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HSV Eraser Protocol
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OlOlOlOl$/(C “(C BASES adjudication SMALL AMOUNT N 0610M04222-ESSALUD-2006 ACQUISITION OF LABORATORY SUPPLIES 1-ENTITY CALLING The Red Asistencial of Lambayeque “Aita Juan Valle” – Legal Essalud with at Plaza Security s / n Chiclayo , RUC 20121357750 2-OBJECT oF PROCESS Acquisition Laboratory Supplies – Hematologae Immunology – Linked to equipment. – Lambayeque Healthcare Network “Aita Juan Valle” 3-VALUE BENCHMARK S / 6444.00 (Six 1444 00/100 nuevos soles) including taxes Calculated at November 15, 2006 4-ACQUISITION SYSTEM This process System Unit Prices 5- SOURCE oF FUNDING financed with resources 6-governed pAYMENT payment perform in soles and within as stipulated in Art.238 of Regulations of the law of the State Procurement, approved by Supreme Decree 084-2004-PCM. it is required to pay the contractor or supplier has met with full delivery of the quantities of the indicated and documents must not contain erasures or deletions of any kind. If it is canceled or declared void this adjudication, Essalud assumes no responsibility for expenses incurred by the bidders for the presentation of proposals. 7-REQUIREMENT AND TECHNICAL SPECIFICATIONS requirement inputs Laboratories, its characteristics and technical specifications subject of this process, Sealan to continuation: ORDENCODIGOITEMDESCRIPCIONCANT.U.MPRECIOREF.REFERENC.TOTALMIN. 70% MAX. 110% ANTI HERPES VIRUS 1 130103863ANTICUERPO IgG200PBA7.861,572.001,100.401,729.20230103900ANTICUERPO HERPES VIRUS 1 IgM200PBA8.101,620.001,134.001,782.00330103864ANTICUERPO ANTI ANTI ANTI HERPES VIRUS HERPES VIRUS 2 2 IgG200PBA8.121,624.001,136.801,786.40430103901ANTICUERPO IgM200PBA8.141,628.001,139.601,790.80 6,444.004,510.807,088.40 Referential Amount: Six in 1444 with 00100 new maximum amount soles: Seven thousand eighty eight with 40/100 new soles minimum Amount: four in 1510 with 80/100 nuevos soles. TECHNICAL SPECIFICATIONS: SEE ANNEX 1 August-Duty submission of proposals submitted in two (2) sealed envelopes (Technique and economic) Envelopes Bern carry the inscription of adjudication of Minor How much N 0610M04222 and business name of the bidder. 9-TECHNICAL PROPOSAL (about N 1) properly foliated MAXIMA EVALUATION 100 POINTS. Shall contain: Document Index. Sworn statement of business information, pointing out the name or business name of the bidder or their legal representative, address, enrollment in Sphere of Records, as appropriate, and indication of the document identity. Sworn statement in accordance with the provisions of Art.76 of Regulation of the Law on State Procurement. 4- Sworn Declaration of being a pequeao Micro company (if applicable) 5- formal promise Consortium (if applicable) 6- authorization Sanitary Company (required) Proof of inscription 7- n in the National Register of Providers (mandatory) 8 Sheet fulfilling the technical specifications required by EsSalud, brochures and catalog supported with 9- Letter of Commitment in which consign conditions and delivery 10- Registration good health Vigente offered (required) 11- Certificate of Analysis (mandatory) 12 Integrity Pact (required) 13- Effective product minimal 06 months (required) 14- Certificate of good Manufacturing Practices and Storage current (National Product) Certificate of Free Sale Anlogo (foreign product). 15- Letter of representation in force, issued by the manufacturer on behalf of the bidder 16- documents (contracts, order d / purchase invoices) proving the bidder’s experience CALIFICACION.- score according to the following factors provide: EVALUATION FACTORS Experience bidder TECNICAPUNTAJE the amount billed by the bidder is eligible for a specified period not exceeding ten (10) years from the date of submission of proposals up to a maximum amount of 10 documents tem. Ms bills do not qualify garbled 100% pricetag From 50-99% of the reference price From 01 to 49% of the reference price 40 30Plazo delivery Delivery period less 05 calendar days Delivery time 06-09 calendarios40 das 20Certificaciones: QUALITY: ISOS 9001, 9002, 13485, European certification CE FDA approval. 20 NOTE: The technical proposals whose score is less than 60 points, Sern dismissed. ECONOMIC 10-PROPOSAL (about N 2) properly foliated MAXIMA EVALUATION 100 POINTS Must contain: 1. well bid price included I. G. V, expressed in nuevos soles and two (2) decimal places. ESSALUD not recognize additional payment of any kind. The offered Unit Price not subject to any adjustment. The economic evaluation be to assign the maximum score to the economic offer lower cost. The rest of the proposals will be assigned scores according the following function: Pi = Om x PMPE Oi Where: i = Proposal Pi = Score of economic offer i Oi = Offer = Offer economic Om i economic cost or lower price PMPE = Score maximum of Proposition economic DETERMINING TOTAL SCORE = 0.60PTi + 0.40PEi PTPI Where: PTPI = Total Score bidder i Pti = Points per Evaluacint Pei technique bidder i = Score for economic evaluation of the bidder i c1 = weighting coefficient for evaluacintcnica (0.60) c2 = weighting coefficient for the economic evaluation (0.40) 11 EXTRA POINTS in aplicacina N 27633 Law regulated with the DSN 003-2001-PCM with N27143 relacina the “Law for the promotion of National Productive Development Temporary” is added additional to the sum of the calificacintcnica and economic obtained by offers of goods produced within the national territory 20%. TOP VALUE TO BENCHMARK authorize the granting of the Award in cases PROPOSALS 12, in which tenders they have reached the highest total cost, and exceeding the reference value set to the limit additional 10% 13- SCHEDULE OF PROCESS OF CALL The Calendar of this call will be governed according to schedule dates to continuation outlined: ETAPASFECHAS convocatoria04 December 2006REGISTRO PARTICIPANT Atencina Office Suppliers – Procurement Office in Av Salaverry N 760 -CHICLAYO 08. : 30 am to 15:00 horas.04 and 5 December 2006 Proposal Submission Atencina Office Suppliers – Procurement Office in Av Salaverry N 760 -CHICLAYO from 8:30 a.m. to 3:00 p.m. horas.06 of. ASSESSMENT December 2006 December 2006 PROPUESTAS11 GRANTING oF gOOD PRO12 December 2006 CONSENT oF tHE AWARD (*) Note (*): the concession is automatically consented, after expiry of the deadlines for interposici n impugnacin resources without sb bidder has exercised that right. SPECIFICATIONS TECNICAS030103863 (1) Antibody IgG anti Herpes virus 1 PBAPRESENTACION: reagents for the detection of IgG antibodies against Herpes virus 1 96 ms packaging tests. Expiry time not less than 6 months from the date of delivery. METHODOLOGY: immunochemiluminescence, electrochemiluminescence Fluorescent Immunoassay Enzimainmunoensayo. Ultima Generation, Quantitative Semiquantitative. With Antgenos SPECIFIC Pptidos Recombinant Herpesvirus 1. synthetics ACCESSORIES: Calibrators, Controls, Accessories, Accessories allowing full realization of the test. BIOLOGICAL SAMPLE: Serum Plasma.030103900 (2) anti virus Herpes 1 IgM antibody PBAPRESENTACION: reagents for the detection of IgM antibodies against Herpes virus 1 96 ms packaging tests. Expiry time not less than 6 months from the date of delivery. METHODOLOGY: immunochemiluminescence, electrochemiluminescence Fluorescent Immunoassay Enzimainmunoensayo. Ultima Generacin without precipitacin, Quantitative Semiquantitative. With Antgenos SPECIFIC Recombinant Herpesvirus synthetics Pptidos 1. Optional with neutralizing Rheumatoid Factor. ACCESSORIES: Calibrators, Controls, Accessories, Accessories, allowing full realization of the test. BIOLOGICAL SAMPLE: Serum or Plasma.030103864 (3) Antibody Herpes Virus 2 IgG anti PBAPRESENTACION: reagents for the detection of IgG antibodies against Herpes virus 2 in 96 ms packaging tests. Expiry time not less than 6 months from the date of delivery. METHODOLOGY: immunochemiluminescence, electrochemiluminescence Fluorescent Immunoassay Enzimainmunoensayo. Ultima Generation, Quantitative Semiquantitative. With Antgenos SPECIFIC Pptidos Recombinant Herpes virus 2. synthetics ACCESSORIES: Calibrators, Controls, Accessories, Accessories, allowing full realization of the test. BIOLOGICAL SAMPLE: Serum Plasma.030103901 (4) anti Herpes Virus 2 IgM antibody PBAPRESENTACION: reagents for the detection of IgM antibodies against Herpes virus 2 in 96 ms packaging tests. Expiry time not less than 6 months from the date of delivery. METHODOLOGY: immunochemiluminescence, electrochemiluminescence Fluorescent Immunoassay Enzimainmunoensayo. Ultima Generacin without precipitacin, Quantitative Semiquantitative. With Antgenos SPECIFIC Recombinant Herpesvirus Pptidos synthetics 2. Optional with neutralizing Rheumatoid Factor. ACCESSORIES: Calibrators, Controls, Accessories, Accessories, allowing full realization of the test. BIOLOGICAL SAMPLE: Serum or Plasma. TECHNICAL SPECIFICATIONS DELQUIPO REQUESTED ON ASSIGNMENT OF USE Immunoassay Analyzer Random (ITEMS: 1, 2, 3, 4). TipoAnalizador Random Access Discretob. MetodologaQuimioluminiscencia, Electrochemiluminescence, enzymatically or similarc Fluorescence. Performance80 or more fotomtricas Horad tests. Capacidad12 or more tests on line e. MuestraTubo primary and / or alcuotaf. DatosInterno processing: Software and hardware for processing calibrations, controls and External results: Software open connection to computer system Laboratory (SIL or LIS). Software and Hardware: Data Management Team for a period of 1 year or more printing: provide software that allows printing format 9 7/8 “x 11”. Include printer, tape and papel.g. AccesoriosFuente emergency power (UPS) Air Conditioning Equipment (if the equipment manual indicates) h. Consumables, controls and complementosTodos materials, consumables and accessories be delivered free of Bern acompaando periodic delivery of reagents in sufficient quantity that according to the protocol of the methodology allows the complete realization of the effective testing and monitoring processes calibraciny scheduled for delivery period and deliveries immediately in case replenishment schedule does not correspond to actual consumption of Calibrators Service: Provide calibration material from the same batch for all the purchase period sufficient to allow the full realization of the tests according to protocol methodology analtica Controls: Provide control material on two levels each of a single batch for the entire period buy enough for the realization of a process control computer on each working day during the purchase period Cleaning Solutions and Accessories amount: in sufficient quantity to allow full realization of the requested tests, calibrations and additional washes, required according to the methodology of trabajoi. TcnicoPrograma support preventive maintenance and technical team available for the carrying out of procedures correctivosj. Operation mode 220V, 60Hz PAGE PAGE 4 ESSALUD Red Asistencial Lambayeque ‘(.12 @ fgl, ‰, – / G W p ̻ݱ˜ƒmƒ]ƒmƒmHƒHƒmƒ(hx-Rh 69B*CJOJQJaJhphhx-RhV[c6 CJOJQJaJ+hx-RhV[c5 B*CJOJQJaJhph(hx-RhV[cB*CJOJQJaJhph0h$-_hV[cB*CJOJQJaJhmH PhSH hV[cCJOJQJ!h #:5 B*CJOJQJhph!h u~5 B*CJOJQJhph!hV[c5 B*CJOJQJhph!h>?5 B*CJOJQJhph@fglˆ‰ . / Q to $ $$ gd>? $ ‘$ L) to $ GDV [c `$ L ) GDV [c $ \x26amp; F “S” ^ “s`” to $ gd> $ `$ L) to $ GDV [c $ to $ GDV [cA :: ‘?: “BCEFW hks Œ $ H {

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> @ 3Fbjbj)) 4’KzKz3> ˆ >>>*CW`-b-b-b-b-b-b-$ .R?1 †- ccc†- >> ›-K#K#K#c > >`-K#c`-K#K#V+@ v+> PH( w!$^+ , -0 -j+ 1›” 1v+djT v+ 1 |,8ccK#ccccc†-†- $ A # PATHOLOGY HERPES HERPES VIRUS EYE SIMPLE (VHS) The vast majority of eye injuries herpticas estn produced by HSV type I. The index of recurrence at 5 years is 40%. Epithelial ulcers respond to antiviral treatment, but afectacin Stromal can not disappear completely, leaving an opaque or nefelin scar. The cutneas injuries, in the absence of sobreinfeccin, heal without scarring sequelae. Conjunctivitis HSV The afectacin primary ocular HSV can manifest as acute follicular conjunctivitis or keratoconjunctivitis with preauricular adenopata and often, with the appearance of vesicles on the skin periauricular. If no vesicles, the DD with adenoviral conjunctivitis is facilitated after careful search were signs of herpticas vesicles are in the eyelid margin. Unlike the primary disease, recurrent blepharitis is a much more localized infection. The lesions heal without scarring injuries. Conjunctivitis often accompanies diffuse and serous secrecin. Occasionally the Gram stain Rose Bengal or fluorescein reveals a dendrtica conjunctival ulcer. Unlike many teraputicas herptica guidelines used when the disease affects the cornea or other ocular structures, herpticas manifestations limited to the conjunctiva require a simple treatment. Antiviral agents or steroids estn not indicated, however, a antibitica ointment can be used to prevent bacterial sobreinfeccin. Patient follow-up should be narrow to rule afectacin dela cornea or ocular adnexal that occur amend the teraputico approach. Primary HSV keratitis usually appears as a diffuse punctate keratitis inespecfica evolving microdendritas the formation of multiple scattered by the cornea. These lesions can lead to linear serpiginous ulcers, errticas, across the corneal surface. treatment are indicated antivirals (acyclovir) so tpica together a A typical NSAIDs and antibitica ointment to prevent infection. In immunocompromised patients can be combined with antiviral treatment typical sistmico and hold for more time than the indicated period. HSV keratitis recurrent outbreaks of recurrent ocular herpes. Ulcers branched and thin on the corneal epithelium dendrticas. Symptoms and signs caractersticos: lacrimation, irritation, photophobia and sometimes blurred vision. In these cases antiviral prophylaxis is indicated by VO (acyclovir, famciclovir, valaciclovir). HESPES OFTLMICO Zoster is a reactivation of the varicella virus zster. It is a ganglioradiculoneuritis. Of the three divisions of the fifth cranial nerve, the first (oftlmica branch) is by far the most frequently affected. (Inervacin of eyelids, forehead, tip of the nose, most of the orbit and ocular adnexa). It is a very aggressive and a large inflammatory component infection. The treatment is what we discussed in the session, the use of corticosteroids tpicos be justified because they reduce the inflammatory response and the precipitates and corneal infiltrates. Always associated with oral and tpicos antivricos. From: Infectious Diseases. Principles and practice. Sixth Editing. 2006. Mandell, Bennett and Dolin Published 13 August 2009, doi: 10.1136 / bmj.b2624Cite esta as: BMJ 2009; 339: b2624 Practice 10-Minute Herpes zoster ophthalmicus Consultation Fook Lam Chang, specialist register in ophthalmology1, Allison Law, General practitioner2 William Wykes, consultant ophthalmologist1 1 Department of Ophthalmology, Southern General Hospital, Glasgow G51 4TF, 2 Southbank Surgery 17-19 Southbank Road, Kirkintilloch G66 1NH Correspondence to: FC Lam HYPERLINK “mailto: fook_chang@hotmail.com” fook_chang @ hotmail .com a 65 year old woman attends your practice with a two day history of a vesicular rash around her right eye. She Past reports general feeling of fatigue and malaise and has-been slightly feverish over the past week. She Had noticed a pain around her right eye even before the skin eruption Began. You should cover issues What is it and why What has she got it? After an attack of chickenpox virus the (varicella zoster) remains dormant in the body. This virus is kept in check by the immune system. However, in 20% of people the virus is reactivated, RESULTING in a painful rash Localised With blisters (shingles). The commonest cause is a weakening of the immune system With Age; MOST Patients are aged over 50 years. Other causes include stress, fatigue, and a weakening of the immune system from other illnesses or from medical treatment handler (such as chemotherapy or immunosuppression). When the eruption Involves the area around the eye (or the ophthalmic division of the trigeminal first nerve), this is called herpes zoster ophthalmicus, irrespective of Whether the actual eye itself is Involved. Ophthalmic herpes zoster accounts for 10-25% of all cases of shingles. Have I got the right diagnosis? The main differential diagnosis is herpes simplex infection. In herpes simplex Usually the Patients are young, and the rash will not follow a dermatome, nor will it obey the midline. In herpes zoster ophthalmicus it is not unusual for the edema to track to the other side of the face, but the rash remains dermatomal in distribution. Can I predict WHO will get eye problems? The appearance of the rash on the tip, the side, or the root of the nose Indicates the Involvement of the nasociliary nerve (Hutchinson’s sign) and a higher risk of ocular Involvement (80%). Age, sex, and severity of skin rash are not good predictors. What are the possible ocular complications? These Usually Develop week from the second after the onset of the rash. Herpetic neuralgia post is by far the commonest complication. Age is a potent risk factor. Antiviral drugs reduce the risk by 50%, but 20% of affected Patients aged over 50 will continue to report pain six months on initial DESPITE antiviral treatment. Less common complications are: Lid complications-ptosis, trichiasis (ingrowing eyelashes), scarring of skin, madarosis (loss of lashes), and Anterior segment complications-conjunctivitis, episcleritis, scleritis, stromal keratitis (inflammation of the corneal stroma, Which can lead to permanent corneal scarring), neurotrophic keratitis (corneal degeneration Caused by the loss or reduction of corneal innervation), anterior uveitis (inflammation of the uveal tract above), and intraocular pressure raised. Rare complications include: Posterior segment complications-acute retinal necrosis (viral retinal infection RESULTING in marked inflammation and retinal death), progressive outer retinal necrosis (viral retinal infection in immunosuppressed Patients-progresses more Rapidly but eye is less inflamed), optic neuritis, and Motor neuropathy, third nerve palsy: such as. Useful reading Dworkin RH, RW Johnson, Breuer J, JW Gnann, Levin MJ, Backonja M, et al. Recommendations for the management of herpes zoster. Clin Infect Dis 2007; 44 (suppl 1): S1-26 Gnann JW Jr, Whitley RJ. Clinical practice. Herpes zoster. N Engl J Med 2002; 347: 340-6, doi: HYPERLINK “http://dx.doi.org/10.1056/NEJMcp013211” 10.1056 / DW NEJMcp013211 Wareham, Breuer J. Herpes zoster (Clinical review). BMJ 2007; 334: 1211-1215, doi: HYPERLINK “http://www.bmj.com/cgi/doi/10.1136/bmj.39206.571042.AE” 10.1136 / bmj.39206.571042.AE Useful information for Patients MedicineNet. Herpes viruses (including the chickenpox virus) and the eyes. HYPERLINK “http://www.medicinenet.com/herpes_of_the_eye/article.htm” www.medicinenet.com/herpes_of_the_eye/article.htm eMedicineHealth. Shingles. HYPERLINK “http://www.emedicinehealth.com/shingles/article_em.htm” www.emedicinehealth.com/shingles/article_em.htm What You should do her visual acuity Eye examinationChecking is vital. A normal vision and a “white” eye are very reassuring; however, be alert to the Hutchinson’s sign. Advise the patient to report any pain, reduced vision, or redness of the eyes, as the need for esta Indicates a repeat assessment and more detailed eye examination. Treatment and managementOral her on antivirals-Start With An antiviral treatment (see box). Antiviral treatment in antiviral treatment herpes zoster Systemic Shortens the healing process of acute herpes zoster and you reduce pain and other acute and chronic complications When Within 72 hours after Given onset of the rash. Older Patients shed the virus for longer and Have a higher risk of complications and still benefit from antivirals Could After This period, Especially If They still have new vesicles forming. Antivirals Should be Considered in all Patients With herpes zoster ophthalmicus, even if They are presenting after 72 hours. Acyclovir, valacyclovir, famciclovir and are accepted in the United Kingdom as first line treatments. They are like in tolerability and safety, but acyclovir is the drug of choice Usually on grounds of cost effectiveness. Some doctors prefer valaciclovir and famciclovir Because of the superior pharmacokinetics and more convenient dosing regimens. Standard duration of treatment is 7-10 days. With supplementary treatment corticosteroids May Shorten the degree and duration of acute zoster pain but have no effect on the development of post-herpetic neuralgia. Analgesia-antivirals, analgesics, and a neuroactive agent handler (such as amitriptyline, gabapentin, or carbamazepine) are effective for acute pain and can be combined. Capsaicin cream to the skin is licensed for post-herpetic neuralgia after the skin lesions healed Have. Bacterial superinfection-Discourage scratching and tell her to keep the area clean With warm compresses to reduce the risk of infection. Antihistamines itching relief. Prescribe oral antibiotics if you suspect superinfection. Advise her to isolation-Avoid contact with Individuals Who Have no history of chickenpox (especially pregnant women) Have Until the vesicles dried up (usually after Several days). ReferralOphthalmology-A visual acuity reduced, a red eye (Indicates inflammation), Hutchinson’s sign, and oculomotor palsy all warrant referral to ophthalmology. Because of the high risk of ocular complications, Patients With Hutchinson’s sign Should be seen Within 1-2 weeks. Patients with a red eye Should be seen within 24 hours to 48 hours, while Patients with a red eye and reduced vision Should be seen the same day or at the very latest the next morning. Physicians and infectious diseases department-More severe disease, multiple dermatomal involvement, or an underlying immunodeficiency Suggest recurrence. Patients With Organ Transplants and Patients on systemic immunosuppression or chemotherapy need closer follow-up and Should be managed in liaison with a hospital physician. Extensive cellulitis will necessitate admission for intravenous antibiotics. Pain clinic-herpetic neuralgia Established post can be very difficult to treat and can persist for years in 10% of Patients With This condition. Should be Treated Neuralgia THEREFORE aggressively. In more severe cases the patient and resistant Should be Referred to a pain clinic before the Pain Becomes chronic and established. Cite this as: BMJ 2009; 339: b2624 This is part of a series of occasional articles on common problems in primary care. The BMJ Welcomes Contributions from GPs Competing interests: None DECLARED. Provenance and peer review: Not commissioned; externally peer reviewed. (Accepted 19 March 2008) INCLUDEPICTURE “http://www.bmj.com/icons/spacer.gif” \\ * MERGEFORMATINET HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=CITEULIKE” \\ O “CiteULike” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/citeulike.gif” \\ * MERGEFORMATINET CiteULike HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=COMPLORE” \\ O “Complore” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/complore.gif” \\ * MERGEFORMATINET Complore HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=CONNOTEA” \\ O “Connotea” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/connotea.gif” \\ * MERGEFORMATINET Connotea HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=DEL_ICIO_US” \\ O “Del.icio.us” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/delicious.gif” \\ * MERGEFORMATINET Del.icio.us HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=DIGG” \\ O “Digg” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/digg.gif” \\ * MERGEFORMATINET Digg HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=FACEBOOK” \\ O “Facebook” \\ t “_blank” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/facebook.gif” \\ * MERGEFORMATINET Facebook HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=REDDIT” \\ O “Reddit” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/reddit.gif” \\ * MERGEFORMATINET HYPERLINK Reddit “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=STUMBLEUPON” \\ O “StumbleUpon” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/stumbleupon.gif” \\ * MERGEFORMATINET StumbleUpon HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=TECHNORATI” \\ O “Technorati” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/technorati.gif” \\ * MERGEFORMATINET Technorati HYPERLINK “http://www.bmj.com/cgi/external_ref?tag_url=http://bmj.com/cgi/content/long/339/aug13_1/b2624\x26amp;title=Herpes+zoster+ophthalmicus+–+Lam+et+al.+339+%28131%29%3A+b2624+–+BMJ\x26amp;doi=10.1136/bmj.b2624\x26amp;link_type=TWITTER” \\ O “Twitter” INCLUDEPICTURE “http://www.bmj.com/icons/shared/socbookmark/twitter.gif” \\ * MERGEFORMATINET Twitter HYPERLINK “http://www.bmj.com/help/social_bookmarks. DTL “What’s this? Relevant Articles Do not forget HIV Kathir YoganathanBMJ 2009 339: b3621. HYPERLINK “http://www.bmj.com/cgi/content/extract/339/sep14_1/b3621” [Extract] HYPERLINK “http://www.bmj.com/cgi/content/full/339/sep14_1/b3621 “[Full Text] Herpes zoster David W Wareham and Judith BreuerBMJ 2007 334: 1211-1215. HYPERLINK “http://www.bmj.com/cgi/content/extract/334/7605/1211” [Extract] HYPERLINK “http://www.bmj.com/cgi/content/full/334/7605/1211 “[Full Text] HYPERLINK” http://www.bmj.com/cgi/reprint/334/7605/1211 “[PDF]” 56

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> 5@ 2Tbjbj22 XXL8, l \x26amp; A2 ” “> $> $> $ @@@@@@@ $ XBRD \\ @> $$ |> $> $> $ @ k @ 7,7,7,> $ X @ 7> $ @ 7.7, -]> † Qf * d> @ 0 \x26amp; A EW + E (??? ? E> $> $ 7,> $> $> $> $> $ @@ – Encephalitis LMBICA AUTOIMMUNE potentially reversible a new entity.. authors: Haddad L, S Marciano, Novillo A, S Boughen, Chvez G, Viaggio B, C. Lopez Saubidet area Presentation: neurologist bibliographical references: New England Journal of Medicine Institute CEMIC. Department of Medicine. Buenos aires. Summary lmbica encephalitis is a picture that develops within a few days or weeks and is characterized by impaired short-term memory, confusion, epilpticas crises ys psychiatric symptoms. there may be temporary lobes afectacin both the electroencephalogram (EEG) and magntica resonance (NMR), inflammatory changes in the liquid and cefalorraqudeo onconeurales antibodies against both channels voltage-dependent potassium (CKVD); Overview is his usual infectious or paraneoplastic origin. The recent identification of patients with this syndrome, but who never develop cancer, have high titles against CKVD, negative onconeurales antibodies and good response to immunosuppressive therapy, results in the description of a new entity: the idioptica lmbica encephalitis or potentially reversible lmbica autoimmune encephalitis (ELAPR). In this entity, clnico another striking fact is the almost constant presence of hyponatremia very difficult handling and frequent initial normal EEG and MRI installed despite the symptoms. We report a 76-year-old male with no previous neurolgicos history that begins with a subacute box seizures (simple partial seizures), severe memory disorders, cognitive disorders, and hyponatremia of normalization difficult. EEG presents with left lentificacin, normal initial MRI, normal CSF, protena 14.3.3 negative, VDRL negative trax tomography of abdomen and pelvis regular, full body PET negative, anti-Hu negative, PSA normal HIV negative, normal thyroid hormones. Descartndose the main differential diagnoses (infectious encephalitis, Hashimoto encephalitis, neurosyphilis, Creutzfeldt-Jakob and infectious herpes simplex encephalitis virus), having improved with steroid treatment, added to the appearance on MRI fourth practiced of feature temporary image, hyperintense on T2, consider this case within the criteria ELAPR we decided to present it as the recent identification of patients with this syndrome has broadened the spectrum of etiolgico limbic encephalitis, inducing think that is a little known syndrome, which currently has more than 30 cases reported worldwide. Introduction The lmbica encephalitis is a rare disease that was identified as a separate entity in 1968 by Corsellis et al.1 clinically characterized by a subacute box personality changes, loss of short term memory and seizures that often leads to dementia. It is usually considered to be most frequent cause paraneoplastic its association with cancer of pulmn of small cells (SCLC) 2. The physiopathologist of paraneoplastic encephalitis lmbica I remained unexplained until an antibody called anti HU I recognized antgenos present in neurons and tumor cells were identified in the serum of patients with SCLC and encephalomyelitis. 15 The potentially reversible lmbica called encephalitis ideoptica or an entity in which the neurolgico clnico picture is similar to paraneoplsico but that the presence of hyponatremia, negative onconeuronal antibodies, adds against voltage-dependent potassium channels (VGKC) in high titles and excellent response to inmunosupresores.5. We report the case of a male patient of 74 years old who has a clnico box and compatible additional studies with this entity and consider it important given that the recent identification of this syndrome has expanded the etiolgico spectrum of types of limbic encephalitis have been reported and only 17 patients in two series 5 to 16. Descripcin Case: male patient 76 years old with a history of arterial hypertension diabetes type 2, dyslipidemia, which in November 2005 began with simple partial seizures engine (flexiny contraction of left arm and left deviation of the corner of the mouth), type the electroencephalogram (EEG) showed a lenificacin left temporal pattern and magntica resonance imaging (MRI) of the brain was normal. Home anticonvulsant diphenylhydantoin treatment with valproic acid and then with no favorable response from what is broken Carbamazepine in January 2006, showing slight improvement but transient why a new MRI was performed more angioresonance brain that also I was normal, one tomography by emission of single photon (SPECT) in which I present temporofrontal perfusion defect in izquiedo and parietofrontal was done right. For box hyponatremia (119mmol / L) secondary to inadequate secrecin of antidiurtica hormone more sensory deterioration is decided to suspend starting treatment with carbamazepine anticonvulsant therapeutics with levetiracetam up the plasmtico sodium 131mmol / L but with neurolgico slight improvement in the picture. Evolve recent memory disorders, severe cognitive deficit, worsening crises every 5 to 10 minutes with progression to the right arm and further decline of plasmtico sodium. The puncin of cefalorraqudeo liquid was normal, with tests chain reaction (PCR) for herpes simplex, the protena 14.3.3 (CJD) and VDRL (syphilis) negative; In laboratory studies erythrocyte sedimentation rate, c-reactive protena, thyroid hormones, antgeno benign prosttico (PSA), were within normal values, serology for HIV was performed, different antibodies (FAN, ASMA is dosaron , AMA, antiLKM), the anti HU and antitiriodeos antibodies were negative. Trax tomography was performed, abdomen and pelvis and tomography by emission tomography (PET) total body being normal. In the fourth brain MRI performed in March evidenced by the increase in volume FLAIR hyperintensity seahorses and a predominance of right side. Figure 1 is interpreted as limbic encephalitis box which initiates treatment with corticosteroids, performing five pulses of methylprednisolone (1 gr.da) showing improvement clinic, continues treatment with 60 mg given deltisone ; Patient course with complete disappearance of partial seizures and confusional state and normalization of sodium plasmtico but persisting with alteration of recent memory. Months later esteriodeo treatment continues falling and presents a new RNM that evidence to disminucin and hyperintensity volume of the right hippocampus. Discussions describe a patient with a compatible clnico lmbica box with encephalitis and neuroimaging feature, which presents favorable response to corticosteroids; This being clinically indistinguishable from other encephalopathic syndrome propose the following differential diagnoses that were dismissed: Wernicke-Korsakoff Syndrome: This brain disorder involving loss of specific brain functions due to the deficit of thiamine , clinically not only affects the central nervous system (memory disorders, cognitive and seizure) but 80% affects the nervous system peripherals, has also oculomotor disturbances and ataxia. Since our patient has no Neuropathy Peripheral, oculomotor disturbances or ataxia, l diagnosis is dismissed. Hashimoto encephalopathy: neurolgico deficit syndrome and neuropsicolgicos particularly in patients with a history of Hashimoto’s thyroiditis despite having thyroid hormone levels within normal ranges. The clnico picture is characterized by the presentation, in acute or subacute, of alteration of consciousness frequently accompanied by focal seizures or generalizadas.2. The neurolgico examination may reveal signs of motor focalizacin, besides bilateral ataxia mioclonas and the alteration of consciousness and / or psychiatric symptoms, despite having good response to corticosteroids dismiss this pathology as our patient has positive antitiriodeos l antibodies needed to diagnose this disease. Creutzfeldt-Jakob disease: this is a degenerative disorder often causes more espordica, the symptoms of this disease can include confusion, memory failures, depression, behavioral changes, lack of coordinaciny visual disturbances. According disease progresses, patients experience rapidly dementia (progressive loss of intellectual abilities), neuromuscular disorders, mioclnicas jolts and atetosis. The EEG shows a typical layout consisting of peridicas pins or bi trifsicas 90% of patients.13 The CSF is usually normal and the presence of a protein highly sensitive 14.3.3 detected 96% and specifies 99% 13 clnico box with a compatible; It is unknown at this protena increases which is present in different sectors of the CNS with maximum concentration in the hippocampus. this pathology is dismissed for not having EEG and present protena 14.3.3 caracterstico negative. The diagnosis of neurosyphilis neurosfilis is usually difficult, for which not enough one element, the CSF VDRL has high specificity, but not so sensitive, false negative results may shed is why it helps the diagnosis and the protein levels of white blood cells count. Being normal in our patient and negative CSF VDRL, l diagnosis is dismissed. Encephalitis virus Hepes simple: the disease of acute character that usually manifests clinically impaired sensorium, fever, headache, signs of partial or generalized epilpticas focalizaciny crisis, in which the PCR for herpes simplex CSF has 98% sensitivity and 100% specificity and 14 being negative in our patient, so the diagnosis is desestim. paraneoplastic limbic encephalitis: A disorder characterized by the occurrence of subacute epilpticas crisis, alteration of short-term memory, confusion and psychiatric disorders such as anxiety, depression and hallucinations; this entity may be affected in remote areas of the nervous system to the limbic system. Generally usually appear months, even years before the appearance of tumor.2 It is due to immune-mediated response, wherein the tumor expresses a protein found in the CNS generating a response inmunolgica production of antibodies that react against the tumor as well as also against SNC.4 addition to the clinic, have afeccin of temporary lobes in the EEG may show focal unilateral temporal epileptiform activity or bilateral or more inespecficos findings (generalized slow waves or temporary focal), being only average in 20% of cases 2,16. In the CSF examination usually find slight lymphocytic pleocytosis with increased content of proteins and in most of the patients studied oligoclonal bands or elevated index immunoglobulin G is also observed. MRI usually shows hyperintensity on T2 or FLAIR uni or bitemporal mainly in hippocampus or amygdala (Figure 2, 3 y 4), alterations in MRI have been described in 65-80% d cases is likely auque percentage increase with radiological monitoring (Figure 2) Up to 60% of patients with paraneoplastic encephalitis lmbica onconeuronal have positive serum antibodies and lcr2 (table 1).; HU anti antibodies are the most frequent generally associated with SCLC. Second estn anti MA 2 associated with testicular tumors; In the absence of these antibodies are the most common tumors of pulmn ca, ca breast, Hodgkin’s disease and teratoma. There are no established protocols for treatment if the condition worsens clinic Alternatives are plasmapheresis, immunoglobulin and immunosuppressive agents such as cyclosporine and tacrolimus corticosteroids, this being not entirely effective therapeutics. We believe the clinic and complementary studies correlate our patient with this entity, but no antibodies present positive onconeurales not show tumor with complementary studies and above all having had good response with corticosteroids leads us to think of a case potentially reversible or idioptica lmbica autoimmune encephalitis. ideoptica potentially reversible or autoimmune encephalitis lmbica: This entity in which there are approximately 25 cases reported, 17 patients have been described including two Series5, 16, the majority being male (14), with an average age of 64 years (between 44 and 79 years), with clnico box similar to that seen in paraneoplastic cases in which more striking as data associated hyponatremia (12 of 17 patients), the RNM patolgica in 15 of 17 patients in whom 11 had bilateral temporal afeccin and 4 afeccin unilateral and stop all negative neoplsicos antibody; Of the 17 patients 9 had excellent response to treatment, 6 a slight improvement, a spontaneous improvement and patient had no response. All patients had high antibody titles VGKC; In the series of patients from the University of Oxford5 the title was between 450 and 5,128 pm (normal value less than 100 pm); These antibodies are directed against channel subtypes k 1.1 and 1.2 kv found in the membrane of neurons of the dentate gyrus but also in other areas of the CNS and PNS, however it is not known that these patients have only encephalitis lmbica, while others with the same antibodies develop neuroomiotonia, presumably by blocking the SNP. Conclusion: We believe our patient in the diagnosis of encephalitis since idioptica presents a consistent picture with lmbica clnico encephalitis, hyponatremia, neuroimaging and good response characteristic with immunosuppressants. Not currently possess the dosage of VGKC is already a difficult technique to perform, which is not yet validated and currently is done only in a laboratory in Europe. Figure 1. Fourth RNM patient case in which increase in volume and hipernitensidad seahorses to predominance of right side of Figure 2 radiological monitoring RNM in T2 showing progressive increase in signal in the hippocampus and amygdala are observed Figure 4 MRI in T2 showing increasing bilateral hippocampi seal in Figure 3 FLAIR MRI in coronal section showing increased signal in Hippocampus right ANTICUERPOTUMOR ASOCIADOANTI HU ANTI-CV2LH SCLC * * And SCLCANTI-MA (1.2 Y 3) CA TESTICULOANTI-VGKC * TABLE 1 thymoma paraneoplastic antibodies associated with different tumors * SCLC: cancer of pulmn type of small cells. * LH: Hodgkin lymphoma. * VGKC: voltage-dependent potassium channels. Bibliography 1. Corsellis JA, GJ Goldberg, AR Norton. “Limbic encephalitis” and Its association with carcinoma. Brain 91: 481-496, 1968. 2 Gultekin SH, Rosenfeld MR, R Voltz, Eichen J, Posner JB, J. Dalmau paraneoplastic limbic encephalitis: neurological symptoms, immunological and tumor association Findings in 50 Patients. Brain 200; 123: 1481-94 3 Buckley C, Oger J, Clover L, et al. Potassium channel antibodies in Patients With two reversible limbic encephalitis. Ann Neurol 50: 73-78, 2001.432 Internal Medicine Vol 42, No. 5 (May 2003) 4 Darnell RB, Posner JB.. Paraneoplastic syndromes Involving the nervous system. N Engl J Med 2003; 349: 1543-1554. 5 Vincent A, Buckley C, Schott JM, Baker I, Dewar B.-K, Detert N, et al. Potassium channel antibody-associated encephalopathy: a Potentially immunotherapy-responsive form of limbic encephalitis. Brain 2004; 127: 701-12 6 J.M.S.Pearce.Paraneoplastic Limbic Encephalitis.European Neurology 2005; 53: 106-. 108. 7 Luis Bataller, MD, Josep Dalmau, Md; Phd. Neurologic paraneoplastic syndromes Neurol Clin N Am 21 (2003). 8 Robert B. Darnell and Jerome B. Posner A new cause of limbic encephalopathy Brain (2005), 128, 1745-1746 9 Edmund James Dunstan, John B. Winier. Autoimmune limbic encephalitis, Causing confusion and fits Rapidly progressive hyponatraemia. Age and Ageing doi: 10.1093 / aging / afl045. 10 Kazuhiro Fukushima, Masahide Yazaki, Kazuma Kaneko, Tomohisa Fushim, Kanji Yamamoto, Takao Hashimot, Kazuhiro Oguchi, Shu-ichi Ikeda. Nonparaneoplastic, Nonherpetic LimbicEncephalitis Episodic With Severe Hypothermia: aseReport EurNeurol2005; 54: 170-174, DOI: 10.1159 / 000089938 11 Bethan Langa, Russell C. Daleb and Angela Vincent. New autoantibody medieted the disorders of the central nervous system. Curr Opin Neurol 2003 16: 351-357. 12 Yasuhiro Watanabe, Yasutaka Sfflmizu, Shinji Ooi, Keiko Tanaka, Encephalitis. Internal Medicine Vol. 42, No. 5 (May 2003). 13 Richard Johnson, M.D.J and Clarence J. Guibs, JR. Creutfeld Jacob Disease and realetedTransmisible spongiforme Encephalopathies.The NEJ of Medicine.nesm dicember 1998. 14 Whirtey RJ, Lakeman. F.Herpes simplex virus infection of the Central Nervous System: Terapeutic and Diagnosys consideration.Clinical Infection Dis. 1995: 20: 414- 420. 15 Dalmau J Graus, RosenblumMK, PosnerJB, Anti HU Associated paraneoplasic encephalomielitis / Sensory neuropathy.A Clinical Study of 71 Patients. Medicene 1992; 71: 59-72. 16 MJ Thieben, MBBS, FRACP; V. Lennon, kD, PhD: Boeve, MD: A.J.Akamit, MD, FRCP and S.Vernino, MD, Phd Pothetially reversible autoimmune limbic encephalitis neuronal potassium channel With antibody.Neurology 2004; 62: 1177-1182. PAGE PAGE 1

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HSV Eraser Protocol
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ࡱ> ` ~Fbjbjss BR> vesicles (few mm in diameter, tense, serous content not umbilicate, with narrow halo of erythema) >> pstula >> scabs. Affects the trunk, roots of limbs, face and scalp (high intensity upper body). Elements presented in all stages unlike smallpox. Zster herpes affects adult, most frequently at older ages. It starts with highly variable prdromos (malaise, asthenia, fever), it has a neural component and a component cutneo. Neural: Pain, itching, accompanied by paresthesia, bloat thoracoabdominal area with regional adenopata frequency. Cutneo: neural days later appear as urticadas and pruritic plaques, vesicles are formed grouped or confluent, tense, serous content, metamrica and unilateral distribution. postherptica Neuralgia: are paresthesias, itching and especially the pain that persist over time. Both processes evolve over 4-6 weeks. The vesicles can be hemorrgicas, desecndose scabs. The most affected areas are: Cervical Torcica Lumbosacral The oftlmica. (S. eye. Eyelid edema, photophobia, keratitis (Cornales walleye may be motor paresis of the eyeball, ptosis, enophthalmos or anisocoria) has opportunistic character and develop in older ptes necr Herpes Zoster. Alphabet: General process in which the plates are cutneas surface lesions, disseminated necrosis is spread HZ HZ recurrence only in immunodeficient treatment:…. 200-800 mg acyclovir c4 / h has an effect on the pain component, postherptica prevents neuralgia in many cases parenterally is equal to severe HS vitamins should be given… B-1 B-6, B-12 should not be local priest carbamazepine can be effective for neuralgia postherp. infectious mononucleosis tica Agent:.. Epstein-Barr virus the incubacin is long 30-50 days after headache, fever, sore throat, lymph nodes tumefaccin, splenomegaly There rash by 25% between the 4th and 6th d. a, maculopapular, affecting the trunk and roots of members. The reaction of Paul-Bunell is + 1/32 (aglutinacin of hemates ram) CMV has a high diffusion, most of subclnicas infections and persist in intracellular virus latency which it has become important in immune deficiencies especially AIDS. It produces less incidence of skin rashes that Epstein-Barr virus. Warts (papillomas vricos) Agent: HPV (human papilloma virus). HPV types in connection with pictures clnicosHPV 1Verruga plant clsicaHPV 2Verruga plant in mosaicosHPV 2, 4, 7Verruga vulgarHPV 3, 10, 27, 16. 18Condiloma 28Verruga planaHPV acuminadoHPV 16, 6, 11Papulosis bowenoideHPV 6 11Enf. Buschke-loewesteinLas common warts: are very common affecting mainly children and adolescents. They are small lumps of Hemispheric hyperkeratotic surface or plateaus. They are located mainly in the hands. Periungual warts are common. On the face and trunk usually solitary. They persist for a long time and disappear spontaneously. Warts are widespread in immunodeficient. Flat warts: affects children and young adults, with the highest incidence in women (they are small flat epidrmicas ppulas of a millimeter or less in diameter, often slightly. . pigmented a dark color appears especially in the face profusely sometimes have a linear disposal, for artifact phenomenon isomorphic Koebner do Epidermodysplasia lewanoswsky-ltz verruciformis.. is a genetic picture . rare recessive inheritance gradually appearing multiple flat warts atpicas no tendency to malignant epithelial tumors remisiny desarrollndose (.. epiteliomas, Alzheimer’s Disease Bowen) plantar warts. it is solitary or with few elements is a Lesi . n queratsica flat or slightly excavated, a few millimeters in diameter, located mainly in the support points is painful to pressure, hindering the march is very persistent genital warts.. they are so General sexually transmitted comprise: Genital warts Bowenoid ppulas the penis.. verrucous carcinoma of genital warts (NFE. Buschke-Loewenstein). also common warts. Histopatolgia: Flat warts >>>>>> acanthosis and hyperkeratosis without papillomatosis. >>>>>> common warts acanthosis, papillomatosis, and parakeratosis. In both there are cells in the mucous vacualizadas upper body Treatment common warts and plantar warts are used tpicos ocusticos queratroliticos: salicylic acid and lactic acid in colodin, acid triclorac tico, saliclica 20-30% Vaseline. It can be used or electrocoagulacin cryotherapy. the extirpacin quirrgica is not recommended queratolticas Flat warts pastes can be applied with salicylic acid, or formulas of retinoic acid or glycolic acid. Disease “hand-foot-mouth” ((Coxsackie virus is common in children, is short evolution and goes undiagnosed. Cause vesiculopustular lesions affecting the oral mucosa, back of the tongue, palate, geniana mucosa. Also some vesicles on the back of fingers and toes. there may be fever and anorexia, abdominal pain or diarrhea. Herpangina can be mistaken for pharyngitis. the causative agent is another Coxsackie. the onset is abrupt with fever, headache, muscle aches and afectacin general condition. they appear pequeas papulovesicular injuries peritonsillar area. Acropapulosis child is observed in school children 5-12 under the profuse outbreak of small papular and erythematous lesions without affecting pruritus . of the extremities symmetrical way There are 2 tables: Acrodermatosis Papulosa child or enf of Giantti-Crosti. HBs Ag positivity has hepatitis B. Giantti-Crosti syndrome: it is related to other viruses: Epstein Barr, CMV,> adenovirus, or ECHO virus. It must be distinguished from other acropapulosis that are only in the hands and are itchy and are caused by or traumticos to irritative effects. (Infantile papular dermatitis). Measles The causative agent is a paramyxovirus. It is contracted early in childhood. The incubacin inicindose is 8-10 days with fever, colds and upper respiratory going conjunctivitis. From the 2nd it gives the Koplik spots are seen in the oral mucosa. Gives presents to the 4th there is a rise of fever, rash morbilliform head starts at the foot shaped ppulas confluent erythematous, pruritic. The 6th da starts the outbreak begins to fade and the 10th descamacin given. Rubella It is caused by a togavirus a banal disease is created, until the serious consequences it has on the fetus when the mother if you have it in the first trimester of pregnancy was demonstrated. The infection starts after 2-3 weeks of incubacin, there prdromos fever, catarrh and conjunctivitis go higher areas. Similar to measles, but more mild. There mastoid adenopatas, cervical suboccipitals and absence of Koplik spots. The rash starts on the face and spreads to the trunk with papuloeritematosos elements. Megaleritema, Fifth disease is caused by a parvovirus das 6-14 after the rash starts on the face, with diffuse erythema that respects the nasolabial folds. The outbreak is spread over 2 days to the trunk and proximal parts of the limbs, in the form of large plates excntrico growth and disposal annular with erythematous and pale center. It is the enf. ms child eruptive feature in its dermatolgica morphology. Cleared 8-10 days. Roseola infantum (exanthem sbito) No virus has been isolated. It begins with fever, followed by a rash with pink maculopapulos elements in the neck, trunk and roots of members, very fleeting, disappearing in days. There adenopatas and easily confused with rubella. It has been identified as the causative agent of Burkitt lymphoma and AIDS, as concausa of hairy leukoplakia. (Microcephaly, cardiac patients deafness, mental retardation, cataracts). The name of fifth disease, currently in disuse, alluding the order in which Haban been identified exantemticas childhood fevers (first: measles, second: scarlet fever, third, rubella, fourth: enf . of filantow-Dukes) PAGE PAGE 1 Phase takes Herpes Zoster, Varicella recurrence Primoinfeccin Herpesvirus Virus varicellae dermatoses gr? s_ +. = D and w … ” ²¢¢ “ “ “s“g“ “ “ “ZVJh s2hRNf5 CJaJhRNfhRNfhRNf5 B*ph h 5 CJOJQJaJh ;`hRNf5 B*\\ ph “hkE hRNf5 6 CJOJQJaJhkE hRNfCJOJQJaJhkE h›O 5 CJOJQJaJh s2h›O 6 CJOJQJaJhkE h›O CJOJQJaJ#h ;`h›O 5 B*OJQJ\\ ph hœ^hE{ OJPJQJ^Jjh…{6UmHnHu< . = e l``$$Ifa$gd 6 ikd$$If–F ” ” p' ' t0'644 the Ifa $ $$ gdRNf $ to $ gd "i $ to $ gd = rgd ... {$ 6 to $ gd ... {6RDF'F-F 345 @ HIabcpz { ' " *+78@Astu~?‡ˆ ‘™š 沮 h 6 h s2h 6 5 B*CJaJph h s2h I CJaJh s2h 6 5 CJaJh s2hRNf5 CJaJhRNfh s2hRNfCJaJh s2h 6 CJaJD 4fZZ $$ Ifa $ gd6kd [$$ If-F "F" Y0p '@ t0'6 the $ 44 Ifgd645 @ IboccZ $ Ifgd6 $$ $$ If Ifa $ gd6kd -F "F" Y0p '@ t0'644 labcp {occZZ $ Ifgd6 Ifa $ gd6kdY $$ $$ If-F "F" Y0p '@ t0 'laoccZ $ Ifgd6 $$ 644 Ifa $ gd6kd $$ If-F "F" Y0p '@ t0'6 44 * oc the Ifa $$ $$ $ gd6kdW If-F "F" Y0p '@ t0'6 * + 44 the 8AT • ‰} t $ IfgdRNf $$ $$ Ifa Ifa $ gdi $ gd6ikd $$ If-F "" p '' t0'6 44 latu? ocWN $ IfgdRNf $$ $$ Ifa Ifa $ gdi $ gd6kd? $$ If-F "F" Y0p ' @ t0'644 la'šocWN $ IfgdRNf $$ Ifa Ifa $ GDI $$ $$ $ gd6kd If-F "F" Y0p '@ t0'6 44 laocWN $ IfgdRNf $$ $$ Ifa Ifa $ gdi $ gd6kd1 $$ If-F "F" Y0p '@ t0'644 laocWN $ IfgdRNf $$ $$ Ifa Ifa $ gdi $ gd6kd $$ If-F "F" Y0p '@ t 0'644 the

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> Y 5pbjbjWW 3 ‘=’ =, fW ]8888„„„˜˜˜˜8 ˜‡Mh44:nnnMMMMMMMMMM$ N Pš*M]„MIMMM*M‰88nn 4‰‰‰M$8 n„nM˜˜8888MM‰L‰ F D \x26amp; ^ “Mn” @ + j> VIRUS ~~q0LGENERALIDADES Estn between replicative ms pequeos organisms. They are “filterable agents” pass through filters. They do not see optical microscopy. Size: 20 to 300 nm in diameter (spherical shape tend to): <25nm (S), 180-200 nm (M),> 250 nm (L). Its genome is constituted by a single type of nucleic acid (which can synthesize independent RNA or DNA). Has a mandatory requirement intracellular growth and structural dependence on the host cell. They are strict parasites (not replicated if the host is not clul). In its structure we found inside out: Nucleus or core of RNA or DNA. In some cases this genetic material is associated with proteins, such as retroviruses (reverse transcriptase). Cpside: surrounding the nucleus, usually protein structure. They are repeating units of proteins called capsmeros. Each capsmero is equal to another. This is the basic structure. In some cases hau one lipdica or carbohydrate cpside surrounding the casing. The area between the casing lipdica cpside and called tegument and is normally occupied by carbohydrates. In some viruses they are projected to the outside structures called espculas, important in replicacin. Also they are called peplmeros. Classification of viruses which cause the disease Place: Neurotrpicos: they multiply in the nervous system. Enterotrpicos: multiply in the digestive system. Type of host: animals, plants, humans, bacterifagos. structural features or replicacin: size, shape, and wrap with or without peplmeros, replicacin site (cytoplasm or nucleus), type of nucleic acid, citoptico effect produced. Taxonmica Classification: Family viridae suffix Example: herpesviridae. Virinae subfamily Ex: a herpes virinae.

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> 5@ Š)bjbj 2 24r X X ˆjjj~†(†(†(†(l ( ~ A )6 * * * + -.L [. (^ A`A`A`A`A`A`A $? CRED “Ajƒ.,” -ƒ.ƒ. “A * +] ™ AA2A2A2ƒ.8 * j + ^ AA2ƒ. ^ AA22A2s2r (= TRj (> +) S †(?0p|= ?t A0 AŠ= F 06F(>~~(> Fj >ƒ.ƒ.A2ƒ.ƒ.ƒ.ƒ.ƒ.„A„A~~d %2~~ 1) What are “STDs” what “Sexually transferable diseases” HYPERLINK “http://www.gesundheitpro.de/Geschlechtskrankheiten-Geschlechtskrankheiten-A050829ANONI013043.html” \\ l “by portable” \\ t “_blank” http://www.gesundheitpro.de/Geschlechtskrankheiten-Geschlechtskrankheiten-A050829ANONI013043.html#uebertragbare Sexually transferable diseases: (Sexually Transmitted Diseases, STD): STD, diseases (also outside of the genitals) are which transmitted through sexual contact, you are not notifiable (except HIV) trichomonas infections genital herpes fungal infections genital warts.. HIV Filzluse yeast infections STDs: the law in STDs to a declaration syphilis gonorrhea ( “gonorrhea”) chancroid ( “chancroid”) (in Europe very rarely) some form of chlamydia ( “lymphogranuloma venereum”) = venereal Lymphknotenentz. invention (rare) 2) When should I go to the doctor? HYPERLINK “http://www.aidshilfe.de/index.php?id=2655\x26amp;sessionLanguage=de\x26amp;sessionCountry=DE” http://www.aidshilfe.de/index. php id = 2655 \x26amp; language = en \x26amp; session session country = dE problems: HYPERLINK “http://www.aidshilfe.de/” http://www.aidshilfe.de/ (sexually transferable diseases immediately to the doctor or health care professional should go to if to himself oR the partner or the partner a sexually transferable infection is found or if you notice the following symptoms: vaginal discharge or itching of urethra blood or mucus admixtures in stool discoloration of urine or stool lnger sustained Druckgefhl in the abdomen abdominal pain or loss of appetite lasting tiredness or fatigue unclear fever Hautausschlge and -rtungen or warts. 3) What diseases are notifiable? What “meldpflichtig” means? (The legal situation in Austria and Germany is equal.): HYPERLINK “http://www.infektionsnetz.at/TextExtMeldepflicht.phtml” http://www.infektionsnetz.at/TextExtMeldepflicht.phtml D: HYPERLINK “http: / /www.gesundheitpro.de/Geschlechtskrankheiten-Geschlechtskrankheiten-A050829ANONI013043.html “\\ l” by portable “\\ t” _blank ” http://www.gesundheitpro.de/Geschlechtskrankheiten-Geschlechtskrankheiten-A050829ANONI013043.html#uebertragbare The law treats sexually transmitted – these are syphilis, gonorrhea ( “gonorrhea”), Soft chancre ( “chancroid”) and a certain form of Chlamydia ( “lymphogranuloma venereum”) – a declaration. That is, the person concerned must be treated by a doctor to eliminate the risk of infection; and the doctor is obliged to disease case – anonymously – to report to the health authorities. 4) number the STDs after their Hufigkeit the global incidence: HYPERLINK “http://de.wikipedia.org/wiki/Sexuell_%C3%BCbertragbare_Erkrankung” http://de.wikipedia.org/wiki/Sexuell_%C3% BCbertragbare_Erkrankung (wHO figures from 1990 worldwide number of new cases) SHAPE \\ * MERGEFORMAT 5) Share the STDs and STDs in the types of pathogens: a) viruses: HIV, genital herpes (herpes simplex as fever blister ), genital warts (human papilloma virus (HPV)), hepatitis B b) bacteria: gonorrhea, syphilis, Soft chancre, venereal Lymphknotenentzndung (chlamydia) c) protozoa: trichomonas infection d) fungi: yeast infections e) multicellular animals: Filzluse 6) briefly describe in brief the following diseases in addition to the above erwhnten links: HYPERLINK “http://www.aidshilfe.de/index.php?id=5363\x26amp;sessionLanguage=de\x26amp;sessionCountry=DE” http: / /www.aidshilfe.de/index.php?id=5363\x26amp;sessionLanguage=de\x26amp;sessionCountry=DE Photos http://de.wikipedia.org/wiki/Sexuell_%C3%BCbertragbare_Erkrankung trichomonas infection Photo: HYPERLINK “http: //de.wikipedia. org / wiki / image: Trich_vag.jpg “\\ o” Trichomonas vaginalis “INCLUDEPICTURE” http://upload.wikimedia.org/wikipedia/de/7/7b/Trich_vag.jpg “\\ * Mergeformatinet transmission: ungeschtzter intercourse , smear infections over wet Gegenstnde, eg Towels (not rare in children), and in damp Rumen (sauna) .Symptome: men: usually asymptomatic women: Scheidenentzndung with purulent, frothy, belriechendem discharge and qulendem JuckreizPrvention (prevention) : condoms and safer sex. Towels should be used only with one person. Hygienic caution. at ground toilets and saunas. Genital herpes (1/5 of the euro piss population) similar cold sores around the mouth Photo: HYPERLINK “http://de.wikipedia.org/wiki/Bild:Herpes_simpex_virus.jpg” \\ o “Herpes Simpex virus” INCLUDEPICTURE “http://upload.wikimedia.org/wikipedia/commons/thumb/f/f2/Herpes_simpex_virus.jpg/180px-Herpes_simpex_virus.jpg” \\ * Mergeformatinet transmission: Contact infection TrpfcheninfektionSymptome: painful Hautvernderungen V. A. on penis, vulva and vagina: itching, Hautrtung, and formation of Blschen that open up eventually; Prvention (Prevention): Condoms and avoiding contact with infektiser Blschenflssigkeit yeast infections (3/4 of all women are affected once in life!) Vagina and vulva (vaginitis or vulvitis) or skin folds of the anal region are the yeast (Candida albicans) infected. This fungus colonized natrlicherweise the vagina, but increase with disturbances of the biological balance of VaginalfloraFoto: HYPERLINK “http://de.wikipedia.org/wiki/Bild:C_albicans_en.jpg” \\ o “Candida albicans” INCLUDEPICTURE ” http://upload.wikimedia.org/wikipedia/commons/thumb/f/f5/C_albicans_en.jpg/280px-C_albicans_en.jpg “\\ * Mergeformatinet transmission: ungeschtzter intercourse symptoms: Women: redness and swelling of üren genital area, severe itching, weilicher (wiped off) lining, vaginal burning and -jucken, discharge; Mnnern: redness and inflammation of the foreskin and the glans, may erase whitish Belge; Prvention (Prevention): Safe sex Filzluse be Colloquially Filzluse often called “Liebeskfer” or vulgar “Sackratten” or “mast sailors” bezeichnet.Foto: HYPERLINK “http://de.wikipedia.org /wiki/Bild:Filzlaus.jpg “\\ o” louse “INCLUDEPICTURE” http://upload.wikimedia.org/wikipedia/de/thumb/9/99/Filzlaus.jpg/300px-Filzlaus.jpg “\\ * Mergeformatinet transmission: close krperlicher contact, sexual contact, rarely contact with contaminated Gegenstnden (eg toilet seat, bed linen, towels) symptoms: at first infection (Miger) itching, with a renewed infection due to an allergic reaction (against lice excrement) very ausgeprgter JuckreizPrvention (prevention): avoid tight Krperkontakten Hygienic caution. at ground toilets and saunas. Chlamydia Chlamydia infection is the most common (reportable) STD. Chlamydia are considered most common cause for infertility. In addition it is the most common cause for blindness in developing countries. WARNING: syphilis can lead to serious Nervenschden to Lhmungen. Tripper leads untreated often lead to infertility. The prospect for a physician / health care professional in case of suspicion of STDs is indispensable! More Links: HYPERLINK “http://www.netdoktor.de/ratschlaege/fakten/geschlechtskrankheiten.htm”Netdoktor: http://www.netdoktor.de/ratschlaege/fakten/geschlechtskrankheiten.htm HYPERLINK” http: // en. wikipedia.org/wiki/Trichomonaden “trichomonas infection 120 million HYPERLINK” http://de.wikipedia.org/wiki/Chlamydien “\\ o” chlamydia “chlamydia 50 million HYPERLINK” http://de.wikipedia.org/wiki/ HIV “\\ o” HIV “HIV 40 million (2000) HYPERLINK” http://de.wikipedia.org/wiki/Humanes_Papilloma-Virus “\\ o” Human papillomavirus “Human papillomavirus 30 million HYPERLINK” http: / /de.wikipedia.org/wiki/Gonorrhoe “\\ o” gonorrhea “gonorrhea (gonorrhea) 25 million HYPERLINK” http://de.wikipedia.org/wiki/Herpes “\\ o” herpes “herpes 20 million HYPERLINK” http: //de.wikipedia.org/wiki/Syphilis “\\ o” syphilis “syphilis 4 million HYPERLINK” http://de.wikipedia.org/wiki/Hepatitis “\\ o” hepatitis “hepatitis B 2 million HYPERLINK” http: / /de.wikipedia.org/wiki/Ulcus_molle “\\ o” chancroid “Chancroids

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



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ࡱ>

HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> `! „ϴQG? ˆg x z 8– H2X ‘Š„ xœl o$I–\x26amp;= Ү ‡ С4V ^ VVZ% Œ m_tƒ \x26amp; ~ $ = 4EwN1ŒSt 3“? /“wz g ? ‘o n Œ ?=™|c O { ƒqC` z7~ = ; 0 Œw ‡!0 ` ? Œ{ ƻ E ` o0ލ/L 0 Œw d ` o0ލ ƒqC` x7N = ݸ ; 0 Œw ` o0ލ ‡ xƒ n „; 0 Œw ‡!0 ` 7 = ݸ w0 aŒ7 M ƒqC` x7n ‰ 7 ͿƒqC` x7 Œ{ ƻ`o0 ލ w † xƒn

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> ‚„ M ! == 2~WW bbjbj lRRRRRRRfnnnn ^ $ ‘lf1t $ 1,111,111 3 521R 21DRR G1DDD. – RR 1D 1D.Dr’V @ RRH / Q (fn.h /] 101.x55h / DffRRRRINFECCIONES HERPES VIRUS AND SIMPLE Varicella Zoster Salvador Pereira Sanz, Luis Ayerbe Garca-Monzn Gema Escribano Romo, Beatriz Manrique Olmedo, Ftima Garca Cores, Irina Larrayoz Lopez, Francisco Javier Panadero Carlavilla. the herpes simplex type 1 (HSV-1), type 2 (HSV-2) virus and virus varicella zoster virus (VZV) are part of the Alphaherpesviridae subfamily, which in turn to the family of human herpesvirus belongs. family to which have been incorporated into new viruses such as human herpes virus type 6 (HHV-6), the human herpes virus type 7 (HHV-7) and human herpes virus 8 (HHV-8). Some of these viruses have been described in recent years and have been interacting with pathologies such as rash s bito (HHV-6) or Kaposi’s sarcoma (HHV-8). the herpes virus infections are a frequent reason for consultation in the usual practice clinic, sometimes generally regarded as a pathology that diagnstica presents little difficulty. This is not always true and herpes viruses may have a very different dermatolgica clinic. The existence of effective antiviral frmacos (acyclovir, valacyclovir, famciclovir …) makes it important to modify the early diagnosis of the disease course. INFECTIONS HSV-1 and HSV-2 etiology The herpes simplex virus HSV-1 and HSV-2 cause various infections affecting mucocutneas surfaces, the central nervous system and occasionally some vscera. The HSV genome is a double stranded DNA molecule of which has a 50% homologous between HSV-1 and HSV-2 types, both subtypes and can distinguish different strains by analysis of restriction endonuclease No viral DNA. The viral genome is involved in a capsule of icosadrica protenica structure formed by 162 capsmeros. It is neurotropic virus, the infection of some neurons does not cause cell death but allows you to integrate the viral genome into the cell; remaining in a dormant state. Later this viral genome can become transcribing suffering reactivations. The infection occurs by direct contact with secretions or genital oral from infected subjects with or without symptomatology. Incubacin period between 1 and 26 days. The HSV-1 infection is usually acquired in childhood, more than 90% of adults have antibodies to HSV-1 to reach the fifth decade of life. Antibodies to HSV-2 usually do not appear until puberty, up to 50% of heterosexual adults who consult in clinics for sexually transmitted diseases (STDs) have antibodies to HSV-2. It has been shown that infection by HSV-2 is an independent risk factor for acquisition and transmission of HIV (HIV) type 1. Manifestations of infection clinics by HIV-1 and HIV-2 virus can cause both visceral lesions or mucocutneas areas, can cause genital infections and orofacial that are clinically indistinguishable. The buccolabial HSV-1 infection has more tendency to relapse than bucolabiales HSV-2 infections, just as genital infection by HSV-2 recurrence of 8 to 10 times more than that caused HSV-1. Gingivostomatitis herptica orofacial infections usually the manifestation of primoinfeccin HSV-1 virus affecting young children and adults in less than 1% of those exposed. This is usually a consistent picture in vesicular lesions on the soft palate, enca, lips, tongue and accompanies fever, malaise, cervical adenopata; having a duration between 3 and 14 days. The reactivation of HSV located in ganglia trigmino usually associated with mucosal ulcerations in the mouth or ulceracin herptica on the edge of the lip, acompandose the excretion of virus in saliva. Atpica patients with dermatitis may have severe orofacial infections (herptico eczema) with the possibility of visceral diseminacin. In immunocompromised patients the infection spreads deep into the layers of skin and mucosa causing necrosis, bleeding and intense pain that prevents eating or drinking. genital infections The first episode of genital herpes is characterized by fever, headache and muscle aches that accompany local symptoms: itching, dysuria, pain, painful inguinal adenopata and vaginal and urethral secrecin. The neck of the uterus and urethra affect 80% of women. HSV proctitis presents with pain, constipation, rectal tenesmus and anorectal secretions. It has been linked with the practice of anal intercourse but also may appear on a latent level of a sacral dermatome infection. DIFFERENTIAL DIAGNOSIS OF HERPES GENITAL1.- sifiltico2.- Canker Canker blando3.- lymphogranuloma venereo4.- granuloma inguinal.5.- reumatolgicas Diseases. Enf. Behcet’s syndrome Erythema Reiter6.- fixed pigmentado.7.- Squamous cell carcinoma of bullous traumtica.9.- pene.8.- Ulcer Disease: Pnfigo and Penfigoide.10.- Other: Contact Dermatitis , erythema multiforme. Whitlow herptico is a digital infection that can occur as a complication of oral or genital herpes or direct inoculation from solutions of continuity of the skin. Coursing with edema, erythema and localized pain in the finger and is clinically indistinguishable panadizo to bacterial secondary infection. Folliculitis herptica: This is a rare manifestation of HSV infection, clinically is similar to a acn I excoriated and injuries are accompanied by burning and itching, does not respond to the usual treatments performed of acne. Generally the diagnosis is obtained by culture or laboratory techniques. Herpes gladiatorum: mucocutnea is an infection caused by HSV and is extended by the trax, ears, face and hands. The extension of the infection is favored by the cutneo trauma that accompanies the practice of the fight. CNS infections and peripherals HSV encephalitis has a bifsica distribution with two peaks one among ages 5 to 30 years and another after 50 years. Ms estn 95% originate from HSV-1. Presents with the sudden appearance of fever and symptoms focal temporal lobe especially neurolgicos. Even with treatment are common sequelae, especially over 35 years. The appearance of HSV meningitis usually occur simultaneously to a primary genital HSV infection. Has an acute course of days and 2-7 presents with headache, fever and photophobia, not typically be neurolgicas injuries. The VHS is the agent that has been associated with more recurrent lymphocytic meningitis (Mollaret syndrome), the HSV infection has been linked also with Bell’s palsy and rare cases transverse myelitis Guillain-Barre ysndrome. Visceral infections occur during viremia pudindose affect multiple organs. The esofgica afectacin presents with odynophagia, dysphagia, substernal pain and weight loss, endoscopy or barium examination do not allow to differentiate a Candida esophagitis, by custicos or posradiacin. Pneumonitis is rare except in severely immunocompromised patients, where it can take as a focal necrotizing pneumonitis, interstitial pneumonitis or. Have mixed flora (bacterial, parasitic and mictica) has a greater than 80% mortality in immunosuppressed patients. HSV does not usually cause hepatitis in immunocompetent patients, the infection of the liver causes fever, a sudden increase in bilirubin, transaminases, leukopenia (28 weeks of gestation and no history of chickenpox in the mother. Preterm infants hospitalized

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> 9 (bjbj .Ÿ Ÿ $ l^^^^^^^rUUUUVœr”0 X X X X X X X X†“ˆ“ˆ“ˆ“ˆ“ˆ“ˆ“$7• W—P “^ X X X X X “`r^^ X X “`r`r`r X^ X^ X†“`r X†“`r `r”u^^”u X X _ r SU p”u”ud “0””u — p —”u`rrr^^^^ ICD10 – CODES KrankheitICD-CodeKrankheitICD-CodeA. Carotid int. Stenosis / -VerschluI65.2AdipositasE66.9Addison-KrankheitE27.2Adenome of KolonD12.6AgressivittF91.1AgranulozytoseD70AIDSB24Akutes AbdomenR10.0AlkoholpsychoseF10.5AlkoholabususF10.1Allergie allgemeinT78.4Allergie MedikamenteT88.7M. AlzheimerG30.9Anal-Ca.C21.0AnalfissurK60.2AnalfistelK60.3Anmie aneurysm -allg.D64.9 -AortaI71.9 -BlutungD62 -dissecans AortaeI71.0 -EisenmangelD50.9Angina pectorisI20.9Angina tonsillarisJ03.9Anorexia nervosaF50.0Aortenklappeninsuff.I35.1AortenklappenstenoseI35.0ApoplexieI64AppendizitisK35.9ARDSJ80Arterielle EmbolieI74.9Arteriitis temporalis (M. Horton) M31.5Arthrosis deformansM15.9Arzneimittel / DrogenabhngigkeitF19.2Asthma bronchialeJ45.9AszitesR18AspirationspneumonieJ69.0Atopisches EkzemL20.9AV-BlockI44.3Blutung R58Bronchial Ca.C34.9Bronchitis acute J20.9Bronchitis chron.J42Budd Chiari SyndromI82 .0Bulimia nervosaF50.2CandidoseB37.9Carotisstenose / -verschluI65.2Cerebrale MassenblutungI61.9CerebralskleroseI67.2Cervix Ca.C53.9Cholangitis with SteinK80.3Cholangitis, vense without SteinK83.0CholezystitisK81.9CholedocholithiasisK80.5CholezystolithiasisK80.2Chronisch Insuff.I83.9Chron. PolyarthritisM05.9ClaviculafrakturS42.0COPDJ44.9Colitis, ischmischK55.9Colitis ulcerosaI85Colon irritabileK58Colon Ca.C18.9Coma diabeticumE14.0Coma hepaticumK72.9Coma hypoglyc.E15Commotio cerebriS06.0Contusio cerebriS06.2Cor pulmonaleI27.9Corpus Ca.C54.9Cushing-SyndromE24.9Cystitis, akutN30. 0DarmblutungK92.2Delirium tremensF10.4Demenz, senileF03DepressioF32.9Diabetes mellitus (insulin dependent) E14.9Diabetes mellitus TypIE10.9Diabetes mellitus TypIIE11.9Diabetisches KomaE14.0Diarrhoe infektisA09Diarrhoe not infektis (chron.) K52.9Digitalisintox.T46.0DiskusprolapsM51.9DivertikulitisK57. 9Drogenabhngigkeit. OpiateF11.2DuodenitisK29.8DysurieR30.0EisenmangelanmieD50.9Embolie / thrombosis art.I74.9EmphysemJ43.9Endokarditis, akutI33.9EpilepsieG40.9EpistaxisR04.0ErysipelA46Erythema nodosumL52ExsikkoseE86ExtrasystolieI49.4FazialispareseG51.0Fettleber, alkoholischeK70.0Fieber unclear GeneseR50.9FurunkelL02.9Gangr nR02GastritisK29.7GastroduodenitisK29.9KrankheitICD-CodeKrankheitICD-CodeGastroenteritis, akutA09Gstroenteritis chron.K52.9Gehirnblutung, intrazerebralI61.9GichtM10.9GalukomH40.9Grand-mal-EpilepsieG40.6GrippeJ10.1H morrhoidenI84.9Hepatitis alkoholischeK70.1Hepatitis chron.K73.9Hepatitis A akutB15.9Hepatitis B akutB16.9Hepatitis B chron B18.1Hepatitis C acutely B16.9Hepatitis C chron.B18.2HepatomegalieR16.0Herpes simplex labialisB00.9Herpes simplex genitalisA60.0Herpes zosterB02.9Herzinfarkt, akutI21.9Herzinfarkt, altI25.2HerinsuffizienzI50.9Herzinsuffizienz left I50.1Herzrythmusst rungenI49.9HiatushernieK44HypakusisH91.9Hypercholesterin mieE78.0Hyperlipid mieE78.2HyperparathyreoidismusE21.3Hypertensive HerzerkrankungI11.9Hyperthyreose allgemeinE05.9Hyperthyreose (M.Basedow) E05.4Hypertonus art. esentiellerI10Hypertonie, renovasculrI15.0Hypertonie, endokrinI15.2Hypertonie other sekund reI15.8Hypertriglycerid mieE78.1HyperventilationR06.4Hypoglyc mieE16.2HypoparathyreoidismusE20.9Hypothyreose postinfektisE03.3Hypothyreose after OPE89.0HypotonieI95.9IkterusR17IleusK56.6Ileus – GallensteinK56.3Ileus – paralytischK56.0Ileus – BridenileusK56.5Inkontinenz, BlaseR32Inkontinenz, DarmR15IschchialgieM54.3Kachexie by NeoplasieC80Kachexie, unklarR64KammerflimmernI49.0KariomyopathieI42.9KarpaltunnelsyndromG56.0KarzinoidE34.0KataraktH26.9KHKI25.1KopfschmerzenR51Larynx-CaC32.9LaxantienabususF55Lebensmittelvergiftung, bakt.A05.9LebermetastaseC78.7Leberzell-Ca.C22.0LeberzellverfettungK76.0Leberzirrhose, alkoholischeK70.3LeistenhernieK40.9Leukmie AMLC92.0Leukmie ALLC 91.0Leukmie CLLC91.1Leukmie CMLC92.1Lues, FrhsyphilisA51.9LumbagoM54.4LungenembolieI26. 9LungenemphysemJ43.9LungendemJ81Lupus erythematosus, syst.M32.9Lyme-BorrelioseA69.2Lymphadenopathie, unklarR59.9M. BechterewM45M. ChronK50.9M. HodgkinC81.9M.BasedowE05.4Magen-CaC16.9MagenblutungK92.2MalariaB54Mallory-Weiss-SyndromK22.6Mamma-CaC50.9MastopathieN60.3Meckel-DivertikelQ43.0Melanom, malignesC43.9Meningitis, bakterielleG00.9Metastasen – GehirnC79.3Metastasen – LeberC78.7Metastasen – LungeC78.0Metastasen – LymphknotenC77.9Metastasen – KnochenC79.5MigrneG43.9MitralklappeninsuffizienzI34.0MitralklappenstenoseI34.2Mononukleose, infekt.B27.9Multiple SkleroseG35Myelodysplast. SyndromD46.9Myokarditis, akutI40.9Myokarditis, chronischI51.4NabelhernieK42NebenniereninsuffizienzE27.4NephrolithiasisN20.9Nephrotisches SyndromN04.9Neuronitis vestibularisH94.0NierenarterienstenoseI70.1KrankheitICD-CodeKrankheitICD Code renal insufficiency, chron.N18.9Nierenkrankheit, zystischeQ61.9Nierenversagen, akutN17.9Nierenzyste, erworbenN28.1Nierenzell-CaC64NikotinabususF17.1 sophagitisK20 sophagus-CaC15.9 sophagusdivertikcl erworb.K22.5sophagusdivertikel angeb.Q39.6 sophagusvarizenI85.9OsophagusvarizenblutungI85.8OsteomyelitisM86.9OsteoporoseM81.9Ovarial-CaC56Ostitis deformans (Paget’s disease) M88.9Panarteriitis nodosaM30.0Pankreas-CaC25.9Pankreatitis, akutK85Pankreatitis, chronischK86.1ParaproteinmieD89.2Parkinson, M., primrerG20Parkinson syndrome, medik.G21.9pAVKI70.2Perikarditis, bakteriellI30.1Perikarditis, constrictivaI31. 1Perikarditis, urmischI32.8Perikarditis, rheumatischI01.0Peritonitis, akuteK65.0PhochromozytomE27.5PhimoseN47Phlebitis, leg, oberfl.I80.0Phlebothrombose, tiefI80.2PhlegmoneL03.9PlasmozytomC90.0PleuraerguJ90PleuritisR09.1Pleuritis with ErguJ90Pneumocystis cariniiB59Pneumocystis carinii (AIDS) B20 .6Pneumonie – generally, bakteriellJ15.9Pneumonie – generally, viralJ12.9Pneumonie – aspiration .J69.0PneumothoraxJ93.9Polyarthritis, chronischeM06.9Polymyalgia rheumaticaM35.3Polyneuropathie, alkoholischG62.1Polyneuropathie. diabetischG63.2Polypen, ColonD12.6PolytoxikomanieF19.2Portale HypertensionK76.6Postthrombotisches Syndr.I83.9PRINDG45.9ProstataadenomN40Prostata-CaC61PsoriasisL40.9Psychoorganische SyndromF07.9Querschnittslhmung without FrakturG82.2Purpura Henoch-HenochD69.0Quincke-OdemT78.3Querschnittslhmung with FrakturT09.3Radiusfraktur, distaleS52.5RadiusschaftfrakturS52.3Raynaud-SyndromI73.0RauchgasvergiftungT59.9RekurrenspareseJ38.0Reflux sophagitisK21.0Rektum-CaC20RektalflstelK60.4RektumprolapsK62.3Rheumatisches FieberI00Rheumatoide ArthritisM05.9SalmonellenenteritisA02.0SarkoidoseD86.9Sch delbasisbruchS02.1Sch deldachfrakturS02.0SchenkelhalsfrakturS72.0Schilddr sen-CaC73Schizophrenie, allg.F20.9Schock, anaphylaktischT78.2Schock, hypovolmischR57.1Schock, kardiogenR57.0Schock, septischA41.9SchwerhrigkeitH91.9Senile DemenzF03Sepsis, allg.A41.9Sick sinus SyndromI49.5Sigmadivertikulose / -itisK57.3Sinusitis, akutJ01.9Sinusitis, chronischJ32.9Sjgren SyndromM35.0SkabiesB86Sklerodermie, syst.M34.9Soor, MundB37.0SplenomegalieR16.1SprueK90.0SprunqqelenksfrakturS82.8Steatosis hepatisK76.0Struma diffusa, hyperthyr.E05.0Struma uninodosa, hyperthyr.E05.1Struma multinod., hyperthyr.E05.2Struma nodosa et diffusa, euthyreotE04.8Struma simplexE04.0SubarachnoidalblutungI60.9Subclavian-Steal-SyndromG45.8Subileus, mech.-spast.K56.6Subileus, paralytischK56.0SynkopeR55Tablettenvergift, AnalgetikaT39.8Tablettenvergift., BenzodiazepineT42.4Tuberkulose, AtmungsorganeA15.9Thrombozytopenie, allg.D69.6KrankheitICD- CodeKrankheitICD-CodeTachyarrhythmie I48Thrombose, leg, tiefI80.1Thrombophlebitis, BeinI80.3Thrombose, art.I74.9Thyreoiditis, HashimotoE06.3Thyreoiditis, acute (bacterial) E06.0Thyreoiditis subacute QuervainE06.1TIAG45.9Tonsillitis, allg.J03.9Tonsillitis, StreptokokkenJ03.0ToxoplasmoseB58. 9TrigeminusneuralgieG50.0Tiefe BeinvenenthromboseI80.1Ulcus crurisI83.2Ulcus ulcer, acute without haemorrhage or PerforationK26.3Ulcus ulcer, duodenal blutendK26.0Ulcus, chronischK26.7Ulcus ulcer, blutendK25.0Ulcus ulcer, chronischK25.7UlnaschaftfrakturS52.2UrmieN19UrethritisN34.1Uterus-CaC55Uterus, LeiomyomeD25. 9VaricosisI83.9VenenklappeninsuffizienzI87.2Vergiftung – AlkoholT51.9Vergiftung – AnalgetikaT39.8Vergiftung – AntidepressivaT43.0Vergiftung – AntidiabetikaT38.3Vergiftung – diazepam, Benzodiazep.T42.4Vergiftung – DigitalisT46.0Vergiftung – drugs, multipleF19.0VirusinfektB34.9Vorhofflimmern / -flatternI48Wegener – GranulomatoseM31.3WPW-SyndromI45 .6ZeckenborrelioseA69.2Zervix-CaC53.9Zirrhose, alkoholischK70.3Zirrhose, bilir, primrK74.3Zirrhose, bilir, sekundrK74.4Zirrhose, cardiaqueK74.6Zirrhose, fat K76.0ZliakieK90.0Zollinger-Ellison E16 .4ZosterB02.9Zwerchfellhernie, Q79.0ZystitisN30.9

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HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> 9 hObjbj . Ÿ Ÿ C l’’’’’’’ F F F F, GdezHzHzHzHzHUNUNUNddddddd $ Fg fi d ’UNMNUNUNUN d O’’zHzH Je O O OUNš’zH’zH d OUN d O O Vro`T’’oazHnH`mzH{ 8C F N^ `oa4`e0 e ` TjMOdTjoa O ’’’’ Prion-Erkrankungen Creutzfeld-Jakob disease incidence: 1-2 per year per 1 million inhabitants Befllt most people in the second Lebenshlfte. hereditary at about 10-15%. Prions (proteinacaeus infectiosus particles) are very resistant protein particles that can multiply in living cells, even though they have no Nukleinsuren. Proved were broadcasts by organ transplants, according to the use of infected neurosurgical instruments and pituitary. Clinical picture is characterized by 3 phases: organic brain syndrome develop severe dementia neurological Funktionsstrungen myoclonus, fasciculation, pyramidal signs, symptoms zerebellre prfinales coma and Dezerebrationssymptome are typical EEG Vernderungen (Periodic Aktivitt with highly strained triphasic Sharp … – and slow wave complexes No entzndlichen Vernderungen in cerebrospinal fluid or blood, but evidence of neuron specific enolase in cerebrospinal fluid (NSE) Average disease duration: 6 years in the brain of the deceased is spongy Vernderungen find (spongiform encephalopathy ..) glial shrinkage Other prion diseases: Kuru (in Guinea transmitted by cannibalism (tremor, cerebellar ataxia, dementia) scrapie (nerve disease in sheep and goats BSE (bovine spongiform encephalopathy) Gerstmann-Strussler-Scheinker syndrome (an autosomal dominant disorder Family might fetal insomnia (FFJ) (autosomal dominant (rapidly progressive insomnia, motor, autonomic and endocrine Funktionsstrungen. Expressive aphasia not flssige spontaneous speech with Agrammatismus literal paraphasias Many nouns, verbs little (telegram style) reading and repetition disturbed Sprachverstndnis is significantly less affected Written also disfigured Region: posterior portions of the inferior frontal gyrus (Area 44) Funikul re myelosis vitamin B12 avitaminosis shortage of vitamin leads to megaloblastic, hyperchromic anemia and symptomatic psychoses. Morphology: Multifocal reversible myelin sheaths loss in RM-Strngen. Later spongise Lckenfelder, irreversible Axonzerfall and glise scarring. Causes of vitamin B12 deficiency: Resorptionsstrungen in the absence of intrinsic factor Dnndarmaffektionen Pathological vitamin B12 consumption at fish tapeworm infestation Ungengende intake of vitamin Slinky progressive training of suffering. Schbe and remissions. Clinic: symmetrical, painful paresthesia of lower extremities abnormal Ermdbarkeit walking Strung of position sense and vibration sense Spinal ataxic Gangstrungen pyramidal signs with paraspastis but abgeschwchte reflexes! Blasenentleerungsstrungen differential diagnosis MS to consider: exclusion through lack of nystagmus, missing chanted language rarely Hirnnervenstrungen and obtained BHR. Therapy: Frhbehandlung with high dose parenteral vitamin B12 -Zufuhr since Rckbildung spinal symptoms can be expected as long as no irreversible Axonschden occurred. Maintenance treatment: 1 x monthly Vit B12 i.m.. Meningitis purulent Tuberkulse Acute lymphozytre Chronic lymphozytre exciting meningococcal pneumococcal Staphylococci Streptococci H. influenzaeTuberkelbakterienLeptospiren Borel Lien listeria Bruzellen Legionella VirenToxoplasmen cysticerci fungi Boeck place hood meningitis or diffuse brain basis meningitis diffuse, often encephalitis and myelitis basilar meningitis or diffuse clinic beginning fever neck stiffness Bewusstseinstr Special advertising acute high Highly Highly Anflle + Lhmungen Slinky Mig Mig Mig Hirnnervenlhmung spinal root symptoms acute Easy Mig Mig Mig Hardly Insidious Mig Hirnnervenlhmung encephalitic + Myelitische SymptomeLiquor appearance Pleozytose Total protein sugar lactate Trb-purulent segment Kernig Stark increases Stark Stark lowered increases Clear Lymphozytr Total protein Stark Stark lowered increases Clear Lymphozytr Hardly erh ht Normal Normal Clear Lymphozytr non increases often lowered Often forecast increases Good to doubtful (pneumococcal!) in targeted therapy doubtful even with targeted therapy. Complication by bonding with Liquorpassage- disability Mostly good Often ungnstig Clinical cardinal symptoms of meningitis headache neck stiffness (neck stiffness) Reizberempfindlichkeit (to light and pain stimuli) nausea, vomiting triggered by elevated intracranial pressure Acute purulent meningitis by different bacteria: streptococcus pneumococcal staphylococcus Haemophilus influenzae Gram-negative enterobacteria NOT by TB bacteria !!!!!!!!!! Way of pathogens in the meninges: -Open -Hmatogen-metastatic -Fortgeleitet brain injury. History usually crescendohaft with perakutem beginning, sometimes initial cerebral seizure. General Krankheitsgefhl symptomatic psychosis severe Bewusstseinsstrungen purulent CSF Epidemic occurrence by Trpfcheninfektion or under a hyperacute sepsis with renal bleeding, coagulation, petechiae and circulatory shock (Waterhouse-Friedrichsen syndrome). Absence of neck stiffness and fever Schlieen meningitis NOT from !!! Koma masked meningism !!! Complications: abscess and Empyembildung Irreversible brain and Nervenschden Epileptic Anflle occlusive hydrocephalus Pyocephalus therapy: antibiotics (cephalosporin of the 3rd generation, aminopenicillin) Tuberkulse meningitis occurs as a dispersion in a miliary on. History: often subacute or chronic. Frhsymptome: languor headaches back pain Mild fever process, especially at the base of the brain (Hirnnervenausflle (especially N. III, VI, VII) Rarely encephalitic or myelitische symptoms Liquor up on low sugar as nonbacterial meningitis diagnosis by PCR or.. LPA from CSF therapy. combination medication: isoniazid, ethambutol, Prothionamid initial glucocorticoids to counteract exudative productive Gewebsvernderungen with Verschwartung the meninges and development of hydrocephalus cysticercosis exciter:.. Taenia solium fin It is a chronic lymphozytre . meningitis affects mainly adults, 5-20 years after the Taenieneier localization:.. IV ventricle basal Grohirn clinic: Mental Hirnnervenlhmungen Vernderungen (dementia, Korsakov, delirium, depression) Epileptic Anflle brainstem symptoms intracranial pressure crises, hydrocephalus, papilledema Multiple calcifications in the R-image Chaotic decades history. Entzndlicher CSF eosinophil, glucose decreased. Herpes virus encephalitis The herpes viruses belong herpes simplex type 1 (HSV1) herpes simplex type 2 (HSV2) varicella zoster (VZV) cytomegalovirus (CMV) Epstein-Barr (EBV), herpes simplex encephalitis almost always triggered by HSV1. Clinic: (fever (headache (symptoms (lethargy (meningism (epileptic Anflle (aphasia Clinically and in the cerebrospinal fluid, CT and MRI viral encephalitis NOT have to be distinguished for all:.. The one that can lead to herniation transtentoriellen therapy : acyclovir !!! ErbgangGeschl.Manifesta-tion age distribution typVerlaufDychennex-chrome rezessivmnnlich0.-3 Lj.Beginn in BeckengrtelMaligne, quickly progressive, death before 25 Lj.Becker-Kienerx-chrome rezessivmnnlich5.-… 15. Lj.Beginn in BeckengrtelBenigne, slowly progressive, yet with 40-50 years gehfhigGliedergrtel-TypAutosomal- rezessivMnnlich and weiblich1.-50th Lj.Beginn in pool, shoulder or generalized mostly slow, with fr hem beginning verkrzte LebenserwartungKongenitale Muskeldystr. type de Lange (malignant) / type Batten-Turner benign) autosomal movements rezessivMnnlich and weiblichprnatalSchwache Kindsbe-, “floppy infant” death on 1st Lj.Fazio-skapulo-humeral M .Autosomal- dominantMnnlich and weiblich3.-50th Lj.Beginn on face and shoulder / ArmBenigne life expectancy slightly vermindertOkulre and Okulopharyn- Geale M.Autosomal- dominantMnnlich and weiblichFrhe childhood to sptes adults-age beginning with ptosis, Ophthalmic plegia ext., Swallow – rungenBenigne, life expectancy hardly gemindertDistale myopathy WelanderAutosomal- dominantMnnlich and weiblich40.-60th LjBeginn to distal muscles of HndeBenigne, almost normal life expectancy Clinical main symptoms of progressive muscular dystrophies: Lhmungen hypotension muscle atrophy Areflexie NO NO fasciculations Sensibilittsstrungen Moya-Moya Mainly in Japan occurring, ungeklrte progressive disease of the Circle of Willis with formation of thin-walled leptomeningeal anastomoses. These anastomoses see the angiogram as clouds of smoke, so the Japanese term “Moya-Moya”. Unknown thiologie. (Recurrent cerebral infarctions (Epileptic Anflle syndrome of total Querschnittslhmung Complete transection of all structures, usually traumatic, sometimes entzndliche genesis First spinal shock with the following symptoms below the Lsion:. Complete flaccid paralysis vollstndige L paralysis of the bladder (atone berlaufblase shock bladder), the intestines and loss of potency loss of Sensibilittfr all qualities areflexia of internal and external reflections failure of GEF and Wrmeregulation After a few days or weeks, it comes through development of spinal automatism to the manifestation of an actual cross section syndrome: possibly in Lsionshhe flaccid paralysis below the Lsion spastic para- / tetraplegia with flexion of the limbs (Beugerreflexsynergien, especially on legs) development of a spastic bladder (at Lsion above TH12) or an autonomous bladder (Complete with Lsion below Th12) Sensibilittsausfall Reflexrckkehr, possibly hyperreflexia and pathological reflexes when neck Markl Sion above C4 (Zwerchfelllhmung. Anterior spinal artery syndrome Ischmische Schdigung of Rckenmarks. Causes: Compression of the anterior spinal artery by space-occupying lesions, such as dislocated herniation Prodromi are Parsthesien and grtelfrmige pain height of spteren Lsion that precede the paralysis hours to days. Within minutes is rapidly but not apoplectiform a anfnglich flaccid paraplegia a. Dissociated Sensibilittsstrung: position and vibration sense intact, only pain and temperature sensation disturbed. If pain and temperature sense not checked up, a psychogenic paralysis may be suspected unlawful as pyramidal signs missing anfnglich. Sphinkterstrungen. Sometimes partly, rarely vollstndige Rckbildung of symptoms. Foster-Kennedy syndrome Tumor Caused initially developed by pressure effect ipsilateral optic atrophy and later with the insertion of a general intracranial pressure contralateral papilledema. The Foster-Kennedy syndrome usually arises from frontal brain tumors. Poliomyelitis caused by neurotropic enterovirus. transmission fkal-orally by dirt or smear infection. Incubation after oral virus uptake: 7-21 days. rarely through vaccinations today. Entzndungserscheinungen in the gray matter of the CNS, preferably in the motor anterior horn cells of Rckenmarks. Clinic shear history: Biphasic. Beginning with: fieberhaf-grippales preliminary slight meningeal irritation symptoms 1-4 days later: renewed fever increase flaccid paralysis form within a few hours to days, usually unregelmige distribution frame of the disease: asymmetric, proximal stressed flaccid paralysis weak / missing reflexes Muscle atrophy Possibly threatening paralysis of the respiratory muscles Motor Hirnnervenstrungen (eye muscles, facial us Schluckstrungen) MISSING Sensibilittsstrungen (initial possibly flchtige Parsthesien) Liquorvernderungen (only granulozytre, then lymphozytre Pleozytose and Eiweivermehrung) a few days after the onset of paralysis can lead to a Rckbildung. Often extensive Lhmungen stay back. In children, the growth of the affected extremities is beeintrchtigt. In Schdigung the trunk muscles leads to severe Wirbelsulendeformitten. Diagnosis: detection of pathogens in the stool to 3 weeks after onset. Antikrper in serum. Therapy: Unknown! Prophylaxis with active vaccination by Sabin oral ingestion abgeschwchter live vaccine or by Salk by i.m. Vaccination with inactivated virus suspension. Oculomotoriuslhmung / ophthalmoplegia The oculomotor nerve supplies levator palpebrae superior superior rectus inferior rectus medial rectus inferior oblique rectus sup. Obliquus inf. Rectus lat. Rectus med. (Abducens) rectus inf. Oblique sup. (Trochlear) with parasympathetic fibers innervate besides, the ciliary muscle (contraction (accommodation), and the sphincter of the pupil. Complete üre and inner Oculomotoriuslhmung ptosis eyeball deviated downward because only functions the abducens and trochlear obtained pupil mydriatic, immobile accommodation of is canceled the patient looks diagonally standing double images whose distance reinforced to look when trying to top causes:.. basal aneurysm trauma Basal meningitis neoplasm the Schdelbasis üre Okulomotoriuslhmung (ophthalmoplegia externa) autonomic innervation of the pupil and Zikiarmuskel receive often originate in the central area, as in peripheral Schdigung the autonomic fibers fail earlier than the somatic. inside Okulomotoriuslhmung ( ophthalmoplegia interna) Only the autonomous fibers gelhmt (pupil is far and immobile, but responds to miotics. If accommodation of is gelhmt, the patient in the Nhe can see not sharp. Mobility of the eyeball is obtained almost always cause the peripheral course. Grundstrungen the cerebellar syndrome Zerebellre ataxia: disordered interplay of movements.

ࡱ> >@ =

HSV Eraser Protocol
Rated 4.8/5 based on 1500 reviews



ࡱ> >@ = “ bjbj 4(e’e’ ~~SSSSS ggggsg 0‡‡‡‡‡‡‡‡SUUUUUU, T S‡‡‡‡‡ SS‡‡– ‡”S‡S‡S ‡S @> ‹‘ R ? 0 S D‡‡‡ ‡‡‡ ‡‡‡‡‡‡‡‡‡~›: (Herpes_lang.doc) Augenrzte warn herpes threatened vision! Almost every person carries the virus in itself. But few know that herpes can backfire million know this feeling: it tingles, it stressed, there is a fire on the lip. And suddenly form aching Blschen. They burst on, crust and heal without treatment after a week again. Herpes alert! But cold sores is by no means a harmless skin disease. What many sufferers do not know: “herpes viruses can also get into the eye and cause there partly serious damages,” warns ophthalmologist and Kongressprsident Dr. Armin Scharrer (Frth) at the 28th International Congress of the German Eye surgeons (DOC), the to 13 June will be held from 11th in Leipzig. “This damages range from simple Bindehautentzndung even blindness. Sometimes the examiner has only a cornea transplant “The fact is. About 90 percent of people in Germany who eventually infected with the herpes simplex virus type 1 (HSV-1), often in childhood. You carry this virus, both lips, as well as ocular herpes can auslsen, a lifetime in itself. For further jeopardize the immune system, infections, stress or mechanical irritation may occur an outbreak. Therefore, approximately 40 percent of adults suffer at least once in their lives under a cold sore. In almost every other sufferers of herpes breaks out more often. Sometimes even several times a year. is given to the Flssigkeit exiting when the burst Blschen particularly infektis. “Even by a thoughtless gesture can the virus be easily transmitted by contact infection in the eye,” says Dr. Scharrer. Also kisses are dangerous in this situation. From the lip to the eye – the distance is short. Augenrzte estimate that at cold sores mountain mature HSV virus in approximately one to ten percent of cases in the eye. Light infections often heal by itself again, especially with a strong immune system. But it can also lead to massive complications. In addition to the herpes simplex virus, there are still another danger: Even Varicella virus (VZV) can threaten eyesight. These are the pathogens from the group of herpes viruses that auslsen the gefrchtete Grtelrose in children and in adults chicken pox. This leads to a painful, streifenfrmigen rash with blisters on a Krperseite. Approximately one fnfte person falls ill during his life even to herpes zoster. Each year there are around 400,000. But: In the approximately 50,000 Grtelrose the face occurs in around 20,000 patients attack the virus on the cornea over. Dr. Scharrer: “So are herpes viruses by far the most common cause for a viral inflammation of the cornea.” This inflammation is manifested in typical complaints. When one eye turns red when it itches and burns or very much Flssigkeit secretes when a Fremdkrpergefhl is ( “it rubs like sand in the eye”) or when the eyes are glued morning on waking, a herpes his disease infection of the reason for this. “Whether these symptoms only a bacterial or a viral Bindehautentzndung or herpes infections of the cornea is present, only a skilled notice,” said Dr. Scharrer. “Therefore, each person should definitely consult an ophthalmologist in these complaints. Especially when at the same time or at different times a herpes disease or Grtelrose occurred. “If there is a oberflchliche herpes infection in the eye, they treated the ophthalmologist usually with eye drops or eye ointments, the antiviral active substances (eg Trifluoridin , idoxuridine, vidarabine, acyclovir). Have the herpes viruses already infested deeper layers of the cornea are also antiviral activity tablets or infusions and antientzndliche agents (steroids / cortisone) are used. A frhzeitige examination by an ophthalmologist and timely treatment can spread into deeper layers often prevent. If the herpes infection is on the eye but become chronic and swelling, Trbungen or scars formed in the cornea, helps in some cases only a corneal transplant. “Projections have the entire cornea and all layers are transplanted,” explains Dr. Scharrer. “Because there is always the risk that the herpes virus sets in the lowest layer, although only the top layers are geschdigt.” At least before Herpes zoster can you now appreciate by vaccination. Since 2013, a vaccine Details listed in Germany, which can reduce the onset of a Grtelrose and occurrence prolonged nerve pain after a herpes zoster outbreak by more than 50 percent. The zoster vaccine is approved for people over 50 years. The injection is required only once and takes place under the skin on the upper arm. Dr. Scharrer: “Because ten to twenty percent of all zoster infections the face and eyes concern and this risk increases significantly just from the age of 50 years, this new vaccine can protect even against zoster eye diseases. However, the decision for or against vaccination should always be made individually. In addition to the general practitioner also Augenrzte advise their patients liked about whether and when vaccination is advisable. “Fr infections with herpes simplex there is still no vaccine verfgbaren. Recent publications about experimental studies in animal models do but hopes zuknftige developments in this area. Fr patients who’ve been diagnosed with a more or less ausgeprgten herpes in the eye, the conjunctiva or eyelids, which has decayed again, new recommendations discussed at the DOC Congress. Dr. Scharrer: “Because the risk for recurrence is here comparatively high, the persons concerned should right now appreciate in the summer against UV radiation and wear a good quality sunglasses. Besides, recommended is the use of eye drops with artificial tears, because the moistening of the eyes can protect against a new outbreak. “\x26amp; “| j X A,h ; hmC5 B*CJOJQJ^JaJph#hOB*CJOJQJ^JaJph#hRvB*CJOJQJ^JaJph,h ; hl6’5 B*CJOJQJ^JaJph/h ; hmC5 B*CJOJQJ\\ ^JaJph)h ; hmCB*CJOJQJ^JaJph)h ; h WB*CJOJQJ^JaJph)h ; hl6’B*CJOJQJ^JaJph)h ; h B*CJOJQJ^JaJph

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