The herpes experts are coming – one is Already Here | WSU News | Washington

By Linda Weiford, WSU News PULLMAN, Wash. – More than 500 scientists from around the globe will gather in Idaho this week to confront the scarlet letter “H. ” Herpes, a common but highly stigmatized That virus has not healed, will be the focus of the 40th Annual International Herpesvirus Workshop in Boise , running Wednesday through Saturday. Nicola Anthony virologist Oversees to herpes simplex virus research lab at WSU. (Photo by Dean Hare, WSU Photo Services) Among the experts will be virologist Anthony Nicola of Washington State University, has spent 20 years WHO studying herpes simplex virus and WHO will convene a workshop session. That estimate Studies worldwide, 90 percent of people carry one or Both strains of herpes simplex, infamously Known for Causing lesions on the mouth or genitals. Not all carriers Develop symptoms. Of Those Who do, many go on to suffer lifelong, sporadic flare-ups. At WSU’s Department of Veterinary Microbiology and Pathology, Nicola probes how herpes simplex tricks the host cell into letting it in, and then a, how it use the cell’s own machinery to inflict two types of disease – One That Primarily strikes above the waist, the other below. Not only is he Credited With esta uncovering how crafty Takes Control of our microbe cells (see Journal of Virology http://www.

ncbi. nlm. nih. gov/pmc/articles/PMC153978/), but recently the National Institutes of Health Awarded $ 1. 9 million him to grant to further uncover the virus’s ruses. “What’s Most striking About the herpesvirus Is That after it causes the initial infection, rather than be cleared from the body, it goes into a latency stage Where It’s reliable to hide in nerve ganglia, beyond the immune system’s reach,” Said Nicola. Later, triggers: such as stress, fatigue or illness can reawaken the virus and set off a new outbreak, I Said. Stigma fuels transmission Type 1 is the oral version Generally Considered of herpes simplex and Type 2 the genital variety. However, it’s now Known That the two types can infect Either area, Said Nicola. Cases of genital herpes Have Been growing since the onslaught of HIV / AIDS, Especially Among teenagers, According to the U. S. Centers for Disease Control and Prevention.

Once Recognized as a major public health concern, the significance of herpes was dwarfed in the 1980s by the AIDS pandemic. Years later, “herpes have a stigma,” Said Nicola. Too Often, shame and humiliation are Associated With the disease, I Said. And That stigma – Along With the silence it breeds – Contributes to herpes’ Because so many people transmission are clueless That They are carriers. While some clusters of Individuals Develop painful sores in the genital area, others With mild symptoms May confuse them for something else or feel no signs at all. “The result is That We Have This large group of people Who Do They Have not know the virus can unknowingly spread it WHO to others,” Said Nicola. In fact, of the 776,000 Americans Who Will Estimated new contract herpes in 2015, Most will not even realize it, According to the CDC. They, in turn, pass it along to Could others. Complications Occur When herpes simplex is spread to the eye, a leading cause of blindness in Industrialized nations. But The most serious form is when it’s passed to newborns going through the birth canal of an infected mother, leading to brain damage and death. Know thy enemy Among other things, studies how the virus Nicola is reliable to reach the host cell’s cytoplasm, or nucleus, it churns out Where millions of new viral particles to launch infection. By detecting lowering levels inside the cell alkaline, “the herpesvirus abandons ship from inside the cell before ITS vesicle’s lysosome – an organelle That acts like a garbage disposal – can destroy it,” He Said.

Herpes simplex virus tricks the host cell into letting it inside. Then, by detecting lower pH levels inside the cell, the virus “abandons ship” from the vesicle before it can be destroyed, moving on to the cell nucleus to cause infection. (Diagram by Anthony Nicola) “Herpesvirus has-been a tough nut to crack. Because of Anthony’s research, We have a better understanding of how it gets inside the host cell to cause infection, “Said David Bloom, a virologist With the University of Florida School of Medicine WHO is co-chairing the workshop Boise. “It’s Important Because herpes is one of the MOST Widespread viruses we know of and eleven it Infects the body, it never leaves. ” Better understanding how esta tiny but complex enemy infiltrates host cells will help scientists identify identity intervention strategies and antiviral drug targets, I explained. Joining Bloom and Nicola at the Boise conference will be Nicola’s three Ph. D. His research lab students from herpes and scientists from 14 other Countries, Including China, Finland, Australia and Norway. Contacts: Anthony Nicola, WSU virologist, 509-335-6030, nicola@vetmed.

wsu. edu Linda Weiford, WSU News, 509-335-7209, linda. weiford@wsu. edu

ocular herpes

An ocular herpes occurs frequently in the form of a bloodshot eye. synonym herpes corneal If by “herpes” is mentioned, it is commonly known. Officially is herpes simplex virus (HSV), which in turn are divided into HSV1 and HSV2. About droplet infection (air) and smear infected during their lifetime about 90% of Bewölkerung with HSV1 and carries the virus then a lifetime in the nerve cells stored with it. The “herpes” occurs then symptomatic apparent when the immune system is weakened. Cause of immune weakness can be a simple infection, another illness that weakens the immune system or an unhealthy lifestyle. More about the causes of ocular herpes, click here. The infection is quite logical: If a person is suffering from cold sores and just suffered an outbreak, he or she can be easily transported by scratching the itchy blisters and then rubbing the eye the virus from one place to another. If the ocular herpes but recognized in time, they can be treated him well and fight definitely the symptoms occur quickly, so there is rarely consequential damages such as restriction of vision. Capitalized herpesviruses form known scabby crust at various parts of the body, but often on the lips (herpes labialis). Likewise, however, the virus can also infect the eye and all other parts of the body.

When ocular herpes (herpes cornea) are often the eyelids and the cornea affected. Rarely, an additional infection of more inner choroid of the eye. Ocular herpes (herpes corneas) is rarely the first appearance point of infection with HSV1 or HSV2, but usually the spread of a recurrent (recurrent) cold sores (herpes labialis). As described above, the herpes simplex virus type 1 is capable of after a single infection that mostly already taken place at a young age to linger for a lifetime in the body in its inactive form. It then settles in the nerve cells and stays there also usually completely unnoticed – until it just comes through an interplay of certain circumstances to a renewed outbreak of the disease. Often it happens that a just undergone cold or other disease has weakened our immune system and this is then no longer able to keep the herpes virus in the nerve cells under control and the viruses multiply again and can spread. Hormonal changes can, especially in women, leading to ocular herpes outbreak. Most often, however is usually an increased level of stress, the strain on the entire body of an unhealthy way and then, of course, in addition to the already strenuous meeting, the upcoming business trip or the big planned wedding to ocular herpes leads and complicates our lives. For more information, also read: ocular herpes causes In an ocular herpes may occur: Redness of the eyes, Foreign body sensation in Blink, strong burning and itching and

an increased secretion of fluid from the eye. Especially in the morning, the eyes are often glued. It can form bubbles on the lids that look similar to the cold sores. Often the cornea is affected by ocular herpes. More rarely it comes to a herpes infection between the Augapfelwand and the choroid. This attack, however, is all the more serious, since the area is under attack, which is necessary for the nutrition of the retina. For this reason, in this case, the risk of blindness. Herpes on the eye indiviudell developing very differently and examination by a doctor is essential. Symptoms Eye Herpes in short: The herpes corneas showing the typical symptoms of inflammation of the cornea (keratitis): eyes redness Foreign body sensation photosensitivity


Sehverschlechterung (Rare) sticky eyes Burning, itching Read more about: ocular herpes symptoms The more often the episodes occur, the more likely the scarring will affect over time, the eyesight, well out into other parts of the eye are affected with the herpes outbreaks. In severe cases, it can lead to an ulcer of the cornea, which can leave a hole in the cornea from breaking up. If the disease becomes chronic, it can lead to further eye diseases by itself. The ocular herpes can of course also occur in babies and toddlers. The disease here differs in no way from that of adults, only the treatment turns out to be a little more complicated, since the cooperation with the youngsters is often not given and is quickly rubbed times with his hands in his burning eyes. So here special attention of the parents is required. Read more about: Herpes Baby Ocular herpes is i. a.

treated with eye drops. The ophthalmologist will prescribe to diagnose ocular herpes (by smear on the eye) eye drops and / or eye ointments with Antivirals-active substances such as: acyclovir, ganciclovir, trifluorothymidine, Trifluridine and idoxuridine. Read more about: Zovirax® eye ointment From a self-treatment without a doctor visit is not recommended strongly in ophthalmic herpes disease! can be supported arranged by the ophthalmologist therapy involving the avoidance of direct sunlight, stress and Eyestrain. The eye drops or eye ointments with antivirals be dosed high for three days, then will be in the usual dosage for another ten days treated.

Prolonged local treatment with antivirals should be avoided as they can cause damage to the epithelial layer of the eye. Against the redness may in ocular herpes by no means ordinary eye drops, so-called “whiteners”, are used, as these cause a lower hydration of the eye, whereby the disease is further aggravated. Hygiene items, such as towels and washcloths should be shared in the family under any circumstances, but fall back as possible on disposable products by the person concerned, until the “herpes” is completely healed. Only by the strictest hygiene can contact infection and thus further spreading be avoided. If the ocular herpes was caused by another infection (bacterial) with, as antibiotics can be prescribed to treat the underlying disease. However, towards the herpesviruses even the antibiotics are ineffective. The herpes of the eye (corneal herpes) is the most common corneal inflammation in adulthood. Depending on the penetration depth of the following forms of herpes corneas can be distinguished: Keratitis dendritica: This form of ocular herpes concerns only the superficial epithelial layer of the cornea. The sensitivity of the cornea may be limited or can be completely eliminated. Keratitis disciformis: The stroma (middle layer of the cornea between the epithelium and the endothelium) is in this form of ocular herpes with affected, the epithelial layer is however intact. In stroma disc-shaped infiltrates are seen. Endothelial keratitis / uveitis: In severe cases of ocular herpes, there is a Einschwemmung of viruses in the aqueous humor, which can then lead to a swelling of the endothelial layer on the back of the epithelium, thereby contributing to glaucoma ( “Green Star”).

As already mentioned, is the most common cause of a renewed outbreak of ocular herpes stress. It is therefore important these be avoided if possible. So important are enough sleep (about eight hours per night), a healthy, balanced diet and a balance to the often stressful everyday life, such as sports or other hobbies. During an acute outbreak is natural to pay attention that the viruses are not additionally deported. So it is always to use your own washcloths and towels to keep their hands off the affected area and not to get too close to other people in the area with the face. The same is true for an outbreak of cold sores: sufferers should always meticulously careful after touching the herpes sores then wipe on the lips in no way her fingers over her eyes. In general should be avoided to scratch the blisters, as disturbing as they may be. Thus, the risk of developing ocular herpes can be greatly reduced already. The first occurrence of the disease is often an infection of the superficial layer. A corresponding treatment this usually heals quickly. But as the herpes viruses remain in the body, the disease can occur repeatedly. To a new eye herpes thrust can cause, among other things: Exterior irritation,

stress and infections In later episodes Herpes on the eye always the deeper layers of the cornea are affected with, which can lead to the cornea to dense scarring. It can then lead to a swelling and clouding of the cornea, which can lead to blindness in the affected eye. Self test eyesight How good is your eyesight? Test your visual acuity online. Answer to 15 short questions. Click here directly to the test eyesight For more information on ocular herpes can be found at: For more information on general topics, see: For an overview of all topics of ophthalmology under Ophthalmology A-Z.

German cure for herpes simplex 1 \x26amp; 2? at herpes simplex 1 forum topic 1140303


Hugo It has nothing to do with the conspiracy theories about the big corporations Suppressing a magic cure or the 100 MPG carburetor. No, I have no financial or quid pro quo connection with any pharmaceutical companies. from http://nocureforherpes. com/ “I know this all sounds like a bummer, but it’s the truth, at least for the foreseeable future, in spite of lots and lots of web sites did claim to have a magical” cure “. Those sites exist to make money. Our sites exist to help people deal with the not-so-dreadful reality of living with herpes alongwith about a quarter of the adult population who have genital herpes. Yes, we can talk about oral herpes, too, more Commonly known as cold sores or fever blisters. Almost all the adult population has did virus Eventually, Whether They know It or not. So what’s the deal and why is not there a cure? The problem with the idea of ​​a “herpes cure” is dass die herpesvirus is not a separate entity like a biological bacteria did kann targeted and killed.

It is a little snippet of DNA did Becomes part of the infected cell’s DNA. “Killing” the virus Involves killing the cell. Since the virus very Effectively hides from the immune system inside the cell, there’s not a good way at this time to kill only the affected cell without harming other similar uninfected cells. Further, the infected cells are silent functioning nerve cells and I’m not much in favor of killing off otherwise perfectly good nerve cells. Alright then, so what do we do about herpes? The very best treatments did we have today fall into two basic categories. One is the Nobel Prize winning antiviral drugs Which act to interfere with re-activation of the virus Which results in outbreaks and viral shedding. The other is enabling our immune system to Effectively keep the virus under control so we’re not bothered by it. Immune enhancement, in my opinion, is best Achieved by eating well, getting enough rest, exercise in moderation and active de-stressing. Yes, there are products on the market billed as immune enhancing “supplements”, But They are less well Evaluated for safety and efficacy than pharmaceuticals, so it’s somewhat a caveat emptor situation with regard to potential side effects. ” Tweet

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AIDS

AIDS and its treatment by Chinese Herbal Medicine © 1996 by Gunter R. Neeb In the beginning of the eighties, on patients with severe immune deficiencies a new virus which found by showing did most of prosthesis patients had antibodies against this virus. Nowadays this virus is known in two types, called Human Immunedeficiency Virus Type 1 and 2 (HIV-1 and HIV-2) of Which the first type is more common in the western world. It belongs to a subgroup of the retrovirus-Class, the Lentovirus-Group, Which lead only slowly to disease after infect Their host, seeking Causing a persisiting infection Which remains inactive for a long time. The symptomeless stage may take 10 Years or more, Which to aproximate maximum of 15 years afterwhich most patients show symptoms of mortal immune-deficiency. The virus’ genome besteht of RNA All which is copied to a complementary RNA-string by the enzyme reverse transcriptase. After the first flu-like acute infection in Which a high viral titer can be seen, the virus has killed and infected many CD4 T-Cells and macrophages. The reaction immunic besteht of antibodies, CD-4 and Helpercells -inflammative cells, as well as of cytotoxic CD 8-Cells. After this acute phase the blood is Effectively frred from the virus with a few circulating CD 4-Cells carrying one copy of the inactive genome of the virus. Although the peripheral blood contains no viruses during the inactive phase, the virus itself Reproduces Actively in the lymphatic tissue, brain, and someother regions.

this chronic infection Gradually destroys the CD4 T-Cells I n in a not yet completely Call explained ‘way. After almost all CD-4 T-Cells are destroyed, i. e. if Their count is below 200 per ul, the imunedeficiency stage is reached, and the syndrome is called AIDS. Gradually lymphatic folliculary dendritic cells are damaged leading to a loss of lymphatic follicles and thereafter a complete destruction of the lymphatic tissue. A similar process destroys the thymusstroma. If a new acute infection with HIV-1 or HIV-2 is Suspected or early enough detected, Several antivirus herbs from Chinese Medicine can be prescribed to secure a low infection rate. Since the toxicity of some is very low, They Can be used even as a preventive measure in order to keep the virus from spreading. One promising candidate among them is “zi hua di ding” or viola yedoensis (Pls. Refer to the Following table). Chinese medical herbs Affecting various viruses: List 1 name

Chinese name Effects on Virus / toxicity or LD50 Viola yedoyensis Zi hua di ding HIV / low toxicity arcticum lappa Niu ban zi HIV / low tox. Andrographis paniculata * Chuan xi lian HIV, ECH011 / 13. 4 g / kg Lithospermum rythrorhizon

Zi cao HIV, Jinke, flu, polio, hepatitis / 681. 13mg / kg Altemanthera philoxeroides Lung xin lian zi cao HIV, flu, encephalitis B. retrovirus rabies virus / 455. 5g / kg Lonicera japonica Jin yin hua HIV, flu (PR8), herpes / orphan (mice) 53g / kg Coptis chinensis Huang-lian

HIV, hepatitis B RuS, Newcastle disease / 24. 3mg / kg Epimedium grandiflorum Yin yang huo HIV, polio, ECHO 6. 9, Coxsackie A9, B4, B5 / 36g / kg Woodwardia unigemmata Guo ji zhong guan jue HIV, flu (PR8, Jinke 68-1,57-4, NewA1, Lee, C1232, D), England. II, polio II, Coxsackie, herpes simplex, encephalitis / 1. 7g / kg (mice) Prunella vulgaris Xia ku cao

HIV / low toxicity Senecio scandens Qian li guang HIV / 302. 6g / kg *** Hypericum japonicum Di he cao HIV, hepatitis B / no obvious toxicity Scutellaria baicalensis Huang qin HIV, flu PR8, (Asian A-flu) Xiantai, rhinovirus 17, adenovirus 7 / 3. 081g / kg * Baphicacanthis folium

Da qing ye flu, mumps, encephalitis B / no obvious toxicity Baphicacanthis rhizoma, radix Ban lan gen flu, hepatitis A, B, mumps, herpes encephalitis B. / no obvious toxicity Bupleuri radix Chai hu flu, smallpox / 4. 7g / kg gland (mice) Ledebouriellae radix Fang feng flu, Columbia SK

Polygonum cuspidatum Hu zhang herpes, ECHO9, ECHO11, adenovirus, hepatitis B. , flu, encephalitis B. Coxsackie A, B, polio II / 1363mg / kg * Forsythiae fructus Lian qiao flu (Asian A), nose-17 / 29. 37g / kg (mice) Taraxaci herba Pu gong ying ECHO11, herpes / 156. 3g / kg

* LD50 is given in the doses of chemical scents of the herb. ** LD50 is given in the doses of alcohol extract, IV injection. *** LD50 is given dependent on the herb produced in China; in some countries this herb is very toxic (Geissman, 1964). There are 20 herbs listed in Table 1. The antiviral spectrum and toxicities of each herb are given. Thirteen of them have inhibitory effects on the HIV. Some others have antivirus effects on other viruses. The HIV virus’ ability to avoid immunity response has much in common with some of thesis viruses: The permanent and latent infection is therefore seen in the Epstein-Barr virus and the herpes simplex virus; the antigenic variation by mutations can be found as well in trypanosomes, the influenza virus and Streptococcus some kinds. The induction of an immune-supression shares the HIV virus withsome Staphylococcus kinds and the Mycobacterium leprae. From an allopathic view, it may not be proper to list thesis herbs until theyhave been tested in vivo against the HIV virus; HOWEVER, from a TCM view, if Certain herbs have been proven effective against a Certain virus in clinical trials, the same mechanism may work against another virus in the human body. TCM holds did any herb or drug taken into the body must first affect the body’s metabolism so it can more did Effectively fight the pathogen. Although the concept of an antivirus is an allopathic approach, TCM’s principle of Treating febrile infectious diseases (qing re jie du, heat-clearing and toxin-Eliminating) with herbal remedies can give this approach a new meaning.

The toxicity of thesis herbs is very low, so They Can be used safely for a long period, even over a lifetime, by HIV-infected persons or, as Mentioned before as a preventive measure. The 13 herbs Which Showed inhibitory effects on HIV in vitro may therefore contribute to the treatment of various complications of AIDS. These patients are known to be subject of opportunistic infections and some kinds of cancer. Those are Caused by organisms Which Usually can be controlled Easily by the immune system, like Pneumocystis carinii pneumonia Causing, as well as E. G. Toxoplasma, the Cytomegaly virusand some Mycobacteria like M. tuberculosis and even some normally harmless kinds like M. avium. The functioning of the cellular defese is Necessary for at effective control of microorganisms thesis. Some patients (about 20%) is therefore affected by organisms, Which are Usually controlled by the humoral defense system like Haemophilus influenzae. THEREFORE mostimportant are probably Those herbs Which stimulate the growth of the T-lymphocytes as listed below: Common name asparagus (root)

atractylodes, white (rhizome) Coix (seed) Coriolus (sclerotium) epimedium (leaves) ganoderma (wholesale) lentinus (wholesale) ligustrum (fruit) ginseng (root) phaseolus, white (bean) huang-jing (rhizome) Besides AIDS Patients are liable to develope tumors like the Kaposi’s Syndrome, a slowly developing Usually proliferation of Bloodvessel cells, and to tumors of the B-Cells, the non-Hodgekin-Lymphomes and such of the brain. This might be explainable by a lacking T-cell response against tumor cells or a chronic stimulation of the B cells, another feature of HIV-infections. Since many herbs in Chinese Medicine havebeen proven to stimulate the immune system or some parts of it, thesis herbs can therefore be used in any stage of the HIV infection, i.

e. When An opportunistic infection the occurs or gene rally to stimulate the immune system. Pls. refer to list 2 below: Chinese medical herbs Affecting the immune system (list 2) Promoting lymphoblast transformation Botanical name Angilica sinensis, acutiloba Astragalus membranaceus Atractylodes macrocephala Codonopsis pilosula Coix lachryma-jobi Coriolus versicolor

Epimedium grandiflorum Ganoderma lucidum Gelatin equi asini Ligustrum lucidum Panax ginseng Phaseolus vulgaris Polygonatum sibiricum Polygonum multiflorum Increasing the Number of White Blood Cells Botanical name Acanthopanax senticosus Astragalus membranaceus Cinnamomum cassia


Codonopsis pilosula Comus officinalis Ganoderma lucidum Gelatin Equi asini Ligustrum lucidum Millettia dielsiana Panax ginseng Phaseolus vulgaris Placenta Homines sapientis Psoralea corylifolia Increasing phagocytosis of neutrophilic WBC Botanical Name Common Name Astragalus membranaceus Huangqi (root)

Atractylodes macrocephala atractylodes, white Dioscorea opposita dioscorea (root) Glycyrrhiza uralensis licorice (root) Panax ginseng ginseng (root) Increasing the Number of Mononucleic Macrophages Botanical Name Common Name Coriolus versicolor Coriolus (sclerotium) Glycyrrhiza uralensis licorice (root) Lentinus edodes lentinus (hole) Promoting Phagocytosis of Macrophages Mononucleic Botanical Name Common Name Acanthopanax senticosus ginseng, Siberian (root) Angilica sinensis, acutiloba tang-kuei (root)

Astragalus membranaceus Huangqi (root) Atractylodes macrocephala atractylodes, white (rhizome) Codonopsis pilosula codonopsis (root) Epimedium grandiflorum Epimedium (leaf) Eucommia Eucommia (bark) Ganoderma lucidum ganoderma, lingzhi (wholesale) Lentinus edodes lentinus (wholesale) Panax ginseng ginseng (root) Polyporus umbellatus polyporus (sclerotium) Psoralea Corylifolia psoralea (seed) Rehmannia glutinosa rehmannia (root) II. Herbs Affecting Non-Specific Humoral Immunities

Inducing the Production of Interferon Botanical Name Common Name Astragalus membranaceus astragalus (root) Phaseolus vulgaris (PHA) phaseolus, white (bean) (Ie Astragalus can promote the production of interferon by viral stimulations. ) Anti-Complementary Activities Botanical Name Common Name Cinnamon cassia cinnamon (twigs) Lentinus edodes lentinus (wholesale) (These herbs have triggering effects on the C3 complementary. ) III.

Herbs Affecting Specific humoral Immunities Those Promoting Hypertrophy of antigen-combining Cells in Mice obsessions at the Early Stage of the immuno-reaction Botanical Name Common Name Angilica sinensis tang-kuei (root) Astragalus membranaceus astragalus (root) Coix lachryma-jobi Coix (seed) Comus officinalis cornus (fruit) Those Promoting Hypertrophy of Antibody-Producing Cells Botanical Name Common Name Asparagus cochinchinensis asparagus (root) Coix lachryma-jobi Coix (seed) Coriolus versicolor Coriolus (sclerotium) Epimedium grandiflorum epimedium (leaves)

Ganoderma Lucidum ganoderma (wholesale) Ligustrum lucidum ligustrum (fruit) Ophiopogon japonica Ophiopogon (root) Polygonatum sibiricum huang-jing (rhizome) Polyporus umbellatus polyporus (sclerotium) Psoralea Corylifolia psoralea (seed) Those Suppressing Hypertrophy of Antibody-Producing Cells Botanical Name Common Name Glycyrrhiza uralensis licorice (root) Those Regulating Hypertrophy of Antibody-Producing Cells Botanical Name Common Name Astragalus membranaceus astragalus (root) Those Increasing Antibody Production

Botanical Name Common Name Astragalus membranaceus Huangqi (root) Coriolus versicolor Coriolus (sclerotium) Epimedium grandiflorum epimedium (leaves) Lentinus edodes lentinus (wholesale) Panaxginseng ginseng (root) Placenta hominis sapientis placenta, human Polygonum multiflorum he-shou-wu (wholesale) Rehmannia glutinosa rehmannia (root) Those Suppressing Antibody Production Botanical Name Common Name Angelica sinensis tang-kuei (root) Glycyrrhiza uralensis licorice (root)

Psoralea Corylifolia psoralea (seed) Zizyphus jujuba jujube (seed) IV. Those Herbs Affecting the Production of Different Types of immunoglobulin (Ig) Herbs Which Affect immunoglobulin (Ig) Promotive Suppressive IgG Lentinus edodes Psoralea Corylifolia Astragalus membranaceus IgA placenta hominis Psoralea Corylifolia (Serum) Rehmannia glutinosa IgA Astragalus membranaceus (Secretion) Ganoderma lucidum Polygoni multi floristic

Epimedium grandiflora floristic IgM Astragalus membranaceus Psoralea Corylifolia Coriolus versicolor IgE Astragalus membrananceus Because of the disruption of the immune system, various allergic reactions can be seen in AIDS and HIV (+) patients, most notably hay fever and allergic sinusitis. The Following list contains herbs Which can be used for different types of allergic conditions: List 3: Herbs Affecting Allergic Reactions Herbs Affecting Type I Allergic Reactions Those Suppressing the secretion of histamines Botanical Name Common Name Ganoderma Lucidum ganoderma (sclerotium) Those Providing relief from bronchial spasms due to histamines and Acetylecholine Botanical Name Common Name

Coriolus versicolor Coriolus (sclerotium) Ganoderma Lucidum ganoderma (sclerotium) Epimedium grandiflorum epimedium (leaves) Placenta hominis sapientis placenta, human Psoralea Corylifolia psoralea (seed) Those Providing Relief of Gastrointestinal Smooth Muscle Spasms Caused by Histamine and Acetylecholine Botanical Name Common Name Angelica sinensis tang-kuei (root) Cinnamomi cortex cinnamon (twigs) Comus officinalis cornus (fruit) Glycyrrhiza uralensis licorice (root) Those Which Suppress Allergic Shock or Allergic Skin Reactions Caused by Foreign protein Botanical Name Common Name

Glycyrrhiza uralensis licorice (root) Panax ginseng ginseng (root) Placenta hominis sapientis placenta, human II. Those Affecting Type II Allergic Reactions (Cytolytic) Those Preventing ABO Hemolysis Botanical Name Common Name Glycyrrhiza uralensis licorice (root) Those Increasing the Number of Platelets Botanical Name Common Name Glycyrrhiza uralensis licorice (root) Placenta hominis sapientis placenta, human Rehmannia glutinosa rehmannia (root)

Those Increasing the Number of Red Blood Cells Botanical Name Common Name Acanthopanax senticosus ginseng, Siberian (root) Codonopsis pilosula codonopsis (root) Equi asini equine gelatine Panax ginseng ginseng (root) (These herbs can therefore affect hemolysis. ) III. Those Affecting Type III Allergic Reactions Botanical Name Common Name Astragalus membranaceus astragalus (root) Glyzyrhiza uralensis licorice (root)

Rehmanniae radix rehmannia (root) IV. Those Affecting Type IV Allergic Reaction (Delayed) Botanical Name Common Name Angelica sinensis tang-kuei (root) Bombyx batryticatus silkworm (wholesale) Glycyrrhiza uralentsis licorice (root) As Mentioned above some kinds of cancer can Occur positive in HIV patients. Since there are many herbs in TCM proven effective against various kinds of cancer, only to be listed here excerpt can, since this would exceed this topic by far. So finally in List 4 are listed only herbs Those Which may be effective against cancers in typical AIDS patients, namely leukemia and lymphoma. Since some of thesis autumn are toxic or cytistatic Themselves, They Should not be used Before They are Indicated. List 4: Herbs for cancers in AIDS patients (late stage) Botanical Name Chinese Name

Polysticticum yun zhi Hedyotis diffusa bai hua she she cao Indigo naturalis qing dai Catharanthi rosei chang chun hua Lysimachiaiae Clethroidis zhen zhu cai Botanical Name Chinese Name Camptothecae Acuminatae xi shu Sarcandra glabra zhong jie feng Curcuma aromatica / zedoaria e zhu Brucea javanica ya dan zi Of thesis herbs the first one in the list, Polystipticum is one of the most interesting herbs, since its toxicity is very low, while it is useful for leukemia, lymphoma and liver cancer, by inhibiting the synthesis of the DNA and RNA of tumor cells , Besides this it antagonizes the immunosuppression Caused by cyclophosphamides and cortisone, while Increasing the amount of macrophages, Promotes phagocytosis and Enhances the Board of blastogenesis and rosette forming, seeking being a promising agent for Both enhancing the immune system and Suppressing tumor developement. At the moment clinical western medicine hasnt yet found any solutions exept medicaments like AZT, Which inhibit the function of the reverse transcriptase and THEREFORE have a toxic effect on human cells as well. Some other proposals are searching to find specific inhibitors of the viral protease or the developement of antibodies for the virus surface or synthezising substances Which ought to block the docking of gp120 to the CD4.

But They are quiet far from ready to be used in clinical practice. THEREFORE Traditional Chinese Medicine might Provide some useful alternative by combining herbs with western medicaments or even use them alone in different stages of HIV-Positive patients and other types of immune deficiency syndromes.

Il virus Hiv dell’Aids, i condilomi e l’genital herpes

Anche se oggi l’attenzione è rivolta soprattutto all’AIDS (trasmessa dal virus dell’HIV), è bene anche ricordare le altre malattie provocate as virus, per esempio i condilomi, le infezioni erpetiche, l’epatite A e B. In particolare , nei casi in cui si verificano rapporti “a rischio” (non protetti con partner occasionali o con un partner stable surface, però, ha avuto a sua volta rapporti “a rischio”) si dovrebbero effettuare controlli medical Periodici, per rilevare, così, l’eventuale presenza di infezioni e iniziare in modo tempestivo il trattamento più idoneo. AIDS Anche se l’è una AIDS non patologia urogenital, può essere trasmessa sessualmente ed è importante perticolarmente esserne informati. Il virus dell’AIDS è un retrovirus (genere di virus A RNA, ossia acido ribonucleico, appartenente alla famiglia Retroviridae, a cui fanno capo tutti i virus tumorali) che attacca alcune cellule del sistema immunitario, i linfociti CD4 principalmente, indebolendolo fino ad annullare la risposta contro virus, batteri, protozoi e funghi. La distruzione del sistema immunitario causa una sindrome che si chiama AIDS (Sindrome da immunodeficienza acquisita). Una persona Affetta since AIDS è maggiormente esposta all infezioni. Quando una persona entra in contatto con il virus dell’HIV può diventare sieropositiva (quindi risulta positiva alla ricerca di Anticorpi dell’HIV nel siero). Questo può verificarsi dopo un certo periodo, detto periodo finestra, che può durare fino a sei mesi. Il test quindi non indica la presenza del virus, ma solo degli Anticorpi specifici che il sistema immunitario ha sviluppato dopo il contatto con il virus. Se il test risulta negativo va comunque ripetuto allo scadere dei 6 mesi, calcolati a partire dall’ultimo episodio ritenuto “a rischio”. Un persona che è positiva al primo test, viene sottoposta ad altri test di conferma. In seguito vengono effettuati test per se valutare e quanto il virus HIV ha danneggiato il sistema immunitario: fondamentale è la conta dei linfociti CD4.


L ‘esame che misura la quantità di virus nel siero è fondamentale perché permette tra l’altro una verifica indiretta dell’efficacia dei farmaci antiretrovirali. Esistono anche alcuni esami che servono a individuare i ceppi mutanti resistenti ai farmaci. Se si una persona ritrovano gravi danni al sistema immunitario e la presenza di infezioni opportunistiche (ossia le infezioni che colpiscono con maggiore facilità i Pazienti affetti as AIDS, per esempio la polmonite), si l’diagnostica AIDS. L’infezione as HIV si trasmette in tre modi: per via ematica: le trasfusioni di sangue infetto possono trasmettere il virus HIV, come anche lo scambio di siringhe infette sessuale per via per via verticale, ossia la madre può trasmettere il virus HIV al figlio durante la gravidanza, al momento del parto o durante l’allattamento. Torna su Condilomi Si tratta di escrescenze della pelle di tipo verrucoso che di colpiscono preferenza le zone genitali, sia nel maschio sia nella femmina. Sono generalmente provocate dal virus HPV (Human Papilloma Virus) e si trasmettono prevalentemente con l’attività sessuale. I sintomi sono molto Lievi, a meno che non si sovrapponga un’altra infezione. Si possono verificare, oltre alla crescita di escrescenze nelle zone genitali, perdite di sangue e prurito.

I condilomi vulvari sono simili all verruche che si possono manifestare in altre parti del corpo. Si diffondono più facilmente in condizioni di umidità elevata e sono più frequenti vaginal in caso di aumento delle perdite vaginali, come nel caso della candidosi. I condilomi più piccoli possono essere con trattati applicazioni locali, quelli più grandi richiedono la Rimozione chirurgica. Nel trattamento di queste lesioni è semper raccomandabile poter effettuare l’esame istologico di tutta la lesione asportata. Se non trattate possono persistere per diversi mesi e persino anni. Le lesioni possono aumentare di numero e spargersi in zone anche distanti. Con il passare del tempo, se non trattate, possono anche scomparire. Poiché i condilomi si possono trasmettere con rapporti non protetti, in caso se ne riscontri la presenza, anche il partner Andra attentamente controllato ed eventualmente curato. Torna su genital herpes L’genital herpes è una patologia causata as virus herpes simplex di tipo II ed è un’infezione a trasmissione sessuale molto somigliante a quella provocata dal virus dell’herpes labial di tipo I. Si tratta delle cosiddette “febbri” ossia source bollicine fastidiose che appaiono sulle labbra ma anche in altre zone della cute solitamente durante un’influenza, un periodo di stress, una intensa esposizione al sole. Esse sono alla dovute replicazione del virus all’interno delle cellule.

La prima volta che si l’contrae genital herpes, i sintomi sono in genere abbastanza importanti: alla presenza di bollicine Bianche, in numero variabile da due venti fino a e più, si accompagna un senso di prurito e tensione fastidiosamente doloroso. Ogni tentativo però di portarsi sollievo grattandosi è inibito dall’estremo dolore che anche lo provoca sfiorarsi. A questi si sintomi unisce inoltre spesso febbre e delle rigonfiamento ghiandole inguinali e rossore generalizzato della parte interessata. Il virus dell’herpes una volta contratto tend a rimanere all’interno del corpo umano per ricominciare a replicare in momenti di deficit immunitario, ma gli Episodi successivi sono molto meno drammatici in quanto il sistema immunitario conserva la memoria del virus e ne Arresta la replicazione in tempi molto più brevi. In presenza dei sintomi descritti è necessario sottoporsi a una visita ginecologica. Il trattamento per eccellenza è quello a base di farmaci antivirali, che non solo mitigano i sintomi ma riducono la durata della malattia. È necessario astenersi dai dai primi rapporti Sessuali fin sintomi e in realtà si pensa che che siano coloro affetti as genital herpes Possano trasmetterlo anche nei periodi di latenza. Un comportamento corretto da parte dei portatori di genital herpes sarebbe quello di comunicare al partner lo stato di possibile contagiosità. Torna su

Symptoms of HIV in the mouth

11/15/2011 by Diego Oral manifestations of HIV are available in all three symptomatic stages of HIV disease. They are serious when HIV disease progressed to the third stage (symptomatic HIV) and occur during this stage and the fourth stage of HIV disease, AIDS. have symptoms of HIV disease, which appear in the mouth, such as the case with other symptoms, is a number of different causes (bacteria, viruses, fungi) and therefore a number of different treatments Acute HIV infection According to the authors of “HIV infection in Primary Care” by R. Michael Buckley and Stephen J. Gluckman there are a number of oral symptoms of acute HIV infection, including ulcers, sore throat, swelling of the throat and thrush, a fungal infection. Some people experience no symptoms during the acute infection, and some people the symptoms are so mild that they go unnoticed. appear Symptoms of acute HIV infection, generally within two to four weeks after infection and clear up within a month. The most important thing about the symptoms of acute HIV infection is not to forget that they Diracodon; This means that even people seeking medical treatment for these symptoms seek alone will not diagnosed based on symptoms with HIV. Asymptomatic HIV After the acute infection HIV enters an asymptomatic phase.


In this phase, which lasts an average of 10 years, there are no symptoms of HIV, in the mouth or elsewhere. Because of this and the generally mild nature of the symptoms of acute HIV infection the only way HIV status is to be tested to know progress against HIV disease. HIV testing is widely available and easily, and to know the status allows appropriate measures are taken in order and to protect others himself. HIVTest. org helps people find more information about HIV testing and testing centers; See Resources below. Symptomatic HIV A number of symptoms of HIV disease may occur or affect mouth during symptomatic HIV infection, the third stage of the disease. Symptomatic HIV takes between one to three years before AIDS develops, and the symptoms are usually either chronic or recurring. Symptoms that may indicate the mouth or surrounding organs include loud HIV-Symptoms. info shortness of breath and dry cough. The mouth is directly influenced by fungal infections, thrush, a type of oral fungus that causes white plaques and sometimes red patches in the mouth, including by HIV InSite be affected. AIDS While AIDS, the fourth and final stage of HIV disease, a number of infections, viruses, fungi and bacteria, including lead to oral symptoms.

This can cause lesions or blisters, and in some cases lead to damage or destruction of oral tissues. Besides thrush other fungi caused oral symptoms AIDS histoplasmosis and Cryptococcus neoformans. Both lead to ulcerations in the mouth; Histoplasmosis on muscosal tissue and Cryptococcus neoformans on the hard palate, after HIV InSite. Among the causes virus symptoms AIDS are herpes viruses, including cold sores, which can be difficult especially in people with weakened immune systems. Another possibility is the oral participation of Herpes Zoster or shingles, causing pain and rash with blisters and which affect the mouth or nearby areas of the face. Cytomegalovirus, another herpes virus, can cause necrotic lesions in the mouth after HIV InSite. Human herpesvirus-4, also known as Epstein-Barr, may be oral hairy leukoplakia, lead white lesions appear, Be hairy and found that along the mucosal tissue. Human papillomaviruses (HPV) cause oral lesions in AIDS. Bacterial infections in the mouth while AIDS can necrotizing ulcerative periodontitis (NUP) and linear cause gingival erythema (LGE). After HIV InSite both can result in loss of soft tissue and bone in the mouth, lead a person regardless of the state of oral hygiene. Cancer can occur in the mouth during AIDS, including Kaposi’s sarcoma and lymphoma. Kaposi’s sarcoma results in lesions that generally purple, but can also blue or red; by HIV InSite they may ulcerate or infected. Lymphoma in the mouth appears as a painless swelling, after HIV InSite, ulcerate can.

treatment Treatment for oral conditions associated with HIV disease varies depending on the specific root of the problem. Many conditions respond favorably to treatment with antiretroviral drugs, the treatment of HIV disease generally used. Others, however, must be precisely aligned with agents such as antibacterial, antiviral, antifungal and antiparasitic drugs. The treatment of any oral manifestation of HIV must be determined in conjunction with a health care professional to ensure that it does not lead effectively and adverse interactions with other medications. Related