatum Hu zhang herpes, ECHO9, ECHO11, adenovirus, hepatitis B. , flu, encephalitis B. Coxsackie A, B, polio II / 1363mg / kg * Forsythiae fructus Lian qiao flu (Asian A), nose-17 / 29. 37g / kg (mice) Taraxaci herba Pu gong ying ECHO11, herpes / 156. 3g / kg

* LD50 is given in the doses of chemical scents of the herb. ** LD50 is given in the doses of alcohol extract, IV injection. *** LD50 is given dependent on the herb produced in China; in some countries this herb is very toxic (Geissman, 1964). There are 20 herbs listed in Table 1. The antiviral spectrum and toxicities of each herb are given. Thirteen of them have inhibitory effects on the HIV. Some others have antivirus effects on other viruses. The HIV virus’ ability to avoid immunity response has much in common with some of thesis viruses: The permanent and latent infection is therefore seen in the Epstein-Barr virus and the herpes simplex virus; the antigenic variation by mutations can be found as well in trypanosomes, the influenza virus and Streptococcus some kinds. The induction of an immune-supression shares the HIV virus withsome Staphylococcus kinds and the Mycobacterium leprae. From an allopathic view, it may not be proper to list thesis herbs until theyhave been tested in vivo against the HIV virus; HOWEVER, from a TCM view, if Certain herbs have been proven effective against a Certain virus in clinical trials, the same mechanism may work against another virus in the human body. TCM holds did any herb or drug taken into the body must first affect the body’s metabolism so it can more did Effectively fight the pathogen. Although the concept of an antivirus is an allopathic approach, TCM’s principle of Treating febrile infectious diseases (qing re jie du, heat-clearing and toxin-Eliminating) with herbal remedies can give this approach a new meaning.

The toxicity of thesis herbs is very low, so They Can be used safely for a long period, even over a lifetime, by HIV-infected persons or, as Mentioned before as a preventive measure. The 13 herbs Which Showed inhibitory effects on HIV in vitro may therefore contribute to the treatment of various complications of AIDS. These patients are known to be subject of opportunistic infections and some kinds of cancer. Those are Caused by organisms Which Usually can be controlled Easily by the immune system, like Pneumocystis carinii pneumonia Causing, as well as E. G. Toxoplasma, the Cytomegaly virusand some Mycobacteria like M. tuberculosis and even some normally harmless kinds like M. avium. The functioning of the cellular defese is Necessary for at effective control of microorganisms thesis. Some patients (about 20%) is therefore affected by organisms, Which are Usually controlled by the humoral defense system like Haemophilus influenzae. THEREFORE mostimportant are probably Those herbs Which stimulate the growth of the T-lymphocytes as listed below: Common name asparagus (root)

atractylodes, white (rhizome) Coix (seed) Coriolus (sclerotium) epimedium (leaves) ganoderma (wholesale) lentinus (wholesale) ligustrum (fruit) ginseng (root) phaseolus, white (bean) huang-jing (rhizome) Besides AIDS Patients are liable to develope tumors like the Kaposi’s Syndrome, a slowly developing Usually proliferation of Bloodvessel cells, and to tumors of the B-Cells, the non-Hodgekin-Lymphomes and such of the brain. This might be explainable by a lacking T-cell response against tumor cells or a chronic stimulation of the B cells, another feature of HIV-infections. Since many herbs in Chinese Medicine havebeen proven to stimulate the immune system or some parts of it, thesis herbs can therefore be used in any stage of the HIV infection, i.

e. When An opportunistic infection the occurs or gene rally to stimulate the immune system. Pls. refer to list 2 below: Chinese medical herbs Affecting the immune system (list 2) Promoting lymphoblast transformation Botanical name Angilica sinensis, acutiloba Astragalus membranaceus Atractylodes macrocephala Codonopsis pilosula Coix lachryma-jobi Coriolus versicolor

Epimedium grandiflorum Ganoderma lucidum Gelatin equi asini Ligustrum lucidum Panax ginseng Phaseolus vulgaris Polygonatum sibiricum Polygonum multiflorum Increasing the Number of White Blood Cells Botanical name Acanthopanax senticosus Astragalus membranaceus Cinnamomum cassia


Codonopsis pilosula Comus officinalis Ganoderma lucidum Gelatin Equi asini Ligustrum lucidum Millettia dielsiana Panax ginseng Phaseolus vulgaris Placenta Homines sapientis Psoralea corylifolia Increasing phagocytosis of neutrophilic WBC Botanical Name Common Name Astragalus membranaceus Huangqi (root)

Atractylodes macrocephala atractylodes, white Dioscorea opposita dioscorea (root) Glycyrrhiza uralensis licorice (root) Panax ginseng ginseng (root) Increasing the Number of Mononucleic Macrophages Botanical Name Common Name Coriolus versicolor Coriolus (sclerotium) Glycyrrhiza uralensis licorice (root) Lentinus edodes lentinus (hole) Promoting Phagocytosis of Macrophages Mononucleic Botanical Name Common Name Acanthopanax senticosus ginseng, Siberian (root) Angilica sinensis, acutiloba tang-kuei (root)

Astragalus membranaceus Huangqi (root) Atractylodes macrocephala atractylodes, white (rhizome) Codonopsis pilosula codonopsis (root) Epimedium grandiflorum Epimedium (leaf) Eucommia Eucommia (bark) Ganoderma lucidum ganoderma, lingzhi (wholesale) Lentinus edodes lentinus (wholesale) Panax ginseng ginseng (root) Polyporus umbellatus polyporus (sclerotium) Psoralea Corylifolia psoralea (seed) Rehmannia glutinosa rehmannia (root) II. Herbs Affecting Non-Specific Humoral Immunities

Inducing the Production of Interferon Botanical Name Common Name Astragalus membranaceus astragalus (root) Phaseolus vulgaris (PHA) phaseolus, white (bean) (Ie Astragalus can promote the production of interferon by viral stimulations. ) Anti-Complementary Activities Botanical Name Common Name Cinnamon cassia cinnamon (twigs) Lentinus edodes lentinus (wholesale) (These herbs have triggering effects on the C3 complementary. ) III.

Herbs Affecting Specific humoral Immunities Those Promoting Hypertrophy of antigen-combining Cells in Mice obsessions at the Early Stage of the immuno-reaction Botanical Name Common Name Angilica sinensis tang-kuei (root) Astragalus membranaceus astragalus (root) Coix lachryma-jobi Coix (seed) Comus officinalis cornus (fruit) Those Promoting Hypertrophy of Antibody-Producing Cells Botanical Name Common Name Asparagus cochinchinensis asparagus (root) Coix lachryma-jobi Coix (seed) Coriolus versicolor Coriolus (sclerotium) Epimedium grandiflorum epimedium (leaves)

Ganoderma Lucidum ganoderma (wholesale) Ligustrum lucidum ligustrum (fruit) Ophiopogon japonica Ophiopogon (root) Polygonatum sibiricum huang-jing (rhizome) Polyporus umbellatus polyporus (sclerotium) Psoralea Corylifolia psoralea (seed) Those Suppressing Hypertrophy of Antibody-Producing Cells Botanical Name Common Name Glycyrrhiza uralensis licorice (root) Those Regulating Hypertrophy of Antibody-Producing Cells Botanical Name Common Name Astragalus membranaceus astragalus (root) Those Increasing Antibody Production

Botanical Name Common Name Astragalus membranaceus Huangqi (root) Coriolus versicolor Coriolus (sclerotium) Epimedium grandiflorum epimedium (leaves) Lentinus edodes lentinus (wholesale) Panaxginseng ginseng (root) Placenta hominis sapientis placenta, human Polygonum multiflorum he-shou-wu (wholesale) Rehmannia glutinosa rehmannia (root) Those Suppressing Antibody Production Botanical Name Common Name Angelica sinensis tang-kuei (root) Glycyrrhiza uralensis licorice (root)

Psoralea Corylifolia psoralea (seed) Zizyphus jujuba jujube (seed) IV. Those Herbs Affecting the Production of Different Types of immunoglobulin (Ig) Herbs Which Affect immunoglobulin (Ig) Promotive Suppressive IgG Lentinus edodes Psoralea Corylifolia Astragalus membranaceus IgA placenta hominis Psoralea Corylifolia (Serum) Rehmannia glutinosa IgA Astragalus membranaceus (Secretion) Ganoderma lucidum Polygoni multi floristic

Epimedium grandiflora floristic IgM Astragalus membranaceus Psoralea Corylifolia Coriolus versicolor IgE Astragalus membrananceus Because of the disruption of the immune system, various allergic reactions can be seen in AIDS and HIV (+) patients, most notably hay fever and allergic sinusitis. The Following list contains herbs Which can be used for different types of allergic conditions: List 3: Herbs Affecting Allergic Reactions Herbs Affecting Type I Allergic Reactions Those Suppressing the secretion of histamines Botanical Name Common Name Ganoderma Lucidum ganoderma (sclerotium) Those Providing relief from bronchial spasms due to histamines and Acetylecholine Botanical Name Common Name

Coriolus versicolor Coriolus (sclerotium) Ganoderma Lucidum ganoderma (sclerotium) Epimedium grandiflorum epimedium (leaves) Placenta hominis sapientis placenta, human Psoralea Corylifolia psoralea (seed) Those Providing Relief of Gastrointestinal Smooth Muscle Spasms Caused by Histamine and Acetylecholine Botanical Name Common Name Angelica sinensis tang-kuei (root) Cinnamomi cortex cinnamon (twigs) Comus officinalis cornus (fruit) Glycyrrhiza uralensis licorice (root) Those Which Suppress Allergic Shock or Allergic Skin Reactions Caused by Foreign protein Botanical Name Common Name

Glycyrrhiza uralensis licorice (root) Panax ginseng ginseng (root) Placenta hominis sapientis placenta, human II. Those Affecting Type II Allergic Reactions (Cytolytic) Those Preventing ABO Hemolysis Botanical Name Common Name Glycyrrhiza uralensis licorice (root) Those Increasing the Number of Platelets Botanical Name Common Name Glycyrrhiza uralensis licorice (root) Placenta hominis sapientis placenta, human Rehmannia glutinosa rehmannia (root)

Those Increasing the Number of Red Blood Cells Botanical Name Common Name Acanthopanax senticosus ginseng, Siberian (root) Codonopsis pilosula codonopsis (root) Equi asini equine gelatine Panax ginseng ginseng (root) (These herbs can therefore affect hemolysis. ) III. Those Affecting Type III Allergic Reactions Botanical Name Common Name Astragalus membranaceus astragalus (root) Glyzyrhiza uralensis licorice (root)

Rehmanniae radix rehmannia (root) IV. Those Affecting Type IV Allergic Reaction (Delayed) Botanical Name Common Name Angelica sinensis tang-kuei (root) Bombyx batryticatus silkworm (wholesale) Glycyrrhiza uralentsis licorice (root) As Mentioned above some kinds of cancer can Occur positive in HIV patients. Since there are many herbs in TCM proven effective against various kinds of cancer, only to be listed here excerpt can, since this would exceed this topic by far. So finally in List 4 are listed only herbs Those Which may be effective against cancers in typical AIDS patients, namely leukemia and lymphoma. Since some of thesis autumn are toxic or cytistatic Themselves, They Should not be used Before They are Indicated. List 4: Herbs for cancers in AIDS patients (late stage) Botanical Name Chinese Name

Polysticticum yun zhi Hedyotis diffusa bai hua she she cao Indigo naturalis qing dai Catharanthi rosei chang chun hua Lysimachiaiae Clethroidis zhen zhu cai Botanical Name Chinese Name Camptothecae Acuminatae xi shu Sarcandra glabra zhong jie feng Curcuma aromatica / zedoaria e zhu Brucea javanica ya dan zi Of thesis herbs the first one in the list, Polystipticum is one of the most interesting herbs, since its toxicity is very low, while it is useful for leukemia, lymphoma and liver cancer, by inhibiting the synthesis of the DNA and RNA of tumor cells , Besides this it antagonizes the immunosuppression Caused by cyclophosphamides and cortisone, while Increasing the amount of macrophages, Promotes phagocytosis and Enhances the Board of blastogenesis and rosette forming, seeking being a promising agent for Both enhancing the immune system and Suppressing tumor developement. At the moment clinical western medicine hasnt yet found any solutions exept medicaments like AZT, Which inhibit the function of the reverse transcriptase and THEREFORE have a toxic effect on human cells as well. Some other proposals are searching to find specific inhibitors of the viral protease or the developement of antibodies for the virus surface or synthezising substances Which ought to block the docking of gp120 to the CD4.

But They are quiet far from ready to be used in clinical practice. THEREFORE Traditional Chinese Medicine might Provide some useful alternative by combining herbs with western medicaments or even use them alone in different stages of HIV-Positive patients and other types of immune deficiency syndromes.

AIDS

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By Jeb Breithaupt of JEB Design / Build People with eye bags or dark circles can look older or tired. Même si cette infection est assez banal et sans risque, elle reste pas moins n’en inesthétique et légèrement douloureuse. We are a nationwide STD testing service did is an alternative to a free STD clinic. , Not everyone’s cold sore is triggered by the same thing. During the winter season Benthic, usually, the lips become so dry thatthey tend to crack and may start bleeding sometimes. And of the two, color can make the bigger difference in the way your roof looks. One of the major causes is aging. Bouton de fièvre: Les symptômes For more savings, we Provide A number of discounted STD testing packages did bundle popular lab tests together. Mijn eigen arts had he veel meer understood en begrip voor ze schreef me pijnstillers (diclofenac, 20 stuks. It is therefore common to have stuffy nose or have headaches in the early morning hours. Excessive cigarette smoking so results in dryness of your lips did may lead to a cut lip Once you’ve settled on a good-quality shingle, consider thesis guidelines for choosing a color:. Heredity Il n’existe pas vraiment de traitement pour l’soulager herpès labial et il n’y a aucun moyen pour guérir les boutons de fièvre.

When choosing who to give your business to, there are some things you shoulderstand consider. ! Oliver will most likely recommend desensitizing toothpaste to help reduce the pain, a fluoride treatment to strengthen the enamel, or sealing any receding gums. HOWEVER, if the cut on the lip is deep, there are chances of bacterial infection. Shutters shoulderstand coordinate. Stress un blanc liquid jaunâtre épais s’écoule of vésicules, We respond Quickly to e-mails, online orders, and messages. ), Maar ook dit deed te veel pijn. Visit us today and welcome the season with a smile! Keep it there for a few minutes. It might be fun to spice up the look of your house with fire-engine red shutters and shingles, but you might deterministic potential buyers who would rather have something more subtle on top of the house. Underlying Medical Conditions Bouton de fièvre: Un compagnon pour la vie cytomegalovirus HIV disease resulting in B20. 3 This is another great cut on lip treatment in a natural manner.

The good news is did shingle manufacturers have Introduced products embedded with reflective granules in a variety of light and mixed colors, not just in white. Revitol so Seems to work better on different types of skin Compared to its cheaper counterparts. Candidiasis HIV disease resulting in B20. 5 Other mycoses HIV disease resulting answered by G M on May 14, 2013 2:19:59 6 Click here to learn more about Revitol Eye Cream. B20. 7 Several infections related to HIV disease B20. Keep in mind though did tea tree oil being, potent, can burn your skin. Other infectious and parasitic diseases due to HIV disease Keep Yourself Hydrated. 9 Unspecified infectious or parasitic disease HIV disease resulting in malignant neoplasms B21 HIV disease resulting answered by A S On Jan 24, 2012 12:53:29 0 For adults this is a full seven to eight hours a night. B21. 1

Burkitt lymphoma HIV disease resulting in B21. Apply some aloe vera gel to your chapped lips. Other types of non-Hodgkin’s lymphoma Extra pillow HIV disease resulting Other little things you can do include using on so you do not sleep flat on your back. 3 other malignant neoplasms of lymphoid, haematopoietic and related tissue due to HIV disease B21. 7 Do this once a day for about five days for best results. B21. Cold compresses help reduce puffiness. Other malignant neoplasms HIV disease resulting in B21. 9 Unspecified malignant neoplasms HIV disease resulting white ulcers in the mouth are also known as oral ulcers or mouth ulcers. Other specified diseases HIV disease resulting Used tea bags Placed under the eyes helps reduce puffiness. 0 Encephalopathy HIV disease resulting in B22. 1 Treatment for seeking a white cut inside the mouth depends on the cause.

B22. If you have surgery done, but do not make changes to prevent the-eye bags or If They are Caused by a nutritional deficiency, They are likely to reappear. Cachexia syndrome due to HIV disease B22. 7 Several diseases classified elsewhere HIV disease resulting Cut a slice of cucumber and rub it over the lips. Other disease states due to HIV disease When to see your doctor 0 Acute HIV infection syndrome B23. 1 (persistent) generalized lymphadenopathy HIV disease resulting in B23. trichinosis Blood and immune abnormalities HIV disease resulting, not elsewhere classified B23. 8 Other specified disease states due to HIV disease B24 Unspecified HIV disease ICD-10 online (WHO version 2016) In the world-renowned medical diagnostic classification system ICD-10 WHO both HIV infection and many diseases as a result can a HIV infection are coded. The exact nature of the sequela is encrypted in the updated last in 2013 by the World Health Organization version of ICD-10 in a fourth location, for example: B20. 6 Pneumocystis pneumonia HIV disease resulting. Classification of HIV infection and AIDS-defining CDC definition, case definitions of the US Centers for Disease Control and Prevention (CDC) is considered a confirmed positive HIV test as proof of HIV infection.

[5] The assessment of HIV infection on the degree of severity including AIDS has changed significantly in the year of 2008. Until then, clinical findings and laboratory values ​​were classified separately and presented as a letter-number code. It also asymptomatic and associated with HIV but not AIDS-defining illnesses were included. [6] Since 2008, the AIDS-defining illnesses are only considered. The division on the number of CD4 + T lymphocytes (helper T cells) and the list of AIDS-defining illnesses, however, remained unchanged. The in practice still widespread letter-number coding is thus not active. It should also be noted that the case definition is primarily of epidemiological surveillance of HIV and AIDS and not as a guide for clinical diagnostics. So can not be lowered again a case classification; she says therefore possibly nothing about the current status of a patient. HIV classification in adults and adolescents * [5] stage CD4 + cell count per microliter in percent of total lymphocytes 1 ≥ 500> 29% and no AIDS-defining illness 200-499 14-18% and no AIDS-defining illness 3 (AIDS) <200 two multifocal within one year) Progressive leukoencephalopathy Salmonella septicemia, recurrent tuberculosis toxoplasmosis, cerebral wasting syndrome (caused by HIV 2 ) cervical carcinoma, invasive European definition, case definition of the European Commission applies a confirmed positive HIV test as proof of HIV infection. [8] [9] A review of HIV infection on the degree of severity is not made. As AIDS is considered proof of HIV infection and one of the European case definition for AIDS established diseases. These are identical to those of the CDC's formation identical (see above). An AIDS diagnosis solely on the basis of laboratory results without any clinical findings (CDC stage 3) was discarded. [10] The European case definition used for the epidemiological surveillance of HIV and AIDS. WHO definition WHO case definition is a confirmed positive HIV test as proof of HIV infection. [11] Immunological and clinical HIV-classification in adults and adolescents * [11] stage symptoms CD4 + cell count per microliter% of lymphocytes 1 Asymptomatic ≥ 500 Asymptomatic disease (s) 2 Mild symptoms 350-499 defined for this stage disease, eg. B. herpes zoster 3 Advanced symptoms <350 defined for this stage disease, eg. , oral Haarzellleukoplakie 4 (AIDS) Severe symptoms <200

AIDS

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From Wikipedia, the free encyclopedia Acquired Immune Deficiency Syndrome, also Acquired immunodeficiency syndrome (English for “acquired immune deficiency syndrome”), usually abbreviated as AIDS or AIDS, denotes a specific combination of symptoms in humans as a result of by infection with the human immunodeficiency virus (HIV virus, HIV ) induced destruction of the immune system occur. [1] for patients it comes to life-threatening opportunistic infections and tumors. AIDS was recognized on December 1, 1981 as a distinct disease. According to UNAIDS 2014 world about 36. 9 million HIV-positive people. 2014 there were about 2 million new HIV infections (d. H. Approximately 5,600 new infections per day), and about 1. 2 million people died as a result of HIV / AIDS. [2] Since the epidemic began, 36 million people died from AIDS. The proportion of HIV-infected people is the worldwide average at about 0. 8% of 15- to 49-year-olds, however, achieved in individual African States values ​​by 25%.

[3] The effective treatment of HIV-infected people with new drugs is AIDS has become rare in Central Europe. [4] Turned out solved from an immune cell HIV The red ribbon as a symbol of solidarity with HIV positive and AIDS-sick people Definition and Classification Classification in the ICD-10 Classification according to ICD-10 B20 Infectious and parasitic diseases due to HIV disease B20. 0 Mycobacterial infections due to HIV disease (Z.

B. Tuberculosis HIV disease resulting) B20. 1 Other bacterial infections due to HIV disease B20. 2 Cytomegalovirus HIV disease resulting B20. 3 Other viral infections due to HIV disease B20. 4

Candidiasis HIV disease resulting B20. 5 Other mycoses HIV disease resulting B20. 6 Pneumocystis pneumonia HIV disease resulting B20. 7 Several infections related to HIV disease B20. 8 Other infectious and parasitic diseases due to HIV disease

B20. 9 Unspecified infectious or parasitic disease due to HIV disease B21 Malignant neoplasms HIV disease resulting B21. 0 Kaposi’s sarcoma due to HIV disease B21. 1 Burkitt lymphoma HIV disease resulting B21. 2

Other types of non-Hodgkin’s lymphoma HIV disease resulting B21. 3 Other malignant neoplasms of lymphoid, haematopoietic and related tissue due to HIV disease B21. 7 Multiple malignant neoplasms HIV disease resulting B21. 8 Other malignant neoplasms HIV disease resulting B21. 9 Unspecified malignant neoplasms HIV disease resulting


B22 Other specified diseases due to HIV disease B22. 0 Encephalopathy HIV disease resulting B22. 1 Lymphoid interstitial pneumonia due to HIV disease B22. 2 Cachexia syndrome due to HIV disease B22. 7

Several diseases classified elsewhere HIV disease resulting B23 Other disease states due to HIV disease B23. 0 Acute HIV infection syndrome B23. 1 (Persistent) generalized lymphadenopathy HIV disease resulting B23. 2 Blood and immune abnormalities HIV disease resulting, not elsewhere classified B23.

8 Other specified disease states due to HIV disease B24 Unspecified HIV disease ICD-10 online (WHO version 2016) In the most recognized medical diagnosis classification system ICD-10 of the WHO, both the HIV-infection as well as numerous diseases can be encoded as a result of HIV infection. The exact nature of the sequela is encrypted in the updated last in 2013 by the World Health Organization version of ICD-10 in a fourth location, for example: B20. 6 Pneumocystis pneumonia HIV disease resulting. Classification of HIV infection and AIDS definition CDC definition According to case definitions of the US Centers for Disease Control and Prevention (CDC), a confirmed positive HIV test is considered proof of HIV infection. [5] The assessment of HIV infection on the degree of severity including AIDS was in 2008 changed significantly , Until then, clinical findings and laboratory values ​​were classified separately and presented as a letter-number code. Here [6] were asymptomatic and involved with HIV related but not AIDS-defining illnesses.

Since 2008, only the AIDS-defining illnesses are still considered. The division on the number of CD4 + T lymphocytes (helper T cells) and the list of AIDS-defining illnesses, however, remained unchanged. The in practice still widespread letter-number coding is thus not active. It should also be noted that the case definition is primarily of epidemiological surveillance of HIV and AIDS and not as a guide for clinical diagnostics. So can not be lowered again a case classification; she says therefore possibly nothing about the current status of a patient. HIV classification in adults and adolescents * [5] stage CD4 + cell counts per microliter in percent all lymphocytes 1 ≥ 500

> 29% and no AIDS-defining illness 2 200-499 14-28% and no AIDS-defining illness 3 (AIDS) <200  two within a year) Progressive multifocal leukoencephalopathy Salmonella septicemia, recurrent tuberculosis Toxoplasmosis, cerebral Wasting syndrome (HIV-related) Cervical cancer, invasive European definition According to case definition of the European Commission a confirmed positive HIV test is considered proof of HIV infection. [8] [9] A review of HIV infection on the degree of severity is not made. As AIDS is considered proof of HIV infection and one of the European case definition for AIDS established diseases. These are identical to those of the CDC's formation identical (see above). An AIDS diagnosis solely on the basis of laboratory results without any clinical findings (CDC stage 3) was discarded. [10] The European case definition used for the epidemiological surveillance of HIV and AIDS. WHO definition According to the WHO case definition a confirmed positive HIV test is considered proof of HIV infection. [11] Immunological and clinical HIV-classification in adults and adolescents * [11] stage symptoms CD4 + cell count per microliter % Of all lymphocytes 1 Asymptomatic ≥ 500 Asymptomatic disease (s) 2 Mild symptoms 350-499 defined for this stage disease, z. B. Herpes Zoster 3 advanced symptoms <350 defined for this stage disease, eg. , oral Haarzellleukoplakie 4 (AIDS) severe symptoms <200

AIDS

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Hi Tino, I think we to conform that – we follow the ethics – no one intentionally, for study purposes, may be infected. The HIV is present and has become the scourge of humanity, is also undisputed. Quote: One suggestion, we separate the issues are slightly different. Basic research and epidemiology, medical care and therapy, policies of individual interest groups, pharmaceutical lobby, world politics, etc. Of course there are crossing points, but if you are cooperating rolls freely, you only get what you wanted before. When it comes to be the demonstration of their own prejudice, everyone can find enough evidence to support its assertion that only does not help us that . . . Even I can agree, we are – as I see – yes only in terms of development and the fight against HIV / AIDS disagree. The one must consider separately the great nevertheless dirty political deal is – if only because of the volume alone – just as clearly – in this case a cut. Regarding the fundamental research, epidemiology, so missing here just the basics.

Except for the fact that HIV was already present in the 20’s, can be found as a result only a big black hole. I follow – in the context of HIV problem – the emergence of new (really new? ) Diseases with a strong interest and try me (now not strictly scientific) to figure it out. Since we have such. B. Marburg (1967), Lassa (1969), Ebola and legionnaire’s disease (both 1976). Apart from the symptoms and course of the disease, it seems to be so that the origin of this Maladen is to look in Africa, although the first appearance at different locations was registered. Are there significant correlations between these “new” diseases and Africa? Favoring any factors in Africa the outbreak of new diseases? Why these diseases appear increasingly on the middle of the last century? Questions, questions – I’m trying to define just only one kind of causality. Greeting Artaxerxes